N. Vadrot

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A Novel Lamin A Mutant Responsible for Congenital Muscular Dystrophy Causes Distinct Abnormalities of the Cell Nucleus

Alice Barateau, C. Vigouroux, B. Buendia, 2017, PloS one.

Protein kinase PKC delta and c‐Abl are required for mitochondrial apoptosis induction by genotoxic stress in the absence of p53, p73 and Fas receptor

D. Pessayre, G. Feldmann, A. Bringuier, 2006, FEBS letters.

Tacrine inhibits topoisomerases and DNA synthesis to cause mitochondrial DNA depletion and apoptosis in mouse liver

D. Pessayre, B. Fromenty, A. Sutton, 2003, Hepatology.

Subliminal Fas stimulation increases the hepatotoxicity of acetaminophen and bromobenzene in mice

D. Pessayre, G. Feldmann, A. Berson, 2004, Hepatology.

Pleiotropic Effects of PI‐3′ Kinase/Akt Signaling in Human Hepatoma Cell Proliferation and Drug‐Induced Apoptosis

Guillaume Fallot, A. Groyer, C. Alexia, 2006, Annals of the New York Academy of Sciences.

The beneficial effect of myostatin deficiency on maximal muscle force and power is attenuated with age

G. Butler-Browne, C. Hourde, A. Bonnieu, 2013, Experimental Gerontology.

Differential responses of proliferative and non-proliferative leukemia cells to oxidative stress

G. Feldmann, M. Foglietti, A. Bringuier, 2005, Free radical research.

Partial Beclin 1 silencing aggravates doxorubicin- and Fas-induced apoptosis in HepG2 cells.

D. Pessayre, D. Bernuau, V. Descatoire, 2006, World journal of gastroenterology.

Cadmium induces mitochondria-dependent apoptosis of normal human hepatocytes

G. Feldmann, F. Conti, A. Bringuier, 2007, Cell Biology and Toxicology.

Mitochondrial DNA Maintenance Is Regulated in Human Hepatoma Cells by Glycogen Synthase Kinase 3β and p53 in Response to Tumor Necrosis Factor α

R. Moreau, A. Mansouri, Nathalie Pilard, 2012, PloS one.

The transcription coactivator ASC-1 is a regulator of skeletal myogenesis, and its deficiency causes a novel form of congenital muscle disease.

B. Buendia, J. Rendu, O. Dubourg, 2016, Human molecular genetics.

Mitochondrial permeability transition as a novel principle of hepatorenal toxicity in vivo

G. Kroemer, P. Boya, H. Vieira, 2002, Apoptosis.

Defective endoplasmic reticulum-mitochondria contacts and bioenergetics in SEPN1-related myopathy

E. Clementi, S. Boncompagni, P. Pinton, 2020, Cell Death & Differentiation.

High hepatic glutathione stores alleviate Fas-induced apoptosis in mice.

D. Pessayre, B. Fromenty, G. Feldmann, 2007, Journal of hepatology.

Potent induction of apoptosis in human hepatoma cell lines by targeted cytotoxic somatostatin analogue AN-238.

D. Pessayre, A. Schally, G. Feldmann, 2005, Journal of hepatology.

Prolonged, but not acute, glutathione depletion promotes Fas‐mediated mitochondrial permeability transition and apoptosis in mice

D. Pessayre, G. Feldmann, A. Moreau, 2001, Hepatology.

Ibuprofen administration attenuates serum TNF-alpha levels, hepatic glutathione depletion, hepatic apoptosis and mouse mortality after Fas stimulation.

D. Pessayre, G. Feldmann, A. Berson, 2008, Toxicology and applied pharmacology.

Tamoxifen Inhibits Topoisomerases, Depletes Mitochondrial DNA, and Triggers Steatosis in Mouse Liver

D. Pessayre, B. Fromenty, G. Feldmann, 2007, Journal of Pharmacology and Experimental Therapeutics.

Chronic Ethanol Consumption Lessens the Gain of Body Weight, Liver Triglycerides, and Diabetes in Obese ob/ob Mice

B. Fromenty, A. Fautrel, M. Robin, 2009, Journal of Pharmacology and Experimental Therapeutics.

Partial leptin deficiency favors diet-induced obesity and related metabolic disorders in mice.

