S. Stanga

发表

BBB damage in aging causes brain iron deposits via astrocyte-neuron crosstalk and Hepc/Fpn1 pathway

A. Roetto, M. Boido, Mariarosa Mezzanotte, 2021, bioRxiv.

Presenilin 2-Dependent Maintenance of Mitochondrial Oxidative Capacity and Morphology

F. Bontemps, L. Bertrand, I. Dewachter, 2017, Front. Physiol..

Drug Screening and Drug Repositioning as Promising Therapeutic Approaches for Spinal Muscular Atrophy Treatment

D. Rasà, M. Boido, S. Stanga, 2020, Frontiers in Pharmacology.

Cachexia, a Systemic Disease beyond Muscle Atrophy

A. Menga, P. Porporato, L. Bindels, 2020, International journal of molecular sciences.

Glycines from the APP GXXXG/GXXXA Transmembrane Motifs Promote Formation of Pathogenic Aβ Oligomers in Cells

S. Constantinescu, I. Dewachter, Steven O. Smith, 2016, Front. Aging Neurosci..

Homeodomain Interacting Protein Kinase 2: A Target for Alzheimer's Beta Amyloid Leading to Misfolded p53 and Inappropriate Cell Survival

M. Memo, G. D’Orazi, L. Nardinocchi, 2010, PloS one.

Deferasirox-Dependent Iron Chelation Enhances Mitochondrial Dysfunction and Restores p53 Signaling by Stabilization of p53 Family Members in Leukemic Cells

S. Ravera, D. Cilloni, C. Panuzzo, 2020, International journal of molecular sciences.

Activation of the Hepcidin-Ferroportin1 pathway in the brain and astrocytic–neuronal crosstalk to counteract iron dyshomeostasis during aging

A. Roetto, M. Boido, Mariarosa Mezzanotte, 2022, Scientific reports.

Characterization of Pterocarpus erinaceus kino extract and its gamma-secretase inhibitory properties.

I. Dewachter, J. Quetin-Leclercq, J. Octave, 2015, Journal of ethnopharmacology.

Deconstruction of Neurotrypsin Reveals a Multi-factorially Regulated Activity Affecting Myotube Formation and Neuronal Excitability

E. D’Angelo, F. Forneris, Anselmo Canciani, 2022, Molecular Neurobiology.

Searching for predictive blood biomarkers: misfolded p53 in mild cognitive impairment.

Giuliano Mazzini, Elena Sinforiani, Marco Racchi, 2012, Current Alzheimer research.

Presenilin-Deficient Neurons and Astrocytes Display Normal Mitochondrial Phenotypes

L. Bertrand, P. Kienlen-Campard, V. Payen, 2020, bioRxiv.

Mitochondrial Dysfunctions: A Red Thread across Neurodegenerative Diseases

A. Vercelli, M. Boido, S. Stanga, 2020, International journal of molecular sciences.

Mitochondria: A Galaxy in the Hematopoietic and Leukemic Stem Cell Universe

D. Cilloni, C. Panuzzo, C. Fava, 2020, International journal of molecular sciences.

The expanding universe of neurotrophic factors: therapeutic potential in aging and age-associated disorders.

C. Lanni, M. Racchi, S. Govoni, 2010, Current pharmaceutical design.

Pharmacogenetics and pharmagenomics, trends in normal and pathological aging studies: focus on p53.

M. Memo, G. Mazzini, E. Sinforiani, 2008, Current pharmaceutical design.

mTORC2 Is Activated under Hypoxia and Could Support Chronic Myeloid Leukemia Stem Cells

D. Cilloni, C. Panuzzo, E. Bracco, 2023, International journal of molecular sciences.

APP‐dependent glial cell line‐derived neurotrophic factor gene expression drives neuromuscular junction formation

G. Vandermeulen, J. Loeffler, N. Zanou, 2016, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Analysis by a highly sensitive split luciferase assay of the regions involved in APP dimerization and its impact on processing

S. Constantinescu, I. Dewachter, Steven O. Smith, 2015, FEBS open bio.

Unfolded p53 in the pathogenesis of Alzheimer's disease: is HIPK2 the link?

G. D’Orazi, D. Uberti, C. Lanni, 2010, Aging.

Specificity of presenilin‐1‐ and presenilin‐2‐dependent γ‐secretases towards substrate processing

Claudia Marinangeli, B. Tasiaux, P. Kienlen-Campard, 2017, Journal of cellular and molecular medicine.

Gamma-Secretase Inhibitor Activity of a Pterocarpus erinaceus Extract

J. Quetin-Leclercq, J. Octave, Claudia Marinangeli, 2013, Neurodegenerative Diseases.

A Role for GDNF and Soluble APP as Biomarkers of Amyotrophic Lateral Sclerosis Pathophysiology

Adrian Ivanoiu, Serena Stanga, A. Ivanoiu, 2018, Front. Neurol..

Amyloid Precursor Protein (APP) Controls the Expression of the Transcriptional Activator Neuronal PAS Domain Protein 4 (NPAS4) and Synaptic GABA Release

P. Gailly, J. Octave, Nathalie Pierrot, 2020, eNeuro.

How to Build and to Protect the Neuromuscular Junction: The Role of the Glial Cell Line-Derived Neurotrophic Factor

P. Kienlen-Campard, M. Boido, S. Stanga, 2020, International journal of molecular sciences.

Unfolded p53 in blood as a predictive signature signature of the transition from mild cognitive impairment to Alzheimer's disease.

M. Memo, G. Mazzini, D. Uberti, 2010, Journal of Alzheimer's disease : JAD.

Amyloid Precursor Protein (APP) controls excitatory/inhibitory synaptic inputs by regulating the transcriptional activator Neuronal PAS Domain Protein 4 (NPAS4)

J. Octave, Nathalie Pierrot, B. Tasiaux, 2018 .

Unfolded p 53 in blood as a predictive signature signature of the transition from mild cognitive impairment to Alzheimer ' s disease . "

G. Mazzini, M. Racchi, S. Stanga, 2019 .

NT-mini, a recombinant tool for the study of Neurotrypsin functionality

F. Forneris, Anselmo Canciani, P. Kienlen-Campard, 2020, bioRxiv.

Recruitment of casein kinase 2 is involved in AbetaPP processing following cholinergic stimulation.

C. Lanni, M. Racchi, S. Govoni, 2010, Journal of Alzheimer's disease : JAD.

Unfolded p53 in the pathogenesis of Alzheimer's disease: is HIPK2 the link?

G. D’Orazi, D. Uberti, C. Lanni, 2010, Aging.

Unfolded p53 in blood as a predictive signature signature of the transition from mild cognitive impairment to Alzheimer's disease.

M. Memo, G. Mazzini, D. Uberti, 2010, Journal of Alzheimer's disease : JAD.