Ka Sin Mak

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Loss of Krüppel-Like Factor 3 (KLF3/BKLF) Leads to Upregulation of the Insulin-Sensitizing Factor Adipolin (FAM132A/CTRP12/C1qdc2)

N. Turner, A. Sainsbury, A. Funnell, 2013, Diabetes.

Phosphorylation of Krüppel-like Factor 3 (KLF3/BKLF) and C-terminal Binding Protein 2 (CtBP2) by Homeodomain-interacting Protein Kinase 2 (HIPK2) Modulates KLF3 DNA Binding and Activity*

K. Quinlan, S. Cordwell, K. Isono, 2015, The Journal of Biological Chemistry.

Generation of Mice Deficient in both KLF3/BKLF and KLF8 Reveals a Genetic Interaction and a Role for These Factors in Embryonic Globin Gene Silencing

Laura J. Norton, A. Perkins, M. Wilkins, 2013, Molecular and Cellular Biology.

The CACCC-Binding Protein KLF3/BKLF Represses a Subset of KLF1/EKLF Target Genes and Is Required for Proper Erythroid Maturation In Vivo

Laura J. Norton, S. Orkin, A. Perkins, 2012, Molecular and Cellular Biology.

Running title: EKLF/KLF1 and KLF3 in erythropoiesis 3

Laura J. Norton, S. Orkin, M. Wilkins, 2012 .

Impaired B Cell Development in the Absence of Krüppel-like Factor 3

Laura J. Norton, M. Cowley, W. Kaplan, 2011, The Journal of Immunology.

Title Replication stress induces mitotic death through parallelpathways regulated by WAPL and telomere deprotection

David C. S. Huang, Anthony J. Cesare, M. Hayashi, 2019 .

Replication stress induces mitotic death through parallel pathways regulated by WAPL and telomere deprotection

David C. S. Huang, Anthony J. Cesare, V. Masamsetti, 2018, Nature Communications.

Erratum to: Repression of chimeric transcripts emanating from endogenous retrotransposons by a sequence-specific transcription factor

Laura J. Norton, M. Crossley, Richard C. M. Pearson, 2016, Genome Biology.

of Krüppel-like Factor 3 Impaired B Cell Development in the Absence

Laura J. Norton, M. Cowley, W. Kaplan, 2011 .

Changes in glycogen structure over feeding cycle sheds new light on blood-glucose control.

F. Warren, D. Stapleton, R. Gilbert, 2014, Biomacromolecules.

Differential regulation of the α-globin locus by Krüppel-like factor 3 in erythroid and non-erythroid cells

K. Quinlan, D. Higgs, D. Vernimmen, 2014, BMC Molecular Biology.

Repression of chimeric transcripts emanating from endogenous retrotransposons by a sequence-specific transcription factor

Laura J. Norton, M. Crossley, Richard C. M. Pearson, 2014, Genome Biology.

Regions outside the DNA-binding domain are critical for proper in vivo specificity of an archetypal zinc finger transcription factor

M. Crossley, Richard C. M. Pearson, Alister P. W. Funnell, 2013, Nucleic acids research.

A CpG mutational hotspot in a ONECUT binding site accounts for the prevalent variant of hemophilia B Leyden.

Michael D. Wilson, D. Odom, P. Flicek, 2013, American journal of human genetics.

PU.1 and Haematopoietic Cell Fate: Dosage Matters

M. Crossley, Richard C. M. Pearson, Alister P. W. Funnell, 2011, International journal of cell biology.

Impaired B Cell Development in the Absence of Krüppel-like Factor 3

Laura J. Norton, M. Cowley, W. Kaplan, 2011, The Journal of Immunology.

Edinburgh Research Explorer Differential regulation of the alpha-globin locus by Kruppel-like factor 3 in erythroid and non-erythroid cells

K. Quinlan, D. Vernimmen, Richard C. M. Pearson, 2022 .