Robert M. Weiss

发表

Transforming Growth Factor-α Acts in Hypothalamic Paraventricular Nucleus to Upregulate ERK1/2 Signaling and Expression of Sympathoexcitatory Mediators in Heart Failure Rats

R. Felder, Yang Yu, Ethan Chen, 2021, Neuroscience.

Gene Therapy With the N-Terminus of Junctophilin-2 Improves Heart Failure in Mice

Chad E. Grueter, D. Hall, Jinxi Wang, 2022, Circulation research.

Hydrogel cultures reveal Transient Receptor Potential Vanilloid 4 regulation of myofibroblast activation and proliferation in valvular interstitial cells

Brian A. Aguado, K. Anseth, M. E. Schroeder, 2022, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

(cid:1) -Adrenergic receptor antagonists ameliorate myocyte T-tubule remodeling following myocardial infarction

Mark E. Anderson, W. Kutschke, F. Miller, 2020 .

Extracellular matrix stiffness controls cardiac valve myofibroblast activation through epigenetic remodeling

Brian A. Aguado, L. Leinwand, K. Anseth, 2022, Bioengineering & translational medicine.

Acute effects of euglycemic‐hyperinsulinemia on myocardial contractility in male mice

E. Abel, W. Kutschke, S. M. Tadinada, 2022, Physiological reports.

Insulin and Insulin-Like Growth Factor 1 Signaling Preserves Sarcomere Integrity in the Adult Heart

Nicholas S. McCarty, P. Galuppo, E. Abel, 2022, Molecular and cellular biology.

Hypersensitivity of Zebrafish htr2b Mutant Embryos to Sertraline Indicates a Role for Serotonin Signaling in Cardiac Development

R. Cornell, Sarah E. Haskell, Bo Hu, 2022, Journal of cardiovascular pharmacology.

Brain Interleukin-17A contributes to neuroinflammation and cardiac dysfunction in rats with myocardial infarction

Yang Yu, Robert M. Weiss, Shun-Guang Wei, 2022, Frontiers in Neuroscience.

MG53 is dispensable for T-tubule maturation but critical for maintaining T-tubule integrity following cardiac stress.

R. Weiss, Long-Sheng Song, Jianjie Ma, 2017, Journal of molecular and cellular cardiology.

An injectable microparticle formulation for the sustained release of the specific MEK inhibitor PD98059: in vitro evaluation and pharmacokinetics

Brittany E. Givens, Youssef W. Naguib, A. Salem, 2020, Drug Delivery and Translational Research.

Genes that Escape X Chromosome Inactivation Modulate Sex Differences in Valve Myofibroblasts

Brian A. Aguado, L. Leinwand, K. Anseth, 2022, Circulation.

Neurohumoral Regulation in Ischemia‐Induced Heart Failure

R. Weiss, J. Francis, A. Johnson, 2001 .

Heart failure induces changes in acid‐sensing ion channels in sensory neurons innervating skeletal muscle

W. Kutschke, Robert M. Weiss, C. Benson, 2015, The Journal of physiology.

Reducing brain TACE activity improves neuroinflammation and cardiac function in heart failure rats

A. Johnson, T. Beltz, B. Xue, 2022, Frontiers in Physiology.

Osteopontin activity modulates sex‐specific calcification in engineered valve tissue mimics

Brian A. Aguado, K. Anseth, M. E. Schroeder, 2022, Bioengineering & translational medicine.

Spontaneous Aortic Regurgitation and Valvular Cardiomyopathy in Mice

L. Leinwand, R. Brooks, Long-Sheng Song, 2015, Arteriosclerosis, thrombosis, and vascular biology.

T-Tubule Remodeling During Transition From Hypertrophy to Heart Failure

Sheng Wei, Heping Cheng, Long-Sheng Song, 2010, Circulation research.

E-C coupling structural protein junctophilin-2 encodes a stress-adaptive transcription regulator

William H. Thiel, William H Thiel, Chad E. Grueter, 2018, Science.

Endoplasmic reticulum stress increases brain MAPK signaling, inflammation and renin-angiotensin system activity and sympathetic nerve activity in heart failure.

R. Felder, Yang Yu, Robert M. Weiss, 2016, American journal of physiology. Heart and circulatory physiology.

β‐Adrenergic receptor antagonists ameliorate myocyte T‐tubule remodeling following myocardial infarction

Mark E. Anderson, R. Weiss, Long-Sheng Song, 2012, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Inhibition of MCU forces extramitochondrial adaptations governing physiological and pathological stress responses in heart

Mark E. Anderson, F. Zhan, Liping Yu, 2015, Proceedings of the National Academy of Sciences.

Cardiac hypertrophy is not a required compensatory response to short-term pressure overload.

R. Kerber, J. Hill, R. Weiss, 2000, Circulation.

Genes That Escape X Chromosome Inactivation Modulate Sex Differences in Valve Myofibroblasts

Brian A. Aguado, L. Leinwand, K. Anseth, 2022, Circulation.

Secreted Factors From Proinflammatory Macrophages Promote an Osteoblast-Like Phenotype in Valvular Interstitial Cells

Brian A. Aguado, K. Anseth, M. E. Schroeder, 2020, Arteriosclerosis, thrombosis, and vascular biology.

MG53 is dispensable for T-tubule maturation but critical for maintaining T-tubule integrity following cardiac stress.

Long-Sheng Song, Jianjie Ma, W. Kutschke, 2017, Journal of molecular and cellular cardiology.