An-Sheng Zhang

发表

Evidence That Inhibition of Hemojuvelin Shedding in Response to Iron Is Mediated through Neogenin*

K. R. Meyers, R. Eisenstein, C. Enns, 2007, Journal of Biological Chemistry.

Suppression of hepatic hepcidin expression in response to acute iron deprivation is associated with an increase of matriptase-2 protein.

H. Tsukamoto, Jiaohong Wang, R. Eisenstein, 2011, Blood.

Processing of hemojuvelin requires retrograde trafficking to the Golgi in HepG2 cells.

C. Enns, An-Sheng Zhang, J. Maxson, 2008, Blood.

Hepatocyte Neogenin Is Required for Hemojuvelin-Mediated Hepcidin Expression and Iron Homeostasis in Mice.

C. Enns, An-Sheng Zhang, S. Jue, 2021, Blood.

The Ectodomain of Matriptase-2 Plays an Important Non-Proteolytic Role in Suppressing Hepcidin Expression in Mice.

C. Enns, An-Sheng Zhang, S. Jue, 2020, Blood.

The catalytic, stem, and transmembrane portions of matriptase-2 are required for suppressing the expression of the iron-regulatory hormone hepcidin

C. Enns, An-Sheng Zhang, P. Mao, 2018, The Journal of Biological Chemistry.

Matriptase-2 suppresses hepcidin expression by cleaving multiple components of the hepcidin induction pathway

C. Enns, An-Sheng Zhang, S. Jue, 2017, The Journal of Biological Chemistry.

The Role of Hepatocyte Hemojuvelin in the Regulation of Bone Morphogenic Protein-6 and Hepcidin Expression in Vivo*

D. Koeberl, C. Enns, An-Sheng Zhang, 2010, The Journal of Biological Chemistry.

Hemojuvelin-Neogenin Interaction Is Required for Bone Morphogenic Protein-4-induced Hepcidin Expression*

H. Tsukamoto, Jiaohong Wang, C. Enns, 2009, The Journal of Biological Chemistry.

Iron Homeostasis: Recently Identified Proteins Provide Insight into Novel Control Mechanisms*

C. Enns, An-Sheng Zhang, 2009, Journal of Biological Chemistry.

Direct interorganellar transfer of iron from endosome to mitochondrion.

Claire M. Brown, O. Shirihai, P. Ponka, 2006, Blood.

An iron-regulated and glycosylation-dependent proteasomal degradation pathway for the plasma membrane metal transporter ZIP14

M. Knutson, C. Enns, Christal A. Worthen, 2014, Proceedings of the National Academy of Sciences.

Inflammatory Arthritis Negatively Regulates Mouse Α Pilr

K. Lin, An-Sheng Zhang, M. Young, 2022 .

Intracellular kinetics of iron in reticulocytes: evidence for endosome involvement in iron targeting to mitochondria.

P. Ponka, A. Sheftel, An-Sheng Zhang, 2005, Blood.

Hepatic macrophage iron aggravates experimental alcoholic steatohepatitis.

V. Gordeuk, H. Tsukamoto, S. French, 2008, American journal of physiology. Gastrointestinal and liver physiology.

The catalytic, stem, and transmembrane portions of matriptase-2 are required for suppressing the expression of the iron-regulatory hormone hepcidin

C. Enns, An-Sheng Zhang, P. Mao, 2018 .

Neogenin Facilitates the Induction of Hepcidin Expression by Hemojuvelin in the Liver*

C. Enns, An-Sheng Zhang, J. Maxson, 2016, The Journal of Biological Chemistry.

Low Intracellular Iron Increases the Stability of Matriptase-2*

H. Tsukamoto, Jiaohong Wang, Akiko Ueno, 2014, The Journal of Biological Chemistry.

Mechanisms of HFE-induced regulation of iron homeostasis: Insights from the W81A HFE mutation

Paige S. Davies, C. Enns, H. Carlson, 2003, Proceedings of the National Academy of Sciences of the United States of America.

involvement in iron targeting to mitochondria Intracellular kinetics of iron in reticulocytes: evidence for endosome

P. Ponka, A. Sheftel, An-Sheng Zhang, 2013 .

Use of Nramp2-transfected Chinese hamster ovary cells and reticulocytes from mk/mk mice to study iron transport mechanisms.

P. Gros, S. Gruenheid, P. Ponka, 2008, Experimental hematology.

The anemia of "haemoglobin-deficit" (hbd/hbd) mice is caused by a defect in transferrin cycling.

