D. Xu

发表

Effect of local and systemic burn microenvironment on neutrophil activation as assessed by complement receptor expression and morphology.

E. Deitch, R. Specian, Q. Lu, 1990, The Journal of trauma.

Mechanisms of endotoxin-induced intestinal injury in a hyperdynamic model of sepsis.

R. Berg, E. Deitch, D. Xu, 1992, The Journal of trauma.

EFFECT OF ACUTE PHASE AND HEAT SHOCK STRESS ON APOPTOSIS IN INTESTINAL EPITHELIAL CELLS (CACO‐2): 228

E. Deitch, Q. Lu, D. Xu, 1996 .

A time course study of the protective effect of mesenteric lymph duct ligation on hemorrhagic shock-induced pulmonary injury and the toxic effects of lymph from shocked rats on endothelial cell monolayer permeability.

E. Deitch, Q. Lu, Da-zhong Xu, 2001, Surgery.

Bacterial translocation is inhibited in inducible nitric oxide synthase knockout mice after endotoxin challenge but not in a model of bacterial overgrowth.

E. Deitch, Q. Lu, D. Xu, 1997, Archives of surgery.

The female gender protects against pulmonary injury after trauma hemorrhagic shock.

E. Deitch, Q. Lu, D. Xu, 2001, Surgical infections.

Caco-2 and IEC-18 intestinal epithelial cells exert bactericidal activity through an oxidant-dependent pathway.

E. Deitch, P. Kvietys, Y. Haskel, 1995, Shock.

Increase in endotoxin-induced mucosal permeability is related to increased nitric oxide synthase activity using the Ussing chamber.

E. Deitch, D. Xu, S. Mishima, 1999, Critical care medicine.

Factors larger than 100 kd in post-hemorrhagic shock mesenteric lymph are toxic for endothelial cells.

E. Deitch, Q. Lu, D. Xu, 2001, Surgery.

Intestinal cytokine response after gut ischemia: role of gut barrier failure.

G. Regel, M. Grotz, E. Deitch, 1999, Annals of surgery.

Gut-derived mesenteric lymph but not portal blood increases endothelial cell permeability and promotes lung injury after hemorrhagic shock.

L. Magnotti, E. Deitch, J. Upperman, 1998, Annals of surgery.

Calcium and phospholipase A2 appear to be involved in the pathogenesis of hemorrhagic shock-induced mucosal injury and bacterial translocation.

E. Deitch, Q. Lu, D. Xu, 1995, Critical care medicine.

Mesenteric lymph from rats subjected to trauma-hemorrhagic shock are injurious to rat pulmonary microvascular endothelial cells as well as human umbilical vein endothelial cells.

E. Deitch, Q. Lu, D. Xu, 2001, Shock.

Gut-derived mesenteric lymph: a link between burn and lung injury.

L. Magnotti, E. Deitch, Q. Lu, 1999, Archives of surgery.

Oral-TPN-induced bacterial translocation and impaired immune defenses are reversed by refeeding.

R. Berg, E. Deitch, Q. Lu, 1991, Surgery.

Endotoxin-induced mesenteric microvascular changes involve iNOS-derived nitric oxide: results from a study using iNOS knock out mice.

W. Durán, E. Deitch, D. Xu, 2000, Shock.

Bacterial translocation from the gut impairs systemic immunity.

R. Berg, E. Deitch, D. Xu, 1991, Surgery.

Hypoxia combined with Escherichia coli produces irreversible gut mucosal injury characterized by increased intestinal cytokine production and DNA degradation.

L. Magnotti, E. Deitch, M. Condon, 2001, Shock.

Elemental diet-induced bacterial translocation associated with systemic and intestinal immune suppression.

E. Deitch, Q. Lu, D. Xu, 1998, JPEN. Journal of parenteral and enteral nutrition.

Elemental diet-induced immune suppression is caused by both bacterial and dietary factors.

R. Berg, E. Deitch, Da-zhong Xu, 1993, JPEN. Journal of parenteral and enteral nutrition.

Effect of heat shock and endotoxin stress on enterocyte viability apoptosis and function varies based on whether the cells are exposed to heat shock or endotoxin first.

E. Deitch, D. Xu, G. Swank, 1996, Archives of surgery.

Mesenteric lymph duct ligation provides long term protection against hemorrhagic shock-induced lung injury.

L. Magnotti, E. Deitch, J. Sambol, 2000, Shock.

Hemorrhagic shock induced up-regulation of P-selectin expression is mediated by factors in mesenteric lymph and blunted by mesenteric lymph duct interruption.

E. Deitch, J. Sambol, D. Granger, 2001, The Journal of trauma.

The effect of hypoxia/reoxygenation on the cellular function of intestinal epithelial cells.

E. Deitch, Q. Lu, Da-zhong Xu, 1999, The Journal of trauma.

Elemental Diet and IV‐TPN-Induced Bacterial Translocation Is Associated with Loss of Intestinal Mucosal Barrier Function Against Bacteria

Andrew A. Marino, E. Deitch, Q. Lu, 1995, Annals of surgery.

The modulatory role of gut hormones in elemental diet and intravenous total parenteral nutrition-induced bacterial translocation in rats.

E. Deitch, Y. Haskel, Q. Lu, 1994, JPEN. Journal of parenteral and enteral nutrition.

The relationships among nitric oxide production, bacterial translocation, and intestinal injury after endotoxin challenge in vivo.

E. Deitch, Q. Lu, D. Xu, 1998, The Journal of trauma.

THE INTESTINAL CYTOKINE RESPONSE AFTER GUT ISCHEMIA: 5

M. Grotz, E. Deitch, J. Ding, 1995 .

Role of bacterial adherence and the mucus barrier on bacterial translocation: effects of protein malnutrition and endotoxin in rats.

E. Deitch, R. Specian, D. Xu, 1997, Annals of Surgery.

Protein malnutrition alone and in combination with endotoxin impairs systemic and gut-associated immunity.

R. Berg, E. Deitch, R. Specian, 1992, JPEN. Journal of parenteral and enteral nutrition.

Opioids modulate human neutrophil and lymphocyte function: thermal injury alters plasma beta-endorphin levels.

E. Deitch, D. Xu, R. M. Bridges, 1988, Surgery.

Effect of acute-phase and heat-shock stress on apoptosis in intestinal epithelial cells (Caco-2).

E. Deitch, Q. Lu, D. Xu, 1996, Critical care medicine.

Injury-enhanced calcium mobilization in circulating rat neutrophils models human PMN responses.

R. Forsythe, E. Deitch, D. Livingston, 2001, Shock.