I. Duband-Goulet

发表

The checkpoint Saccharomyces cerevisiae Rad9 protein contains a tandem tudor domain that recognizes DNA

B. Alpha-Bazin, S. Zinn-Justin, B. Gilquin, 2007, Nucleic acids research.

Inherited Defects of the ASC-1 Complex in Congenital Neuromuscular Diseases

A. Ferreiro, I. Duband-Goulet, J. Meunier, 2021, International journal of molecular sciences.

ASC‐1 Is a Cell Cycle Regulator Associated with Severe and Mild Forms of Myopathy

S. Coppens, E. Pierce-Hoffman, L. Servais, 2019, Annals of neurology.

Loss of a DNA binding site within the tail of prelamin A contributes to altered heterochromatin anchorage by progerin

E. Delbarre, B. Buendia, H. Worman, 2010, FEBS letters.

Chromatin reconstitution on small DNA rings. III. Histone H5 dependence of DNA supercoiling in the nucleosome.

Y. Zivanovic, P. Schultz, P. Oudet, 1990, Journal of molecular biology.

LBR, a chromatin and lamin binding protein from the inner nuclear membrane, is proteolyzed at late stages of apoptosis.

B. Buendia, J. Courvalin, I. Duband-Goulet, 1998, Journal of cell science.

The transcription coactivator ASC-1 is a regulator of skeletal myogenesis, and its deficiency causes a novel form of congenital muscle disease.

B. Buendia, J. Rendu, O. Dubourg, 2016, Human molecular genetics.

Inhibition of multicellular development switches cell death of Dictyostelium discoideum towards mammalian-like unicellular apoptosis.

P. Petit, J. Tissier, J. Ameisen, 2001, European journal of cell biology.

Lamin A/C-promoter interactions specify chromatin state–dependent transcription outcomes

E. Delbarre, B. Buendia, P. Collas, 2013, Genome Research.

Inner nuclear membrane protein LBR preferentially interacts with DNA secondary structures and nucleosomal linker.

J. Courvalin, I. Duband-Goulet, 2000, Biochemistry.

The carboxyl-terminal region common to lamins A and C contains a DNA binding domain.

S. Zinn-Justin, H. Worman, J. Courvalin, 2003, Biochemistry.

The R439C mutation in LMNA causes lamin oligomerization and susceptibility to oxidative stress

S. Zinn-Justin, J. Broers, P. Steijlen, 2009, Journal of cellular and molecular medicine.

Lamin ChIP from Chromatin Prepared by Micrococcal Nuclease Digestion.

I. Duband-Goulet, 2016, Methods in molecular biology.

The p.R482W substitution in A-type lamins deregulates SREBP1 activity in Dunnigan-type familial partial lipodystrophy.

Alice Barateau, N. Briand, C. Vigouroux, 2015, Human molecular genetics.

Distinct features of lamin A-interacting chromatin domains mapped by ChIP-sequencing from sonicated or micrococcal nuclease-digested chromatin

B. Buendia, P. Collas, Eivind G. Lund, 2015, Nucleus.

The Carboxyl-terminal Nucleoplasmic Region of MAN1 Exhibits a DNA Binding Winged Helix Domain*

S. Zinn-Justin, B. Gilquin, H. Worman, 2006, Journal of Biological Chemistry.

Human mismatch repair protein MSH6 contains a PWWP domain that targets double stranded DNA.

S. Zinn-Justin, B. Gilquin, I. Callebaut, 2008, Biochemistry.

Chromatin reconstitution on small DNA rings. IV. DNA supercoiling and nucleosome sequence preference.

S. Douc-Rasy, I. Duband-Goulet, A. Prunell, 1992, Journal of molecular biology.

Subcellular localization of SREBP1 depends on its interaction with the C-terminal region of wild-type and disease related A-type lamins.

B. Buendia, P. Roussel, S. Zinn-Justin, 2011, Experimental cell research.

Methods for chromatin assembly and remodeling.

A. Hamiche, Khalid Ouararhni, I. Duband-Goulet, 2004, Methods.

Octamer displacement and redistribution in transcription of single nucleosomes.

M. O'Donohue, A. Hamiche, I. Duband-Goulet, 1994, Nucleic Acids Research.

Human mismatch repair protein MSH6 contains a PWWP domain that targets double stranded DNA.

S. Zinn-Justin, B. Gilquin, I. Callebaut, 2008, Biochemistry.

The transcription coactivator ASC-1 is a regulator of skeletal myogenesis, and its deficiency causes a novel form of congenital muscle disease.

B. Buendia, J. Rendu, O. Dubourg, 2016, Human molecular genetics.