S. Ryan

发表

Expression of an E1A/E7 Chimeric Protein Sensitizes Tumor Cells to Killing by Activated Macrophages but Not NK Cells

K. Morris, Tanya A. Miura, J. Routes, 2004, Journal of Virology.

Dissimilar immunogenicities of human papillomavirus E7 and adenovirus E1A proteins influence primary tumor development.

J. Steinke, H. Li, J. Routes, 2000, Virology.

MHC class I molecules on adenovirus E1A-expressing tumor cells inhibit NK cell killing but not NK cell-mediated tumor rejection.

J. Ryan, M. Nakamura, Mary C. Nakamura, 2001, International immunology.

Adenovirus E1A Oncogene Expression in Tumor Cells Enhances Killing by TNF-Related Apoptosis-Inducing Ligand (TRAIL)1

T. Potter, Tanya A. Miura, J. Routes, 2000, The Journal of Immunology.

Oncogenicity of human papillomavirus- or adenovirus-transformed cells correlates with resistance to lysis by natural killer cells

J. Routes, S. Ryan, 1995, Journal of virology.

CD4(+) T-cells are required for the establishment of maedi-visna virus infection in macrophages but not dendritic cells in vivo.

I. Mcconnell, B. Blacklaws, K. Eriksson, 1999, Virology.

Altered UV resistance and UV mutational spectrum in repair-proficient murine fibroblasts expressing endonuclease V.

D. Kusewitt, T. E. Sherburn, J. Ji, 1998, Mutation research.

Excision repair of pyrimidine dimers in human peripheral blood lymphocytes: comparison between mitogen stimulated and unstimulated cells.

S. Freeman, S. Ryan, 1988, Mutation research.

CREB (cAMP response element binding protein) and C/EBPalpha (CCAAT/enhancer binding protein) are required for the superstimulation of phosphoenolpyruvate carboxykinase gene transcription by adenoviral E1a and cAMP.

D. Klemm, J. Routes, S. Ryan, 2000, The Biochemical journal.

Adenovirus serotype 5 E1A sensitizes tumor cells to NKG2D-dependent NK cell lysis and tumor rejection

L. Lanier, A. Cerwenka, K. Morris, 2005, The Journal of experimental medicine.

In vivo EXPOSURE TO ULTRAVIOLET RADIATION ENHANCES PATHOGENIC EFFECTS OF MURINE LEUKEMIA VIRUS, LP‐BMS, IN MURINE ACQUIRED IMMUNODEFICIENCY SYNDROME

D. Kusewitt, G. Shopp, S. Ryan, 1992, Photochemistry and photobiology.

Adenovirus E1A, Not Human Papillomavirus E7, Sensitizes Tumor Cells to Lysis by Macrophages Through Nitric Oxide- and TNF-α-Dependent Mechanisms Despite Up-Regulation of 70-kDa Heat Shock Protein1

K. Morris, Tanya A. Miura, J. Routes, 2003, The Journal of Immunology.

Adenovirus E1A Proteins Regulate Phosphoenolpyruvate Carboxykinase Gene Transcription through Multiple Mechanisms (*)

D. Klemm, J. Routes, S. Ryan, 1996, The Journal of Biological Chemistry.

In vivo depletion of CD8+ cells does not affect primary maedi visna virus infection in sheep.

I. Mcconnell, B. Blacklaws, K. Eriksson, 1999, Veterinary immunology and immunopathology.

Macrophages Kill Human Papillomavirus Type 16 E6-Expressing Tumor Cells by Tumor Necrosis Factor Alpha- and Nitric Oxide-Dependent Mechanisms

M. Ellison, K. Morris, J. Routes, 2005, Journal of Virology.

The interaction of adenovirus E1A with p300 family members modulates cellular gene expression to reduce tumorigenicity

T. Potter, Tanya A. Miura, J. Routes, 2007, Journal of cellular biochemistry.

FREQUENCY OF ULTRAVIOLET RADIATION‐INDUCED MUTATION AT THE hprt LOCUS IN REPAIR‐PROFICIENT MURINE FIBROBLASTS TRANSFECTED WITH THE denV GENE OF BACTERIOPHAGE T4

D. Kusewitt, S. Ryan, R. Ley, 1993, Photochemistry and photobiology.

Inhibition of IFN-stimulated gene expression and IFN induction of cytolytic resistance to natural killer cell lysis correlate with E1A-p300 binding.

H. Li, S. Bayley, D. Klemm, 1996, Journal of immunology.

Infections with Human Papillomavirus: Are Intralesional Therapies a Promising Approach?

C. Burkhart, S. Ryan, A. Khorbi, 2018 .