N. Ostlie

发表

Experimental myasthenia gravis in congenic mice. Sequence mapping and H‐2 restriction of T helper epitopes on the α subunits of Torpedo californica and murine acetylcholine receptors

M. Bellone, Sijin Lei, N. Ostlie, 1991, European journal of immunology.

C 57 BL / 6 Mice Effects on Experimental Myasthenia Gravis or IL-12 Has Different γ Absence of IFN

P. Karachunski, B. Conti-Fine, N. Ostlie, 2000 .

Absence of IFN-γ or IL-12 Has Different Effects on Experimental Myasthenia Gravis in C57BL/6 Mice1

P. Karachunski, B. Conti-Fine, N. Ostlie, 2000, The Journal of Immunology.

Human Bronchial Epithelial and Endothelial Cells Express α7 Nicotinic Acetylcholine Receptors

E. Albuquerque, E. Pereira, A. Maus, 2001 .

Immunopathogenesis of Myasthenia Gravis

M. Milani, B. Diethelm-Okita, B. Conti-Fine, 2003 .

Subcutaneous administration of T-epitope sequences of the acetylcholine receptor prevents experimental myasthenia gravis

R. Garman, P. Karachunski, D. Okita, 1999, Journal of Neuroimmunology.

Adoptive protection from experimental myasthenia gravis with T cells from mice treated nasally with acetylcholine receptor epitopes

J. Lindstrom, P. Karachunski, M. Milani, 2002, Journal of Neuroimmunology.

C57BL/6 mice genetically deficient in IL-12/IL-23 and IFN-γ are susceptible to experimental autoimmune myasthenia gravis, suggesting a pathogenic role of non-Th1 cells.

R. Caspi, M. Milani, D. Okita, 2007, The Journal of Immunology.

Clustering of B and T Epitopes Within Short Sequence Regions of the Nieotinic Acetylcholine Receptor

P. Karachunski, M. Bellone, B. Conti-Fine, 1995, Scandinavian journal of immunology.

The Susceptibility to Experimental Myasthenia Gravis of STAT6−/− and STAT4−/− BALB/c Mice Suggests a Pathogenic Role of Th1 Cells 1

M. Milani, B. Conti-Fine, N. Ostlie, 2004, The Journal of Immunology.

BALB/c Mice Suggests a Pathogenic Role of Th1 Cells

N. Ostlie, 2003 .

CD4+ T and B cells cooperate in the immunoregulation of Experimental Autoimmune Myasthenia Gravis

M. Milani, B. Conti-Fine, N. Ostlie, 2006, Journal of Neuroimmunology.

Transgenic Expression of IL-10 in T Cells Facilitates Development of Experimental Myasthenia Gravis1

M. Kronenberg, P. Karachunski, B. Conti-Fine, 2001, The Journal of Immunology.

Protective Role of Th2 Cells in Mouse Experimental Myasthenia Gravis

P. Karachunski, D. Okita, B. Conti-Fine, 2000 .

Interleukin-4 deficiency facilitates development of experimental myasthenia gravis and precludes its prevention by nasal administration of CD4+ epitope sequences of the acetylcholine receptor

P. Karachunski, D. Okita, B. Conti-Fine, 1999, Journal of Neuroimmunology.

Residues within the α subunit sequence 304-322 of muscle acetylcholine receptor forming autoimmune CD4+ epitopes in BALB/c mice

P. Karachunski, M. Bellone, N. Ostlie, 1994 .

Effect of absorbed and nonabsorbed anti-H-2 antiserum on mouse blastocysts.

C. Warner, N. Ostlie, D. J. Spannaus, 1985, The Journal of experimental zoology.

Myasthenia gravis: effect on antibody binding of conservative substitutions of amino acid residues forming the main immunogenic region of the nicotinic acetylcholine receptor.

J. Lindstrom, N. Ostlie, B. Conti-Tronconi, 1993, Journal of receptor research.

T helper function of CD4+ cells specific for defined epitopes on the acetylcholine receptor in congenic mouse strains.

A. Manfredi, M. Bellone, N. Ostlie, 1992, Journal of autoimmunity.

Nasal Administration of Synthetic Acetylcholine Receptor T Epitopes Affects the Immune Response to the Acetylcholine Receptor and Prevents Experimental Myasthenia Gravis a

P. Karachunski, D. Okita, B. Conti-Fine, 1998, Annals of the New York Academy of Sciences.

C57BL/6 Mice Genetically Deficient in IL-12/IL-23 and IFN-γ Are Susceptible to Experimental Autoimmune Myasthenia Gravis, Suggesting a Pathogenic Role of Non-Th1 Cells1

R. Caspi, M. Milani, D. Okita, 2007, The Journal of Immunology.

The I-Abm12 mutation, which confers resistance to experimental myasthenia gravis, drastically affects the epitope repertoire of murine CD4+ cells sensitized to nicotinic acetylcholine receptor.

M. Bellone, N. Ostlie, B. Conti-Tronconi, 1991, Journal of immunology.

Prevention of experimental myasthenia gravis by nasal administration of synthetic acetylcholine receptor T epitope sequences.

P. Karachunski, D. Okita, B. Conti-Fine, 1997, The Journal of clinical investigation.

Mechanisms by which the I‐ABM12 Mutation Influences Susceptibility to Experimental Myasthenia Gravis: a Study in Homozygous and Heterozygous Mice

P. Karachunski, M. Bellone, B. Conti-Fine, 1995, Scandinavian journal of immunology.

Killing of mouse blastocyst stage embryos by cytotoxic T lymphocytes directed to major histocompatibility complex antigens.

C. Warner, N. Ostlie, M. A. Ewoldsen, 1987, Journal of Immunology.

Absence of IL-4 Facilitates the Development of Chronic Autoimmune Myasthenia Gravis in C57BL/6 Mice1

M. Milani, D. Okita, B. Conti-Fine, 2003, The Journal of Immunology.

Cryptic epitopes on the nicotinic acetylcholine receptor are recognized by autoreactive CD4+ cells.

A. Manfredi, P. Karachunski, M. Bellone, 1993, Journal of immunology.

Epitope Repertoire of Th1 and Th2 Cells Reactive with the Mouse Muscle AChR α Subunit in C57Bl/6 Mice

M. Milani, D. Okita, B. Conti-Fine, 2003, Annals of the New York Academy of Sciences.

in C57BL/6 Mice of Chronic Autoimmune Myasthenia Gravis Absence of IL-4 Facilitates the Development

M. Milani, D. Okita, B. Conti-Fine, 2002 .

Immunization of bm12 Mice with High Doses of Acetylcholine Receptor Overcomes Their Resistance to Experimental Autoimmune Myasthenia Gravis a

P. Karachunski, J. Lindstrom, D. Okita, 1998, Annals of the New York Academy of Sciences.

Preferential pairing of T and B cells for production of antibodies without covalent association of T and B epitopes

P. Karachunski, M. Bellone, N. Ostlie, 1994, European journal of immunology.

T Cells and Cytokines in the Pathogenesis of Acquired Myasthenia Gravis

M. Milani, B. Conti-Fine, N. Ostlie, 2003, Annals of the New York Academy of Sciences.

Absence of IFN-gamma or IL-12 has different effects on experimental myasthenia gravis in C57BL/6 mice.

P. Karachunski, B. Conti-Fine, N. Ostlie, 2000, Journal of immunology.