Q. Zhu

发表

Structural basis for chromosome X-linked agammaglobulinemia: a tyrosine kinase disease.

J. Sowadski, D. Vetrie, M. Vihinen, 1994, Proceedings of the National Academy of Sciences of the United States of America.

Structural basis for pleckstrin homology domain mutations in X-linked agammaglobulinemia.

M. Zvelebil, M. Vihinen, H. Ochs, 1995, Biochemistry.

Wiskott-Aldrich syndrome/X-linked thrombocytopenia: WASP gene mutations, protein expression, and phenotype.

S. Kanner, H. Ochs, A. Aruffo, 1997, Blood.

Inhaled IFN-γ for persistent nontuberculous mycobacterial pulmonary disease due to functional IFN-γ deficiency

H. Ochs, W. Liles, T. Hallstrand, 2004, European Respiratory Journal.

Inhaled IFN-gamma for persistent nontuberculous mycobacterial pulmonary disease due to functional IFN-gamma deficiency.

H. Ochs, T. Hallstrand, W. Liles, 2004, The European respiratory journal.

Serial Profiling of SARS-CoV-2 Antigens and Antibodies in COVID-19 Patient Plasma

T. Gibson, D. Walt, W. Marasco, 2020, medRxiv.

Human single-chain Fv intrabodies counteract in situ huntingtin aggregation in cellular models of Huntington's disease

D. Housman, J. Lecerf, J. Huston, 2001, Proceedings of the National Academy of Sciences of the United States of America.

Extended half-life and elevated steady-state level of a single-chain Fv intrabody are critical for specific intracellular retargeting of its antigen, caspase-7.

D. Danley, W. Marasco, J. Huston, 1999, Journal of immunological methods.

Deletion within the Src homology domain 3 of Bruton's tyrosine kinase resulting in X-linked agammaglobulinemia (XLA)

M. Vihinen, H. Ochs, O. Witte, 1994, Journal of Experimental Medicine.

The Wiskott-Aldrich syndrome and X-linked congenital thrombocytopenia are caused by mutations of the same gene.

U. Francke, J. Derry, H. Ochs, 1995, Blood.

Phase 1 safety and pharmacokinetics studies of BRII-196 and BRII-198, SARS-CoV-2 spike-targeting monoclonal antibodies

Y. Li, C. Li, Y. Zhang, 2021, medRxiv.

Characterization of B-cell lines established from two X-linked severe combined immunodeficiency patients: interleukin-15 binds to the B cells but is not internalized efficiently.

H. Ochs, K. Sugamura, K. Schooley, 1995, Blood.

Unique mutations of Bruton's tyrosine kinase in fourteen unrelated X-linked agammaglobulinemia families.

H. Ochs, J. Winkelstein, S. Chen, 1994, Human molecular genetics.

Fatal hemophagocytic lymphohistiocytosis associated with Epstein-Barr virus infection in a patient with a novel mutation in the signaling lymphocytic activation molecule-associated protein.

H. Ochs, J. Crowe, J. Whitlock, 2003, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.

Maternal-fetal transmission of SIV in macaques: disseminated adenovirus infection in an offspring with congenital SIV infection.

H. Ochs, M. Thouless, C. Tsai, 1991, Journal of medical primatology.

Tyrosine kinase signaling regulates Wiskott-Aldrich syndrome protein function, which is essential for megakaryocyte differentiation.

H. Ochs, T. Takenawa, A. Aruffo, 1997, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

The pleckstrin homology domain of the Wiskott-Aldrich syndrome protein is involved in the organization of actin cytoskeleton.

T. Morio, H. Ochs, T. Takenawa, 1999, Clinical immunology.

Intracellular antibodies (intrabodies) and their therapeutic potential.

W. Marasco, W A Marasco, Q. Zhu, 2008, Handbook of experimental pharmacology.

Regulation of antibody responses: the role of complement and adhesion molecules.

H. Ochs, S. Nonoyama, R. Wedgwood, 1993, Clinical immunology and immunopathology.

Intra-amniotic inoculation of pigtailed macaque (Macaca nemestrina) fetuses with SIV and HIV-1.

H. Ochs, C. Tsai, R. Benveniste, 1993, Journal of medical primatology.