Shujun Liu

发表

Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain

Masaki Tanaka, S. Waxman, J. A. Black, 2004, Pain.

Dendritic spine remodeling following early and late Rac1 inhibition after spinal cord injury: evidence for a pain biomarker.

S. Waxman, L. Bangalore, Shujun Liu, 2016, Journal of neurophysiology.

Dynamics of sodium channel Nav1.5 expression in astrocytes in mouse models of multiple sclerosis

S. Waxman, J. A. Black, Shujun Liu, 2014, Neuroreport.

Exacerbation of experimental autoimmune encephalomyelitis after withdrawal of phenytoin and carbamazepine

S. Waxman, J. A. Black, Shujun Liu, 2007, Annals of neurology.

A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons.

A. M. Rush, S. Dib-Hajj, S. Waxman, 2006, Proceedings of the National Academy of Sciences of the United States of America.

Functional profiles of SCN9A variants in dorsal root ganglion neurons and superior cervical ganglion neurons correlate with autonomic symptoms in small fibre neuropathy.

S. Dib-Hajj, S. Waxman, G. Lauria, 2012, Brain : a journal of neurology.

Long-term protection of central axons with phenytoin in monophasic and chronic-relapsing EAE.

Stephen G Waxman, S. Waxman, J. A. Black, 2006, Brain : a journal of neurology.

The fates of internalized NaV1.7 channels in sensory neurons: Retrograde cotransport with other ion channels, axon-specific recycling, and degradation

S. Dib-Hajj, S. Waxman, F. Dib-Hajj, 2022, The Journal of biological chemistry.

Dendritic spine dysgenesis contributes to hyperreflexia after spinal cord injury.

S. Waxman, Shujun Liu, A. Tan, 2015, Journal of neurophysiology.

Contactin Associates with Sodium Channel Nav1.3 in Native Tissues and Increases Channel Density at the Cell Surface

A. M. Rush, S. Dib-Hajj, S. Waxman, 2004, The Journal of Neuroscience.

A painful neuropathy-associated Nav1.7 mutant leads to time-dependent degeneration of small-diameter axons associated with intracellular Ca2+ dysregulation and decrease in ATP levels

S. Waxman, G. Lauria, J. Hoeijmakers, 2016, Molecular pain.

Contributions of NaV1.8 and NaV1.9 to excitability in human induced pluripotent stem-cell derived somatosensory neurons

S. Waxman, M. Estacion, F. Dib-Hajj, 2021, Scientific Reports.

KCNQ variants and pain modulation: a missense variant in Kv7.3 contributes to pain resilience

S. Dib-Hajj, S. Waxman, M. Estacion, 2021, Brain communications.

Resilience to Pain: A Peripheral Component Identified Using Induced Pluripotent Stem Cells and Dynamic Clamp

S. Dib-Hajj, S. Waxman, F. Dib-Hajj, 2018, The Journal of Neuroscience.

Sodium channels contribute to microglia/macrophage activation and function in EAE and MS

A. Lo, S. Waxman, J. Newcombe, 2005, Glia.

Sodium channel activity modulates multiple functions in microglia

S. Waxman, Shujun Liu, J. Black, 2009, Glia.

Atypical changes in DRG neuron excitability and complex pain phenotype associated with a Nav1.7 mutation that massively hyperpolarizes activation

S. Dib-Hajj, S. Waxman, S. Walker, 2018, Scientific Reports.

Transection of the Main Corticospinal Tract in Rats Conditioned H-Reflex Increase Persists After

J. Wolpaw, A. English, S. Waxman, 2015 .

Adult Rats Motoneurons of the Sacrocaudal Spinal Cord of Persistent Sodium Currents and Repetitive Firing in

W. Rymer, C. Heckman, S. Waxman, 2015 .

Conserved but not critical: Trafficking and function of NaV1.7 are independent of highly conserved polybasic motifs

S. Dib-Hajj, S. Waxman, F. Dib-Hajj, 2023, Frontiers in Molecular Neuroscience.

Nav1.5 in astrocytes plays a sex‐specific role in clinical outcomes in a mouse model of multiple sclerosis

S. Waxman, O. A. Samad, Shujun Liu, 2018, Glia.

Kv7-specific activators hyperpolarize resting membrane potential and modulate human iPSC-derived sensory neuron excitability

S. Dib-Hajj, S. Waxman, M. Estacion, 2023, Frontiers in Pharmacology.

Slowly Progressive Axonal Degeneration in a Rat Model of Chronic, Nonimmune-Mediated Demyelination

S. Waxman, I. Duncan, A. Compston, 2010, Journal of neuropathology and experimental neurology.

Neuropathy‐associated NaV1.7 variant I228M impairs integrity of dorsal root ganglion neuron axons

S. Waxman, I. Merkies, C. Faber, 2013, Annals of neurology.

Paclitaxel increases axonal localization and vesicular trafficking of Nav1.7

S. Dib-Hajj, S. Waxman, F. Dib-Hajj, 2021, Brain : a journal of neurology.

Building sensory axons: Delivery and distribution of NaV1.7 channels and effects of inflammatory mediators

S. Dib-Hajj, S. Waxman, F. Dib-Hajj, 2019, Science Advances.

Contribution of sodium channels to lamellipodial protrusion and Rac1 and ERK1/2 activation in ATP‐stimulated microglia

S. Waxman, M. Estacion, H. Ahn, 2014, Glia.

Burn injury-induced mechanical allodynia is maintained by Rac1-regulated dendritic spine dysgenesis

S. Waxman, O. A. Samad, Shujun Liu, 2013, Experimental Neurology.

RatsFiring and Spasticity in Chronic Spinal Role of Persistent Sodium and Calcium Currents in

M. Gorassini, S. Waxman, F. Calabro, 2015 .

Ca2+ toxicity due to reverse Na+/Ca2+ exchange contributes to degeneration of neurites of DRG neurons induced by a neuropathy-associated Nav1.7 mutation.

S. Waxman, J. A. Black, G. Lauria, 2015, Journal of neurophysiology.

Long-term protection of central axons with phenytoin in monophasic and chronic-relapsing EAE. Commentary

S. Waxman, B. C. Hains, Kenneth J. Smith, 2006 .

Molecular Contactin Associates with Sodium Channel Nav 1 . 3 in Native Tissues and Increases Channel Density at the Cell Surface

S. Dib-Hajj, S. Waxman, L. Tyrrell, 2004 .

Ih current stabilizes excitability in rodent DRG neurons and reverses hyperexcitability in a nociceptive neuron model of inherited neuropathic pain

Shujun Liu, D. Vasylyev, S. G. Waxman, 2023, The Journal of physiology.

Nav1.7 as a chondrocyte regulator and therapeutic target for osteoarthritis

A. Hettinghouse, Wenhuo Hu, M. Attur, 2024, Nature.

Compartment-specific regulation of NaV1.7 in sensory neurons after acute exposure to TNF-α

S. Dib-Hajj, Dong-hyun Kim, Shujun Liu, 2024, Cell reports.