Di Feng

Phosphorylation of ACTN4 Leads to Podocyte Vulnerability and Proteinuric Glomerulosclerosis.

Thomas C. Ferrante, Carlos F. Ng, D. Ingber, 2020, Journal of the American Society of Nephrology : JASN.

The role of alpha-actinin-4 in human kidney disease

M. Pollak, C. Dumontier, Di Feng, 2015, Cell & Bioscience.

Disease-causing mutation in α-actinin-4 promotes podocyte detachment through maladaptation to periodic stretch

R. Krishnan, J. Notbohm, E. Del Gado, 2018, Proceedings of the National Academy of Sciences.

Functional Validation of an Alpha-Actinin-4 Mutation as a Potential Cause of an Aggressive Presentation of Adolescent Focal Segmental Glomerulosclerosis: Implications for Genetic Testing

R. Krishnan, M. Pollak, G. Birrane, 2016, PloS one.

Phosphorylation of key podocyte proteins and the association with proteinuric kidney disease.

Di Feng, 2020, American journal of physiology. Renal physiology.

Mechanical challenges and cytoskeletal impairments in focal segmental glomerulosclerosis.

M. Pollak, C. Dumontier, Di Feng, 2018, American journal of physiology. Renal physiology.

The role of alpha-actinin-4 in human kidney disease

M. Pollak, C. Dumontier, Di Feng, 2015, Cell & Bioscience.

The role of alpha-actinin-4 in human kidney disease

M. Pollak, C. Dumontier, Di Feng, 2015, Cell & Bioscience.