Anastazia M. Older Aguilar

发表

Mutation at Positively Selected Positions in the Binding Site for HLA-C Shows That KIR2DL1 Is a More Refined but Less Adaptable NK Cell Receptor Than KIR2DL3

P. Parham, P. Norman, L. Abi-Rached, 2012, The Journal of Immunology.

Primate‐specific regulation of natural killer cells

P. Parham, P. Norman, L. Abi-Rached, 2010, Journal of medical primatology.

Humans Differ from Other Hominids in Lacking an Activating NK Cell Receptor That Recognizes the C1 Epitope of MHC Class I

P. Parham, L. Abi-Rached, L. Guethlein, 2010, The Journal of Immunology.

Evolution of Killer Cell Ig-Like Receptor (KIR) Genes: Definition of an Orangutan KIR Haplotype Reveals Expansion of Lineage III KIR Associated with the Emergence of MHC-C1

P. Parham, L. Abi-Rached, L. Guethlein, 2007, The Journal of Immunology.

Coevolution of Killer Cell Ig-Like Receptors with HLA-C To Become the Major Variable Regulators of Human NK Cells

E. Adams, P. Parham, L. Abi-Rached, 2010, The Journal of Immunology.

KIR2DS4 is a product of gene conversion with KIR3DL2 that introduced specificity for HLA-A*11 while diminishing avidity for HLA-C

D. Bushnell, P. Parham, P. Norman, 2009, The Journal of experimental medicine.

Natural variation at position 45 in the D1 domain of lineage III killer cell immunoglobulin-like receptors (KIR) has major effects on the avidity and specificity for MHC class I

P. Parham, L. Abi-Rached, L. Guethlein, 2011, Immunogenetics.

Rhesus macaque KIR bind human MHC class I with broad specificity and recognize HLA-C more effectively than HLA-A and HLA-B

P. Parham, L. Walter, L. Guethlein, 2011, Immunogenetics.

Meeting report: Convening on the influenza human viral challenge model for universal influenza vaccines, Part 1: Value; challenge virus selection; regulatory, industry and ethical considerations; increasing standardization, access and capacity

Diane J Post, P. Roberts, J. Bresee, 2019, Vaccine.