Y. Kanamoto

Retinoic acid stimulation of the sodium/iodide symporter in MCF-7 breast cancer cells is mediated by the insulin growth factor-I/phosphatidylinositol 3-kinase and p38 mitogen-activated protein kinase signaling pathways.

G. Brent, E. Ohashi, T. Kogai, 2008, The Journal of clinical endocrinology and metabolism.

A thyroid-specific far-upstream enhancer in the human sodium/iodide symporter gene requires Pax-8 binding and cyclic adenosine 3',5'-monophosphate response element-like sequence binding proteins for full activity and is differentially regulated in normal and thyroid cancer cells.

G. Brent, J. Hershman, T. Kogai, 2002, Molecular endocrinology.

Inhibitory effects of aryl trans-4-(aminomethyl)cyclohexanecarboxylate and aryl trans-4-(guanidinomethyl)cyclohexanecarboxylate on serine proteases, and their antiallergic effects.

Y. Kanamoto, M. Muramatu, M. Kanemoto, 1982, Hoppe-Seyler's Zeitschrift fur physiologische Chemie.

The Modified Firefly Luciferase Reporter Gene (luc+) but not Renilla Luciferase is Induced by all-trans Retinoic Acid in MCF-7 Breast Cancer Cells

G. Brent, T. Kogai, Y. Kanamoto, 2003, Breast Cancer Research and Treatment.

Differential regulation of sodium/iodide symporter gene expression by nuclear receptor ligands in MCF-7 breast cancer cells.

G. Brent, E. Ohashi, T. Kogai, 2005, Endocrinology.

Systemic Retinoic Acid Treatment Induces Sodium/Iodide Symporter Expression and Radioiodide Uptake in Mouse Breast Cancer Models

F. Moatamed, G. Brent, T. Kogai, 2004, Cancer Research.