A. Pépin

发表

Calcification of human valve interstitial cells is dependent on alkaline phosphatase activity.

F. Dagenais, P. Mathieu, P. Voisine, 2005, The Journal of heart valve disease.

Paradoxical low-flow, low-gradient aortic stenosis despite preserved left ventricular ejection fraction: new insights from weights of operatively excised aortic valves.

P. Pibarot, C. Couture, J. Dumesnil, 2014, European heart journal.

Distribution of SPARC during neovascularisation of degenerative aortic stenosis

P. Pibarot, F. Dagenais, P. Mathieu, 2006, Heart.

Expression of bone-regulatory proteins in human valve allografts

P. Pibarot, F. Dagenais, P. Mathieu, 2006, Heart.

Increased biglycan in aortic valve stenosis leads to the overexpression of phospholipid transfer protein via Toll-like receptor 2.

Y. Bossé, J. Després, P. Pibarot, 2010, The American journal of pathology.

Inhibition of ectonucleotidase with ARL67156 prevents the development of calcific aortic valve disease in warfarin-treated rats.

P. Pibarot, P. Mathieu, P. Moreau, 2012, European journal of pharmacology.

ATP acts as a survival signal and prevents the mineralization of aortic valve.

Y. Bossé, J. Després, P. Pibarot, 2012, Journal of molecular and cellular cardiology.

Oxidized low-density lipoprotein, angiotensin II and increased waist cirumference are associated with valve inflammation in prehypertensive patients with aortic stenosis.

J. Després, P. Pibarot, C. Couture, 2010, International journal of cardiology.

Refining Molecular Pathways Leading to Calcific Aortic Valve Stenosis by Studying Gene Expression Profile of Normal and Calcified Stenotic Human Aortic Valves

Y. Bossé, P. Pibarot, P. Mathieu, 2009, Circulation. Cardiovascular genetics.

317 Mechanical Stress Enhances Aortic Valve Calcification: Implication For Bicuspid Aortic Valve Mineralization

P. Pibarot, Marie-Chloé Boulanger, P. Mathieu, 2012 .

ATP acts as a survival signal and prevents the mineralization of aortic valve.

Y. Bossé, P. Pibarot, M. McKee, 2012, Journal of molecular and cellular cardiology.

Increased biglycan in aortic valve stenosis leads to the overexpression of phospholipid transfer protein via Toll-like receptor 2.

Y. Bossé, P. Pibarot, C. Couture, 2010, The American journal of pathology.