J. Hu

发表

An artifact associated with using trypan blue exclusion to measure effects of amyloid beta on neuron viability.

E. El-Fakahany, J. Hu, E. El‐Fakahany, 1994, Life sciences.

beta-Amyloid 25-35 activates nitric oxide synthase in a neuronal clone.

E. El-Fakahany, J. Hu, 1993, Neuroreport.

Role of Intercellular and Intracellular Communication by Nitric Oxide in Coupling of Muscarinic Receptors to Activation of Guanylate Cyclase in Neuronal Cells

E. El-Fakahany, J. Hu, Jingru Hu, 1993, Journal of neurochemistry.

Complex allosteric modulation of cardiac muscarinic receptors by protamine: potential model for putative endogenous ligands.

C. Forray, E. El-Fakahany, J. Hu, 1992, Molecular pharmacology.

Modulation by certain conserved aspartate residues of the allosteric interaction of gallamine at the m1 muscarinic receptor.

E. El-Fakahany, N. H. Lee, J. Hu, 1992, The Journal of pharmacology and experimental therapeutics.

Allosteric interaction of dynorphin and myelin basic protein with muscarinic receptors.

E. El-Fakahany, J. Hu, J. Hu, 1993, Pharmacology.

Agonist‐induced down‐regulation of m1 muscarinic receptors and reduction of their mRNA level in a transfected cell line

C. Forray, E. El-Fakahany, J. Hu, 1990, FEBS letters.

Intricate regulation of nitric oxide synthesis in neurons.

E. El-Fakahany, J. Hu, Jingru Hu, 1996, Cellular signalling.

p-Fluoro-hexahydro-sila-difenidol exhibits poor selectivity between M3 and M1 muscarinic receptors.

E. El-Fakahany, J. Hu, Y. Meng, 1990, Membrane biochemistry.

Effects of agonist efficacy on desensitization of phosphoinositide hydrolysis mediated by m1 and m3 muscarinic receptors expressed in Chinese hamster ovary cells.

E. El-Fakahany, J. Hu, S. Z. Wang, 1991, The Journal of pharmacology and experimental therapeutics.

An arginine residue conserved in most G protein-coupled receptors is essential for the function of the m1 muscarinic receptor.

E. El-Fakahany, J. Hu, Shou-Zhen Wang, 1994, Molecular pharmacology.

Human Ca(2+) receptor extracellular domain. Analysis of function of lobe I loop deletion mutants.

P. J. Steinbach, A. Spiegel, P. Goldsmith, 2001, The Journal of biological chemistry.

Effects of a calcimimetic compound and naturally activating mutations on the human Ca2+ receptor and on Ca2+ receptor/metabotropic glutamate chimeric receptors.

K. Jacobson, A. Spiegel, Jianxin Hu, 2000, Endocrinology.

Cardioselective derivatives of 2,2-diphenyl-2-ethylthioacetate do not discriminate between m2 and m3 muscarinic receptors expressed in CHO cells.

S. Scapecchi, F. Gualtieri, E. El-Fakahany, 1995, Pharmacology.