P. Ponka, A. Sheftel, An-Sheng Zhang, 2006, Experimental hematology.

Endoplasmic reticulum stress controls iron metabolism through TMPRSS6 repression and hepcidin mRNA stabilization by RNA-binding protein HuR

Manuel D. Díaz-Muñoz, H. Guillou, A. Polizzi, 2020, Haematologica.

Grab and go: transferrin uptake in erythropoiesis.

C. Enns, An-Sheng Zhang, 2022, Blood.

Neogenin-mediated Hemojuvelin Shedding Occurs after Hemojuvelin Traffics to the Plasma Membrane*

P. Bjorkman, C. Enns, Fan Yang, 2008, Journal of Biological Chemistry.

Increased Iron Loading Induces Bmp6 Expression in the Non-Parenchymal Cells of the Liver Independent of the BMP-Signaling Pathway

H. Tsukamoto, Jiaohong Wang, Akiko Ueno, 2013, PloS one.

Neogenin Interacts with Matriptase-2 to Facilitate Hemojuvelin Cleavage*

Caroline A Enns, Riffat Ahmed, An-Sheng Zhang, 2012, The Journal of Biological Chemistry.

Control of systemic iron homeostasis by the hemojuvelin-hepcidin axis.

An-Sheng Zhang, 2010, Advances in nutrition.

Interaction of the hereditary hemochromatosis protein HFE with transferrin receptor 2 is required for transferrin-induced hepcidin expression.

H. Tsukamoto, C. Enns, M. Kramer, 2009, Cell metabolism.

Iron in mammals: pathophysiological mechanisms of overload and deficiency in relation to disease

C. Enns, An-Sheng Zhang, Martha B. Johnson, 2005 .

Interaction of Hemojuvelin with Neogenin Results in Iron Accumulation in Human Embryonic Kidney 293 Cells*

A. West, P. Bjorkman, C. Enns, 2005, Journal of Biological Chemistry.

Iron regulation by hepcidin.

C. Enns, An-Sheng Zhang, Ningning Zhao, 2013, The Journal of clinical investigation.

Discovery and function of hepcidin in iron homeostasis

C. Enns, An-Sheng Zhang, Ningning Zhao, 2022 .

Matriptase-2- and Proprotein Convertase-cleaved Forms of Hemojuvelin Have Different Roles in the Down-regulation of Hepcidin Expression*

C. Enns, An-Sheng Zhang, J. Maxson, 2010, The Journal of Biological Chemistry.

Bone Morphogenetic Protein-6 Mutations Take Their Place in Iron Overload Diseases.

C. Enns, An-Sheng Zhang, M. Kleven, 2016, Gastroenterology.

Molecular mechanisms of normal iron homeostasis.

Caroline A Enns, An-Sheng Zhang, C. Enns, 2009, Hematology. American Society of Hematology. Education Program.

Extrahepatic deficiency of transferrin receptor 2 is associated with increased erythropoiesis independent of iron overload

W. Fleming, C. Enns, D. Goldman, 2020, The Journal of Biological Chemistry.

Localization of iron metabolism-related mRNAs in rat liver indicate that HFE is expressed predominantly in hepatocytes.

H. Tsukamoto, S. Xiong, C. Enns, 2004, Blood.

levels independently of transferrin receptor 1 in TRVb cells The hereditary hemochromatosis protein, HFE, lowers intracellular iron

W. Fleming, C. Enns, H. Carlson, 2013 .

Stoichiometries of transferrin receptors 1 and 2 in human liver.

C. Enns, An-Sheng Zhang, M. Chloupková, 2010, Blood cells, molecules & diseases.

The hereditary hemochromatosis protein, HFE, lowers intracellular iron levels independently of transferrin receptor 1 in TRVb cells.

W. Fleming, C. Enns, H. Carlson, 2005, Blood.

Evidence for the interaction of the hereditary haemochromatosis protein, HFE, with the transferrin receptor in endocytic compartments.

M. Lampson, T. McGraw, Paige S. Davies, 2003, The Biochemical journal.

The Tumor Suppressor, P53, Decreases the Metal Transporter, ZIP14

M. Knutson, C. Enns, An-Sheng Zhang, 2017, Nutrients.

Adhesion of Thymocytes to Bone Marrow Stromal Cells: Regulation by bFGF and IFN‐γ

M. Barda-Saad, D. Zipori, L. Rozenszajn, 1997, Stem cells.

Matriptase-2 regulates iron homeostasis primarily by setting the basal levels of hepatic hepcidin expression through a nonproteolytic mechanism

M. Kawaguchi, C. Enns, An-Sheng Zhang, 2023, The Journal of biological chemistry.