D. Pessayre, B. Fromenty, A. Bado, 2008, American journal of physiology. Endocrinology and metabolism.

Abnormal Cellular Phenotypes Induced by Three TMPO/LAP2 Variants Identified in Men with Cardiomyopathies

F. Chapon, B. Buendia, C. Rooryck, 2023, Cells.

Gene- and exon-expression profiling reveals an extensive LPS-induced response in immune cells in patients with cirrhosis.

R. Moreau, D. Valla, A. Groyer, 2013, Journal of hepatology.

Lipopolysaccharide-induced mitochondrial DNA depletion.

D. Pessayre, R. Moreau, P. Nahon, 2011, Antioxidants & redox signaling.

The p.R482W substitution in A-type lamins deregulates SREBP1 activity in Dunnigan-type familial partial lipodystrophy.

Alice Barateau, N. Briand, C. Vigouroux, 2015, Human molecular genetics.

Inducible Nitric Oxide Synthase (iNOS) Activity Could Be Responsible for Resistance or Sensitivity to IFN-γ-Induced Apoptosis in Several Human Hepatoma Cell Lines

G. Feldmann, A. Bringuier, A. Legrand, 2006 .

Study of the effects of interferon a on several human hepatoma cell lines: analysis of the signalling pathway of the cytokine and of its effects on apoptosis and cell proliferation

G. Feldmann, B. Lardeux, A. Bringuier, 2004, Liver international : official journal of the International Association for the Study of the Liver.

Distinct Fiber Type Signature in Mouse Muscles Expressing a Mutant Lamin A Responsible for Congenital Muscular Dystrophy in a Patient

Alice Barateau, B. Buendia, S. Batonnet-Pichon, 2017, Cells.

Subcellular localization of SREBP1 depends on its interaction with the C-terminal region of wild-type and disease related A-type lamins.

B. Buendia, P. Roussel, S. Zinn-Justin, 2011, Experimental cell research.

Toxicity of alpidem, a peripheral benzodiazepine receptor ligand, but not zolpidem, in rat hepatocytes: role of mitochondrial permeability transition and metabolic activation.

D. Pessayre, A. Sutton, T. Tordjmann, 2001, The Journal of pharmacology and experimental therapeutics.

Sex-Specific Patterns of Diaphragm Phospholipid Content and Remodeling during Aging and in a Model of SELENON-Related Myopathy

A. Ferreiro, Andrew Tri Van Ho, M. Moulin, 2023, Biomedicines.

Inducible nitric oxide synthase (iNOS) activity could be responsible for resistance or sensitivity to IFN-gamma-induced apoptosis in several human hepatoma cell lines.

G. Feldmann, A. Bringuier, A. Legrand, 2006, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.

Mitochondrial CYP2E1 is sufficient to mediate oxidative stress and cytotoxicity induced by ethanol and acetaminophen.

B. Fromenty, M. Robin, V. Descatoire, 2011, Toxicology in vitro : an international journal published in association with BIBRA.

Progesterone induces BRCA1 mRNA decrease, cell cycle alterations and apoptosis in the MCF7 breast cancer cell line.

L. Mandelbrot, G. Feldmann, B. Lardeux, 2005, Anticancer research.

Defective endoplasmic reticulum-mitochondria contacts and bioenergetics in SEPN1-related myopathy

E. Clementi, S. Boncompagni, P. Pinton, 2020, Cell Death & Differentiation.

The transcription coactivator ASC-1 is a regulator of skeletal myogenesis, and its deficiency causes a novel form of congenital muscle disease.

B. Buendia, J. Rendu, O. Dubourg, 2016, Human molecular genetics.