M. Leduc

ERK1 is dispensable for mouse pancreatic beta cell function but is necessary for glucose-induced full activation of MSK1 and CREB

M. Ravier, J. Pouysségur, J. Tanti, 2017, Diabetologia.

Specific maternal microchimeric T cells targeting fetal antigens in β cells predispose to auto-immune diabetes in the child.

R. Scharfmann, L. Rachdi, K. Khosrotehrani, 2011, Journal of autoimmunity.

Fetal-cell microchimerism, lymphopoiesis, and autoimmunity

K. Khosrotehrani, S. Aractingi, M. Leduc, 2009, Archivum Immunologiae et Therapiae Experimentalis.

Pregnancy allows the transfer and differentiation of fetal lymphoid progenitors into functional T and B cells in mothers

K. Khosrotehrani, M. Oster, S. Uzan, 2008, The Journal of Immunology.

Pregnancy Allows the Transfer and Differentiation of Fetal Lymphoid Progenitors into Functional T and B Cells in Mothers1

K. Khosrotehrani, M. Oster, S. Uzan, 2008, The Journal of Immunology.

Limited functional capacity of microchimeric fetal hematopoietic progenitors acquired by mothers during pregnancy.

K. Khosrotehrani, M. Oster, S. Aractingi, 2010, Experimental hematology.

Fetal Cell Microchimerism, a Low‐Grade Naturally Occurring Celltherapy

K. Khosrotehrani, S. Aractingi, M. Leduc, 2010 .

β-Arrestin2 plays a key role in the modulation of the pancreatic beta cell mass in mice

M. Ravier, J. Bockaert, S. Dalle, 2014, Diabetologia.