E. Pásztor

发表

Myofilament protein carbonylation contributes to the contractile dysfunction in the infarcted LV region of mouse hearts.

I. Édes, Á. Balogh, Z. Papp, 2014, Cardiovascular research.

Mistyping of Angiotensinogen M235T Alleles

I. Édes, Z. Papp, A. Tóth, 2006, Hypertension Research.

High-fat diet-induced reduction in nitric oxide-dependent arteriolar dilation in rats: role of xanthine oxidase-derived superoxide anion.

I. Édes, A. Koller, Z. Papp, 2006, American journal of physiology. Heart and circulatory physiology.

Increased Cyclooxygenase-2 Expression and Prostaglandin-Mediated Dilation in Coronary Arterioles of Patients With Diabetes Mellitus

T. Fülöp, I. Édes, A. Koller, 2006, Circulation research.

High-fat diet-induced obesity leads to increased NO sensitivity of rat coronary arterioles: role of soluble guanylate cyclase activation.

A. Feher, I. Édes, A. Koller, 2008, American journal of physiology. Heart and circulatory physiology.

Inhibitory effect of hypoxic condition on acetylcholine release is partly due to the effect of adenosine released from the tissue

B. Kiss, B. Sperlágh, E. Vizi, 1990, Brain Research Bulletin.

Cell-to-cell variability in troponin I phosphorylation in a porcine model of pacing-induced heart failure

V. Lionetti, F. Recchia, I. Édes, 2012, Basic Research in Cardiology.

Myeloperoxidase impairs the contractile function in isolated human cardiomyocytes.

A. Borbély, I. Édes, Á. Balogh, 2015, Free radical biology & medicine.

Protein Kinase C Contributes to the Maintenance of Contractile Force in Human Ventricular Cardiomyocytes*

A. Borbély, I. Édes, Á. Balogh, 2009, Journal of Biological Chemistry.

Heme-induced contractile dysfunction in human cardiomyocytes caused by oxidant damage to thick filament proteins.

J. Eaton, I. Édes, É. Csősz, 2015, Free radical biology & medicine.