Shuang Zhao

Metabolic activation of 2-hydroxyamino-1-methyl-6-phenylimidazo[4,5-b]pyridine in Syrian hamsters congenic at the N-acetyltransferase 2 (NAT2) locus.

M. Doll, D. Hein, A. Fretland, 2003, Toxicological sciences : an official journal of the Society of Toxicology.

The T341C (Ile114Thr) polymorphism of N-acetyltransferase 2 yields slow acetylator phenotype by enhanced protein degradation.

J. States, M. Doll, D. Hein, 2004, Pharmacogenetics.

Quinoxaline – Induced DNA Adduct Formation and Mutagenesis in DNA Repair – Deficient Chinese Hamster Ovary Cells Expressing Human Cytochrome P 4501 A 1 and Rapid or Slow Acetylator N-Acetyltransferase 2

W. Pierce, J. States, M. Doll, 2007 .

2-Amino-3,8-Dimethylimidazo-[4,5-f]Quinoxaline–Induced DNA Adduct Formation and Mutagenesis in DNA Repair–Deficient Chinese Hamster Ovary Cells Expressing Human Cytochrome P4501A1 and Rapid or Slow Acetylator N-Acetyltransferase 2

W. Pierce, J. States, M. Doll, 2007, Cancer Epidemiology Biomarkers & Prevention.

Functional characterization of single-nucleotide polymorphisms and haplotypes of human N-acetyltransferase 2.

J. States, M. Doll, D. Hein, 2007, Carcinogenesis.

Functional characterization of the A411T (L137F) and G364A (D122N) genetic polymorphisms in human N-acetyltransferase 2

M. Doll, D. Hein, Y. Zang, 2007, Pharmacogenetics and genomics.

2‐amino‐1‐methyl‐6‐phenylimidazo [4,5‐b] pyridine‐induced DNA adducts and genotoxicity in chinese hamster ovary (CHO) cells expressing human CYP1A2 and rapid or slow acetylator N‐acetyltransferase 2

W. Pierce, W. Mcgregor, J. States, 2007, Molecular carcinogenesis.