M. Dorfman

发表

Hypothalamic inflammation and gliosis in obesity.

J. Thaler, M. Dorfman, 2015, Current opinion in endocrinology, diabetes, and obesity.

Leptin action in the ventromedial hypothalamic nucleus is sufficient, but not necessary, to normalize diabetic hyperglycemia.

G. Taborsky, T. H. Meek, G. Morton, 2013, Endocrinology.

Microglial Inflammatory Signaling Orchestrates the Hypothalamic Immune Response to Dietary Excess and Mediates Obesity Susceptibility.

B. Barres, Mariko L. Bennett, M. Valdearcos, 2017, Cell metabolism.

Astrocyte IKKβ/NF-κB signaling is required for diet-induced obesity and hypothalamic inflammation

J. D. Douglass, J. Thaler, M. Dorfman, 2017, Molecular metabolism.

Evidence That in Uncontrolled Diabetes, Hyperglucagonemia Is Required for Ketosis but Not for Increased Hepatic Glucose Production or Hyperglycemia

G. Taborsky, T. H. Meek, G. Morton, 2015, Diabetes.

Neonatal Exposure to Estradiol Valerate Programs Ovarian Sympathetic Innervation and Follicular Development in the Adult Rat1

A. Paredes, H. Lara, M. Dorfman, 2008, Biology of reproduction.

Microglial inflammatory activation paradoxically improves glucose tolerance during diet-induced obesity

A. Niraula, M. Valdearcos, M. Robblee, 2022, bioRxiv.

CX3CL1 Action on Microglia Protects from Diet-Induced Obesity by Restoring POMC Neuronal Excitability and Melanocortin System Activity Impaired by High-Fat Diet Feeding

A. Barria, J. D. Douglass, J. Thaler, 2021, bioRxiv.

Loss of Ntrk2/Kiss1r signaling in oocytes causes premature ovarian failure.

S. Ojeda, Baoji Xu, G. Dissen, 2014, Endocrinology.

Kisspeptin receptor haplo-insufficiency causes premature ovarian failure despite preserved gonadotropin secretion.

S. Ojeda, G. Dissen, F. Gaytán, 2014, Endocrinology.

Prostaglandin PGE2 receptor EP4 regulates microglial phagocytosis and increases susceptibility to diet-induced obesity

A. Niraula, J. Frey, J. Thaler, 2021, bioRxiv.

Microglial Inflammatory Signaling Orchestrates the Hypothalamic Immune Response to Dietary Excess and Mediates Obesity Susceptibility.

B. Barres, Mariko L. Bennett, M. Valdearcos, 2018, Cell metabolism.

Deletion of Protein Kinase C λ in POMC Neurons Predisposes to Diet-Induced Obesity

M. Schwartz, M. Sajan, R. Farese, 2017, Diabetes.

Chronic Intermittent Cold Stress Activates Ovarian Sympathetic Nerves and Modifies Ovarian Follicular Development in the Rat1

S. Arancibia, H. Lara, M. Dorfman, 2003, Biology of reproduction.

Changes in sympathetic nerve activity of the mammalian ovary during a normal estrous cycle and in polycystic ovary syndrome: Studies on norepinephrine release

A. Mayerhofer, V. D. Ramirez, M. Guimarães, 2002, Microscopy research and technique.

Chronic-Intermittent Cold Stress in Rats Induces Selective Ovarian Insulin Resistance1

E. Stener-Victorin, V. D. Ramirez, H. Lara, 2009, Biology of reproduction.

Using Lentiviral Vectors as Delivery Vehicles for Gene Therapy

S. Ojeda, J. McBride, F. Galimi, 2012 .

Sex differences in microglial CX3CR1 signalling determine obesity susceptibility in mice

T. H. Meek, G. Morton, Thomas H. Meek, 2017, Nature Communications.

Cold Stress Induces Metabolic Activation of Thyrotrophin‐Releasing Hormone‐Synthesising Neurones in the Magnocellular Division of the Hypothalamic Paraventricular Nucleus and Concomitantly Changes Ovarian Sympathetic Activity Parameters

F. Rage, D. Grouselle, S. Arancibia, 2006, Journal of neuroendocrinology.

Excessive ovarian production of nerve growth factor elicits granulosa cell apoptosis by setting in motion a tumor necrosis factor α/stathmin-mediated death signaling pathway.

S. Ojeda, G. Dissen, O. Söder, 2011, Reproduction.

Central injection of fibroblast growth factor 1 induces sustained remission of diabetic hyperglycemia in rodents

R. Bergman, D. Wasserman, M. Schwartz, 2016, Nature Medicine.

NTRK1 and NTRK2 receptors facilitate follicle assembly and early follicular development in the mouse ovary.

S. Ojeda, B. Kerr, A. Paredes, 2009, Reproduction.

Cell and molecular mechanisms behind diet‐induced hypothalamic inflammation and obesity

B. Kerr, M. Dorfman, Y. Ávalos, 2018, Journal of neuroendocrinology.

Glia: silent partners in energy homeostasis and obesity pathogenesis

J. D. Douglass, J. Thaler, M. Dorfman, 2017, Diabetologia.

Cold‐induced Glutamate Release in vivo from the Magnocellular Region of the Paraventricular Nucleus is Involved in Ovarian Sympathetic Activation

F. Rage, D. Grouselle, S. Arancibia, 2010, Journal of neuroendocrinology.

Functional identification of a neurocircuit regulating blood glucose

D. Olson, M. Schwartz, M. Myers, 2016, Proceedings of the National Academy of Sciences.

Hypothalamic Inflammation: Marker or Mechanism of Obesity Pathogenesis?

B. Wisse, M. Schwartz, Stephan J Guyenet, 2013, Diabetes.

DB121605 2629..2634

B. Wisse, M. Schwartz, Stephan J Guyenet, 2013 .

152-OR: Evidence Linking Loss of Hypothalamic Perineuronal Nets to Obesity Pathogenesis

M. Schwartz, Yuanchao Ye, E. Schur, 2022, Diabetes.

Erratum: Kisspeptin receptor haplo-insufficiency causes premature ovarian failure despite preserved gonadotropin secretion (Endocrinology (2014) 155:8 (3088-3097) DOI: 10.1210/en.2014-1110)

S. Ojeda, G. Dissen, F. Gaytán, 2015 .

Neurotrophins acting via TRKB receptors activate the JAGGED1-NOTCH2 cell-cell communication pathway to facilitate early ovarian development.

S. Ojeda, G. Dissen, B. Kerr, 2011, Endocrinology.

Obesity-associated microglial inflammatory activation paradoxically improves glucose tolerance.

A. Niraula, M. Valdearcos, M. Robblee, 2023, Cell metabolism.

Functional identification of a neurocircuit regulating blood glucose

D. Olson, M. Schwartz, M. Myers, 2016, Proceedings of the National Academy of Sciences.

Deletion of Protein Kinase C λ in POMC Neurons Predisposes to Diet-Induced Obesity

M. Schwartz, M. Sajan, R. Farese, 2017, Diabetes.