E. Y. Osorio

发表

Transcriptional profiling of the spleen in progressive visceral leishmaniasis reveals mixed expression of type 1 and type 2 cytokine-responsive genes

A. Pekosz, M. Band, Álvaro Hernández, 2014, BMC Immunology.

The Malnutrition-Related Increase in Early Visceralization of Leishmania donovani Is Associated with a Reduced Number of Lymph Node Phagocytes and Altered Conduit System Flow

J. Barnes, B. Travi, P. Melby, 2013, PLoS neglected tropical diseases.

Efficacy of histamine H1 receptor antagonists azelastine and fexofenadine against cutaneous Leishmania major infection

B. Travi, P. Melby, Alex G Peniche, 2020, PLoS neglected tropical diseases.

Pathologic Inflammation in Malnutrition Is Driven by Proinflammatory Intestinal Microbiota, Large Intestine Barrier Dysfunction, and Translocation of Bacterial Lipopolysaccharide

N. Ajami, M. Loeffelholz, I. Ito, 2022, Frontiers in Immunology.

A secondary wave of neutrophil infiltration causes necrosis and ulceration in lesions of experimental American cutaneous leishmaniasis

B. Travi, P. Melby, Diana L. Bonilla, 2017, PloS one.

Inflammatory stimuli alter bone marrow composition and compromise bone health in the malnourished host

Z. Gugala, B. Travi, P. Melby, 2022, Frontiers in Immunology.

Progressive Visceral Leishmaniasis Is Driven by Dominant Parasite-induced STAT6 Activation and STAT6-dependent Host Arginase 1 Expression

C. Byus, B. Travi, P. Melby, 2012, PLoS pathogens.

Growth Factor and Th2 Cytokine Signaling Pathways Converge at STAT6 to Promote Arginase Expression in Progressive Experimental Visceral Leishmaniasis

B. Travi, P. Melby, Omar Saldarriaga, 2014, PLoS pathogens.

In-situ proliferation contributes to the accumulation of myeloid cells in the spleen during progressive experimental visceral leishmaniasis

B. Travi, P. Melby, E. Y. Osorio, 2020, PloS one.

Transcriptional Profiling in Experimental Visceral Leishmaniasis Reveals a Broad Splenic Inflammatory Environment that Conditions Macrophages toward a Disease-Promoting Phenotype

B. Luxon, H. Spratt, B. Travi, 2017, PLoS pathogens.

Splenic CD4+ T Cells in Progressive Visceral Leishmaniasis Show a Mixed Effector-Regulatory Phenotype and Impair Macrophage Effector Function through Inhibitory Receptor Expression

H. Spratt, B. Travi, P. Melby, 2017, PloS one.

Malnutrition-related parasite dissemination from the skin in visceral leishmaniasis is driven by PGE2-mediated amplification of CCR7-related trafficking of infected inflammatory monocytes

B. Travi, P. Melby, L. Soong, 2023, PLoS neglected tropical diseases.

Deficiency of Lymph Node-Resident Dendritic Cells (DCs) and Dysregulation of DC Chemoattractants in a Malnourished Mouse Model of Leishmania donovani Infection

J. Barnes, A. White, B. Travi, 2014, Infection and Immunity.

Transcriptional profiling of the spleen in progressive visceral leishmaniasis reveals mixed expression of type 1 and type 2 cytokine-responsive genes

A. Pekosz, M. Band, Álvaro Hernández, 2014, BMC Immunology.

Clinical, parasitologic, and immunologic evolution in dogs experimentally infected with sand fly-derived Leishmania chagasi promastigotes.

B. Travi, P. Melby, Shu-Hwae Lee, 2009, The American journal of tropical medicine and hygiene.

Comparative Evaluation of Lesion Development, Tissue Damage, and Cytokine Expression in Golden Hamsters (Mesocricetus auratus) Infected by Inocula with Different Leishmania (Viannia) braziliensis Concentrations

C. Pirmez, O. Moreira, J. G. Valverde, 2014, Infection and Immunity.