K. Sakata

发表

Fushi-ka (defective apoptosis) and rheumatic autoimmune diseases: an overview on the regulation of Fas-mediated T cell apoptotic signal transduction

N. Ogawa, N. Talal, H. Dang, 1997 .

Production of an Eosinophil Chemotactic Lymphokine by a Monocyte‐Derived Factor From Patients With

M. Hirashima, K. Muramoto, H. Hayashi, 1988, Journal of leukocyte biology.

Production of fibroblast proliferative cytokines from T lymphocytes stimulated by a B cell lymphoma line and their functional heterogeneity.

M. Hirashima, M. Ando, M. Matsumoto, 1994, Immunology letters.

Galectin-9 accelerates transforming growth factor beta3-induced differentiation of human mesenchymal stem cells to chondrocytes.

T. Niki, M. Hirashima, A. Matsukawa, 2009, Bone.

Beneficial effect of galectin 9 on rheumatoid arthritis by induction of apoptosis of synovial fibroblasts.

T. Niki, V. Kuchroo, A. Yamauchi, 2007, Arthritis and rheumatism.

Fas (CD95)‐transduced signal preferentially stimulates lupus peripheral T lymphocytes

N. Talal, A. Escalante, J. Cheng, 1998, European journal of immunology.

Galectin-9 ameliorates immune complex-induced arthritis by regulating Fc gamma R expression on macrophages.

T. Niki, A. Yamauchi, M. Hirashima, 2009, Clinical immunology.

Combined anti-tumor necrosis factor-α therapy and DMARD therapy in rheumatoid arthritis patients reduces inflammatory gene expression in whole blood compared to DMARD therapy alone

H. Mitsuya, H. Volk, G. Burmester, 2012, Front. Immun..

Neutrophil chemotactic factors produced by a cell line from thyroid carcinoma.

M. Kawakita, K. Takatsuki, M. Takeya, 1992, Cancer research.

Successful induction of severe destructive arthritis by the transfer of in vitro‐activated synovial fluid T cells from patients with rheumatoid arthritis (RA) in severe combined immunodeficient (SCID) mice

M. Tsukano, A. Sakata, K. Sakata, 1996, Clinical and experimental immunology.

[A possible mechanism of interstitial pneumonia during interferon therapy with sho-saiko-to].

K. Takatsuki, K. Okajima, K. Sakata, 1995, Nihon Kyobu Shikkan Gakkai zasshi.

Spontaneous production of eosinophil chemotactic factors by T lymphocytes from patients with subcutaneous angioblastic lymphoid hyperplasia with eosinophilia.

M. Hirashima, H. Tsuda, K. Iyama, 1986, Clinical immunology and immunopathology.

A human T-cell lymphotropic virus type-I carrier with chronic renal failure, aplastic anemia, myelopathy, uveitis, Sjögren's syndrome and panniculitis.

H. Yamasaki, Motomi Osato, M. Kawakita, 1996, Internal medicine.

Inappropriate apoptosis of salivary and lacrimal gland epithelium of immunodeficient NOD-scid mice.

M. Humphreys-Beher, N. Talal, A. Peck, 1998, Clinical and experimental rheumatology.

A HTLV-I carrier who showed various symptoms and antibodies of autoimmune diseases.

K. Takatsuki, M. Mizuki, J. Nojima, 1993, Internal medicine.

Selective Potentiation of Lymphocyte‐Derived Macrophage Chemotactic Factor Release in Complete Freund's Adjuvant‐Treated Guinea Pigs

M. Hirashima, K. Muramoto, H. Hayashi, 1989, Journal of leukocyte biology.

TGF-β1 Null Mutation Leads to CD154 Upregulated Expression in Affected Tissues

J. Letterio, N. Talal, H. Dang, 2001, Inflammation.

Role of Fas/FasL interaction in physiology and pathology: the good and the bad.

N. Talal, A. Sakata, K. Sakata, 1998, Clinical immunology and immunopathology.

Galectin-9 suppresses the generation of Th17, promotes the induction of regulatory T cells, and regulates experimental autoimmune arthritis.

T. Niki, V. Kuchroo, A. Yamauchi, 2008, Clinical immunology.

Bcl-2 family expression in salivary glands from patients with primary Sjögren's syndrome: involvement of Bax in salivary gland destruction.

N. Ogawa, N. Talal, H. Mcguff, 1998, Clinical immunology and immunopathology.

Neutrophil chemotactic factors produced by a cell line from thyroid carcinoma.

M. Kawakita, K. Takatsuki, M. Takeya, 1992, Cancer research.

Difference in requirement of macrophage products for concanavalin A-induced production of eosinophil chemotactic factor and macrophage chemotactic factor from guinea pig lymphocytes in vitro.

M. Hirashima, T. Yoshimura, H. Hayashi, 1984, Journal of immunology.

Primary adult T cell leukemia of bone: two patients with primary bone lesion showing monoclonal integration of HTLV-I proviral DNA.

M. Matsuoka, K. Yamaguchi, K. Takatsuki, 1996, Leukemia.

The regulation of tissue eosinophilia. IV. Purification and properties of eosinophil-directed chemotactic inhibitory factors from complete Freund's adjuvant-treated guinea pigs.

M. Hirashima, H. Hayashi, K. Sakata, 1986, Cellular immunology.

Possible mechanisms for the elevation of serum beta 2-microglobulin levels in adult T-cell leukemia.

H. Tsuda, K. Takatsuki, K. Sakata, 1992, International journal of hematology.

Establishment of a human T-cell line constitutively producing several eosinophil chemotactic lymphokines and their functional heterogeneity on eosinophils.

M. Hirashima, T. Matsumoto, R. Matsumoto, 1992, Lymphokine and cytokine research.

The predictive value of p53, Ki-67 and angiogenetic factors in primary hypopharyngeal carcinoma.

P. Dong, T. Nakashima, K. Mori, 2001, The Kurume medical journal.

The regulation of tissue eosinophilia. V. Induction of lymphocyte-derived eosinophil chemotactic inhibitory factor production by a macrophage product from complete Freund's adjuvant-treated guinea pigs.

M. Hirashima, H. Hayashi, K. Sakata, 1987, Cellular immunology.

Eosinophil Chemotactic Factors From Granuloma of Kimura's Disease, With Special Reference to T Lymphocyte‐Derived Factors

M. Hirashima, K. Iyama, K. Muramoto, 1986, Journal of leukocyte biology.

Isolation of an eosinophil chemotactic lymphokine as a natural mediator for eosinophil chemotaxis from concanavalin A-induced skin reaction sites in guinea-pigs.

M. Hirashima, K. Sakata, K. Tashiro, 1984, Clinical and experimental immunology.

Involvement of retinoic acid‐inducible gene‐I in inflammation of rheumatoid fibroblast‐like synoviocytes

M. Hirashima, T. Nakajima, C. Ohyama, 2008, Clinical and experimental immunology.

Galectin-9 suppresses the generation of Th17, promotes the induction of regulatory T cells, and regulates experimental autoimmune arthritis.

T. Niki, V. Kuchroo, A. Yamauchi, 2008, Clinical immunology.