A. Britschgi

发表

The Tumor Suppressor Gene Hypermethylated in Cancer 1 Is Transcriptionally Regulated by E2F1

M. Tschan, D. Leprince, K. Helin, 2009, Molecular Cancer Research.

The Tumor Suppressor Gene Hypermethylated in Cancer 1 Is Transcriptionally Regulated by E 2 F 1

M. Tschan, D. Leprince, K. Helin, 2009 .

Targeting PI3K, HER2 and the IL-8/JAK2 axis in metastatic breast cancer: Which combination makes the whole greater than the sum of its parts?

M. Bentires-Alj, T. Radimerski, A. Britschgi, 2013, Drug resistance updates : reviews and commentaries in antimicrobial and anticancer chemotherapy.

Impact of grassland farming intensification on the breeding ecology of an indicator insectivorous passerine, the Whinchat Saxicola rubetra: Lessons for overall Alpine meadowland management

Reto Spaar, R. Arlettaz, A. Britschgi, 2006 .

A high-throughput drug screen reveals means to differentiate triple-negative breast cancer

G. G. Galli, R. Okamoto, Vincent Unterreiner, 2022, Oncogene.

The death‐associated protein kinase 2 is up‐regulated during normal myeloid differentiation and enhances neutrophil maturation in myeloid leukemic cells

B. Torbett, M. Tschan, M. Fey, 2007, Journal of leukocyte biology.

A high-throughput drug screen reveals means to differentiate triple-negative breast cancer

G. G. Galli, R. Okamoto, Vincent Unterreiner, 2022, Oncogene.

Low Autophagy (ATG) Gene Expression Is Associated with an Immature AML Blast Cell Phenotype and Can Be Restored during AML Differentiation Therapy

B. Torbett, M. Tschan, H. Simon, 2018, Oxidative medicine and cellular longevity.

Cellular Resistance Mechanisms to Targeted Protein Degradation Converge Toward Impairment of the Engaged Ubiquitin Transfer Pathway.

J. Bischoff, Filip Roudnicky, T. Kam-Thong, 2019, ACS chemical biology.

The tumor suppressor gene DAPK2 is induced by the myeloid transcription factors PU.1 and C/EBPα during granulocytic differentiation but repressed by PML‐RARα in APL

B. Torbett, T. Haferlach, G. Behre, 2014, Journal of leukocyte biology.

JAK2/STAT5 inhibition circumvents resistance to PI3K/mTOR blockade: a rationale for cotargeting these pathways in metastatic breast cancer.

H. Brinkhaus, M. Murakami, M. Bentires-Alj, 2012, Cancer cell.

PIK3CAH1047R induces multipotency and multi-lineage mammary tumours

Michael B. Stadler, R. Cardiff, Tim C. Roloff, 2015, Nature.

Epigallocatechin‐3‐gallate induces cell death in acute myeloid leukaemia cells and supports all‐trans retinoic acid‐induced neutrophil differentiation via death‐associated protein kinase 2

M. Tschan, M. Fey, A. Tobler, 2010, British journal of haematology.

The Hippo kinases LATS1 and 2 control human breast cell fate via crosstalk with ERα

Michael B. Stadler, G. Bonamy, A. Orth, 2017, Nature.

Calcium-activated chloride channel ANO1 promotes breast cancer progression by activating EGFR and CAMK signaling

I. Clay, H. Brinkhaus, P. Raman, 2013, Proceedings of the National Academy of Sciences.

Gonadal alterations in male whitefish Coregonus fatioi: no evidence for genetic damage reducing viability in early life stages.

T. Wahli, N. Yoccoz, C. Wedekind, 2008, Diseases of aquatic organisms.

Male dominance linked to size and age, but not to 'good genes' in brown trout (Salmo trutta)

G. Evanno, R. Müller, C. Wedekind, 2007, BMC Evolutionary Biology.

DAPK2 is a novel E2F1/KLF6 target gene involved in their proapoptotic function

M. Tschan, K. Helin, M. Fey, 2008, Oncogene.

Mesenchymal precursor cells maintain the differentiation and proliferation potentials of breast epithelial cells

H. Brinkhaus, E. Cabuy, M. Bentires-Alj, 2014, Breast Cancer Research.

Reply to: Hippo signalling maintains ER expression and ER+ breast cancer growth

J. Couto, M. Bentires-Alj, A. Britschgi, 2021, Nature.

Hippo signalling maintains ER expression and ER+ breast cancer growth

Jianmin Zhang, M. Rosenfeld, Randy L. Johnson, 2021, Nature.

Induction of autophagy is a key component of all-trans-retinoic acid-induced differentiation in leukemia cells and a potential target for pharmacologic modulation.

M. Tschan, N. Mongan, S. McKenna, 2015, Experimental hematology.

Tyrosine phosphatase SHP2 increases cell motility in triple-negative breast cancer through the activation of SRC-family kinases

Michael B. Stadler, N. Aceto, J. Wyckoff, 2014, Oncogene.

Identification of Novel Death-Associated Protein Kinase 2 Interaction Partners by Proteomic Screening Coupled with Bimolecular Fluorescence Complementation

M. Fussenegger, B. Gerrits, M. Tschan, 2015, Molecular and Cellular Biology.

From Degraders to Molecular Glues: New Ways of Breaking Down Disease‐Associated Proteins

A. Britschgi, A. Ricci, Nagel Yvonne Alice, 2021 .

Blocking the Autophagy Gene 5 (ATG5) Impairs ATRA-Induced Myeloid Differentiation, and ATG5 Is Downregulated in AML

M. Tschan, M. Fey, A. Tobler, 2008 .

Abstract LB-205: The calcium activated chloride channel ANO1 promotes breast cancer progression by activating EGFR- and CAMK-signaling.

I. Clay, H. Brinkhaus, P. Raman, 2013 .

Epithelial Protein-Tyrosine Phosphatase 1B Contributes to the Induction of Mammary Tumors by HER2/Neu but Is Not Essential for Tumor Maintenance

B. Neel, N. Aceto, U. Mueller, 2011, Molecular Cancer Research.

PIK3CAH1047R induces multipotency and multi-lineage mammary tumours

Michael B. Stadler, R. Cardiff, Tim C. Roloff, 2015, Nature.

JAK2/STAT5 inhibition circumvents resistance to PI3K/mTOR blockade: a rationale for cotargeting these pathways in metastatic breast cancer.

M. Murakami, M. Bentires-Alj, T. Radimerski, 2012, Cancer cell.

Tyrosine phosphatase SHP2 increases cell motility in triple-negative breast cancer through the activation of SRC-family kinases

Michael B. Stadler, N. Aceto, J. Wyckoff, 2014, Oncogene.

Hippo inactivation feeds tumor-initiating cells

M. Bentires-Alj, S. Duss, A. Britschgi, 2012, Breast Cancer Research.