D. Lodwick

发表

Insertion/deletion polymorphism in the angiotensin-converting enzyme gene and risk of and prognosis after myocardial infarction.

A. Morice, N. Samani, J. Thompson, 1996, Journal of the American College of Cardiology.

Insertion/deletion polymorphism in the angiotensin-converting enzyme gene and risk of restenosis after coronary angioplasty

N. Samani, A. Gershlick, D. D. Bono, 1995, The Lancet.

Early opening of sarcolemmal ATP-sensitive potassium channels is not a key step in PKC-mediated cardioprotection.

S. Brennan, M. Sims, R. D. Rainbow, 2015, Journal of molecular and cellular cardiology.

Kir6.2 limits Ca(2+) overload and mitochondrial oscillations of ventricular myocytes in response to metabolic stress.

N. Standen, R. D. Rainbow, D. Lodwick, 2013, American journal of physiology. Heart and circulatory physiology.

Mitochondrial ROS production and subsequent ERK phosphorylation are necessary for temperature preconditioning of isolated ventricular myocytes

D. Lodwick, N. Storey, Y. Bhagatte, 2012, Cell Death and Disease.

Distribution of Kir6.0 and SUR2 ATP-sensitive potassium channel subunits in isolated ventricular myocytes.

H. Singh, C. Lawrence, R. D. Rainbow, 2003, Journal of molecular and cellular cardiology.

dam methylation in the archaebacteria.

J. Harris, W. Grant, J. Almond, 1986, Journal of general microbiology.

Mutagenic DNA repair genes on plasmids from the ‘pre-antibiotic era’

S. Sedgwick, P. Strike, V. Hughes, 1989, Molecular and General Genetics MGG.

Analysis of phenotypic consequences of renin gene polymorphism in Lyon rats

N. Samani, C. Gomez-Sanchez, J. Sassard, 1997, Journal of hypertension.

Distribution of sequences homologous to the impCAB operon of TP110 among bacterial plasmids of different incompatibility groups

P. Strike, D. Lodwick, 1991, Molecular and General Genetics MGG.

Genetic analysis of thermolabile methylenetetrahydrofolate reductase as a risk factor for myocardial infarction.

N. Samani, J. Thompson, D. Martin, 1996, QJM : monthly journal of the Association of Physicians.

Hypertension in the spontaneously hypertensive rat and the sex chromosomes.

N. Samani, M. Kaiser, M. Vincent, 1994, Hypertension.

SUR2A C‐terminal fragments reduce KATP currents and ischaemic tolerance of rat cardiac myocytes

N. Standen, N. Davies, R. D. Rainbow, 2004, The Journal of physiology.

A gene differentially expressed in the kidney of the spontaneously hypertensive rat cosegregates with increased blood pressure.

N. Samani, M. Lathrop, C. Dubay, 1993, The Journal of clinical investigation.

Sulfonylurea receptors regulate the channel pore in ATP-sensitive potassium channels via an intersubunit salt bridge.

Hussein N. Rubaiy, G. W. Vuister, Hussein N Rubaiy, 2014, The Biochemical journal.

Proximal C-terminal domain of sulphonylurea receptor 2A interacts with pore-forming Kir6 subunits in KATP channels.

N. Davies, P. Watson, Harprit Singh, 2004, The Biochemical journal.

DNA sequence analysis of the imp UV protection and mutation operon of the plasmid TP110: identification of a third gene.

D. Owen, P. Strike, D. Lodwick, 1990, Nucleic acids research.

Chromosomal assignment of the human SA gene to 16p13.11 and demonstration of its expression in the kidney.

N. Samani, C. G. See, D. Callen, 1994, Biochemical and biophysical research communications.

The rat SA gene shows genotype-dependent tissue-specific expression.

N. Samani, M. Kaiser, D. Lodwick, 1994, Clinical science.

Differential regulation of ventricular adrenomedullin and atrial natriuretic peptide gene expression in pressure and volume overload in the rat.

N. Samani, C. Aalkjaer, M. Kaiser, 1998, Clinical science.

Kir6.2-D323 and SUR2A-Q1336: an intersubunit interaction pairing for allosteric information transfer in the KATP channel complex

Hussein N. Rubaiy, Hussein N Rubaiy, S. Brennan, 2020, The Biochemical journal.

Syntaxin-1A Actions on Sulfonylurea Receptor 2A Can Block Acidic pH-induced Cardiac KATP Channel Activation*

Y. Leung, A. Tinker, Youhou Kang, 2006, Journal of Biological Chemistry.

Receptor double-trouble in preeclampsia

D. Lodwick, 2001, Nature Medicine.

Molecular analysis of the subtype‐selective inhibition of cloned KATP channels by PNU‐37883A

T. Kamishima, D. Lodwick, J. Quayle, 2004, British journal of pharmacology.

Glycoprotein IIIa polymorphism and risk of myocardial infarction.

