K. Copeland

发表

CD8+ T-cells: function and response to HIV infection.

Naveed Gulzar, N. Gulzar, Karen F T Copeland, 2004, Current HIV research.

Properties of HTLV-I transformed CD8+ T-cells in response to HIV-1 infection.

N. Gulzar, J. Kim, K. Copeland, 2010, Virology.

Infection of CD8+CD45RO+ Memory T-Cells by HIV-1 and Their Proliferative Response

N. Gulzar, G. Harris, J. Sanchez-Dardon, 2008, The open AIDS journal.

Production of CD8+ T cell nonlytic suppressive factors by CD28, CD38, and HLA-DR subpopulations.

A. Badley, K. Rosenthal, N. Hawley-Foss, 2003, AIDS research and human retroviruses.

RANTES production by T cells and CD8-mediated inhibition of human immunodeficiency virus gene expression before initiation of potent antiretroviral therapy predict sustained suppression of viral replication.

K. Rosenthal, F. Smaill, M. Smieja, 2000, The Journal of infectious diseases.

Inhibition of apoptosis in T cells expressing human T cell leukemia virus type I Tax.

J. Heeney, P. Krammer, J. Goudsmit, 1994, AIDS research and human retroviruses.

CD8+ T cell suppressor factors and the control of infection, replication and transcription of human immunodeficiency virus.

K. Copeland, 2001, Archivum immunologiae et therapiae experimentalis.

11 – Recombinant Adenoviruses as Vectors for Mucosal Immunity

K. Rosenthal, K. Copeland, W. S. Gallichan, 1996 .

Inhibition of HIV-1 entry into cells.

K. Copeland, 2006, Recent patents on anti-infective drug discovery.

CD8+ T-cell-mediated suppression of HIV-1 long terminal repeat-driven gene expression is not modulated by the CC chemokines RANTES, macrophage inflammatory protein (MIP)-1 a and MIP-1 b

K. Rosenthal, C. Richards, K. Copeland, 2018 .

CD 8 + T Cell Suppressor Factors and the Control of Infection , Replication and Transcription of Human Immunodeficiency Virus

K. Copeland, 2002 .

Modulation of HIV transcription by CD8+ cells is mediated via multiple elements of the long terminal repeat

D. Maslove, A. Badley, N. Hawley-Foss, 2001, Clinical and experimental immunology.

CD8+ T‐cell‐mediated suppression of HIV‐1 long terminal repeat‐driven gene expression is not modulated by the CC chemokines RANTES, macrophage inflammatory protein (MIP)‐1α and MIP‐1β

K. Rosenthal, C. Richards, K. Copeland, 1997 .

CD8+ T cell‐mediated suppression of HIV long terminal repeat‐driven gene expression is not associated with improved clinical status

K. Rosenthal, K. Copeland, F. Smaill, 1997, AIDS.

Human immunodeficiency virus-1 infection protects against a Tc1-to-Tc2 shift in CD8(+) T cells.

N. Gulzar, K. Copeland, Janina Q. Jiang, 2011, Human immunology.

CD8+ T cell supernatants of HIV type 1-infected individuals have opposite effects on long terminal repeat-mediated transcription in T cells and monocytes.

K. Rosenthal, K. Copeland, J. Leith, 1997, AIDS research and human retroviruses.

Defibrinated bovine plasma inhibits retroviral transcription by blocking p52 activation of the NFkappaB element in the long terminal repeat.

R. Jacobs, K. Copeland, B. Jefferson, 2007, Canadian journal of veterinary research = Revue canadienne de recherche veterinaire.

T helper cell activation and human retroviral pathogenesis.

J. Heeney, K. Copeland, 1996, Microbiological reviews.

Modulation of HIV-1 transcription by cytokines and chemokines.

K. Copeland, 2005, Mini reviews in medicinal chemistry.

Suppression of activation of the human immunodeficiency virus long terminal repeat by CD8+ T cells is not lentivirus specific.

K. Rosenthal, K. Copeland, P. J. McKay, 1995, AIDS research and human retroviruses.

Demethylation of the gene expressing a yolk protein precursor in quail.

K. Copeland, A. M. Gibbins, 1989, Poultry science.

Cytochemical analysis of human T cell leukaemia virus I LTR-regulated beta-galactosidase gene expression using a novel integrated cell system.

J. Heeney, J. Goudsmit, D. Derse, 1993, Journal of virological methods.

Detection of human T-cell leukaemia virus 1 permissive cells using cell lines producing selectable recombinant virions.

J. Heeney, D. Derse, K. Copeland, 1994, Journal of virological methods.

T helper cell activation and human retroviral pathogenesis

J. Heeney, K. Copeland, 1996 .

T cell-derived suppressive activity: evidence of autocrine noncytolytic control of HIV type 1 transcription and replication.

K. Rosenthal, D. Bienzle, C. Richards, 1999, AIDS research and human retroviruses.

Non-lytic control of human immunodeficiency virus replication and transcription by CD8+ T cells

K. Copeland, 2001 .

Calcium-mediated inhibition of phorbol ester and Tax trans-activation of the human T cell leukaemia virus type 1.

J. Heeney, J. Goudsmit, K. Copeland, 1994, The Journal of general virology.

Sensitive gas-liquid chromatographic method for chloramphenicol in animal tissues using electron-capture detection.

W. Black, R. Forster, D. Campbell, 1983, Journal of chromatography.

Comparative efflux of saquinavir, ritonavir and lopinavir from primary human cells.

O. Janneh, R. V. van Heeswijk, K. Copeland, 2010, Drug metabolism letters.

Effects of Highly Active Antiretroviral Therapy and Immune Recovery on CD8+ T-Cell-Mediated Inhibition of HIV-1 Transcription

J. Angel, K. Copeland, Bilge Diker, 2006, Journal of acquired immune deficiency syndromes.

Suppression of the human immunodeficiency virus long terminal repeat by CD8+ T cells is dependent on the NFAT-1 element.

K. Rosenthal, K. Copeland, P. J. McKay, 1996, AIDS research and human retroviruses.

Persistently HIV-1 seronegative Nairobi sex workers are susceptible to in vitro infection.

K. MacDonald, F. Plummer, K. Rosenthal, 2000, The Canadian journal of infectious diseases = Journal canadien des maladies infectieuses.

Proportion of HIV-1 infected CD8+CD4- T lymphocytes in vivo.

N. Gulzar, D. Cameron, K. Copeland, 2008, Current HIV research.

CD8+ T-cell-mediated suppression of HIV-1 long terminal repeat-driven gene expression is not modulated by the CC chemokines RANTES, macrophage inflammatory protein (MIP)-1 alpha and MIP-1 beta.

K. Rosenthal, C. Richards, K. Copeland, 1997, AIDS.

Comparison of the response to T-cell activation by integrated HIV-1 and HTLV-1 LTR-lacZ vectors.

J. Heeney, R. V. van Lier, S. Fiering, 1995, Virology.