N. Samani, D. Lodwick, 1997, Cardiovascular research.

Failure of the heat-shock protein 70 locus to cosegregate with blood pressure in spontaneously hypertensive rat x Wistar—Kyoto rat cross

N. Samani, J. Sassard, M. Vincent, 1993, Journal of Hypertension.

Analysis of quantitative trait loci for blood pressure on rat chromosomes 2 and 13. Age-related differences in effect.

M. Bihoreau, N. Samani, D. Gauguier, 1996, Hypertension.

Phenotype and Genotype Comparison of Hereditary Hypertriglyceridemic (hHTG) and Brown‐Norway (BN) Rats Identification of Quantitative Trait Loci (QTLs) for the Insulin Resistance Syndrome a

N. Samani, D. Gašperíková, P. Kovacs, 1997, Annals of the New York Academy of Sciences.

Exon repetition in mRNA.

N. Samani, I. Eperon, N J Samani, 1999, Proceedings of the National Academy of Sciences of the United States of America.

Analysis of the role of angiotensinogen in spontaneous hypertension.

N. Samani, M. Kaiser, F. Cumin, 1995, Hypertension.

Angiotensin II increases vascular permeability factor gene expression by human vascular smooth muscle cells.

B. Williams, D. Lodwick, B. Gallacher, 1995, Hypertension.

Plasmid genes Affecting DNA Repair and Mutation

P. Strike, D. Lodwick, DAVID LODWICK, 1987, Journal of Cell Science.

Functional complementation between chromosomal and plasmid mutagenic DNA repair genes in bacteria

S. Sedgwick, P. Strike, D. Lodwick, 1991, Molecular and General Genetics MGG.

Dimorphism in the P2Y1 ADP Receptor Gene Is Associated With Increased Platelet Activation Response to ADP

N. Samani, J. Thompson, A. Goodall, 2004, Arteriosclerosis, thrombosis, and vascular biology.

ms1, a novel stress‐responsive, muscle‐specific gene that is up‐regulated in the early stages of pressure overload‐induced left ventricular hypertrophy

N. Samani, G. Brooks, D. Lodwick, 2002, FEBS letters.

Apolipoprotein E polymorphism does not predict risk of restenosis after coronary angioplasty.

N. Samani, J. Thompson, A. Gershlick, 1996, Atherosclerosis.

Nucleotide sequence of the 16S ribosomal RNA gene from the haloalkaliphilic archaeon (Archaebacterium) Natronobacterium magadii, and the phylogeny of halobacteria

W. Grant, J. Almond, John Walker, 1991 .

Molecular biology of the renin cascade in hypertension Report of a Meeting of Physicians and Scientists, University of Leicester

J. D. Swales, P. Weston, H. Thurston, 1994, The Lancet.

Vascular Endothelial Growth Factor and Microvascular Permeability

D. Bates, D. Lodwick, B. Williams, 1999, Microcirculation.

Corrigendum to: ms1, a novel stress‐responsive, muscle‐specific gene that is up‐regulated in the early stages of pressure overload‐induced left ventricular hypertrophy (FEBS 26169)

Gavin Brooks, Harin Mahadeva, David Lodwick, 2002 .

Genetic analysis of the SA and Na+/K+‐ATPase α1 genes in the Milan hypertensive rat

N. Samani, L. Zagato, G. Casari, 1998, Journal of hypertension.

The SA gene: predisposition to hypertension and renal function in man.

N. Samani, G. Watt, S. Harrap, 1995, Clinical science.

Increased expression of the SA gene in the kidney of the spontaneously hypertensive rat is localized to the proximal tubule

N. Samani, K. West, D. Lodwick, 1994, Journal of hypertension.

A major polymorphism in the rat SA gene caused by the insertion of a LINE element

N. Samani, D. Lodwick, S. Frantz, 1996, Mammalian Genome.

Kallikrein gene ‘knock-down’ by small interfering RNA transfection induces a profibrotic phenotype in rat mesangial cells

I. Pawluczyk, K. Harris, D. Lodwick, 2008, Journal of hypertension.

Kallikrein gene ‘ knock-down ’ by siRNA transfection induces a pro-fibrotic phenotype in rat mesangial cells

I. Pawluczyk, K. Harris, D. Lodwick, 2009 .

Resistance to activated protein C and risk of premature myocardial infarction

N. Samani, Daniel Martin, D. Lodwick, 1994, The Lancet.

Kir6.2 limits Ca(2+) overload and mitochondrial oscillations of ventricular myocytes in response to metabolic stress.

N. Standen, R. D. Rainbow, D. Lodwick, 2013, American journal of physiology. Heart and circulatory physiology.

Sulfonylurea receptors regulate the channel pore in ATP-sensitive potassium channels via an intersubunit salt bridge.

Hussein N. Rubaiy, G. W. Vuister, R. D. Rainbow, 2014, The Biochemical journal.