S. A. Francis

Knockdown of occludin expression leads to diverse phenotypic alterations in epithelial cells.

R. D. Lynch, A. Yu, E. Schneeberger, 2005, American journal of physiology. Cell physiology.

Cholesterol efflux stimulates metalloproteinase-mediated cleavage of occludin and release of extracellular membrane particles containing its C-terminal fragments.

R. D. Lynch, E. Schneeberger, K. McCarthy, 2010, Experimental cell research.

Alterations in cell cholesterol content modulate Ca2+-induced tight junction assembly by MDCK cells

R. D. Lynch, E. Schneeberger, M. Stankewich, 1996, Lipids.

Cholesterol depletion alters detergent-specific solubility profiles of selected tight junction proteins and the phosphorylation of occludin.

C. Thiele, E. Schneeberger, K. McCarthy, 2007, Experimental cell research.

Rapid reduction of MDCK cell cholesterol by methyl-beta-cyclodextrin alters steady state transepithelial electrical resistance.

E. Schneeberger, R. Rogers, J. Lai, 1999, European journal of cell biology.

Inducible expression of claudin-1-myc but not occludin-VSV-G results in aberrant tight junction strand formation in MDCK cells.

E. Schneeberger, R. Rogers, J. Lai, 2000, Journal of cell science.

Identification of a Cytoskeleton-bound Form of Phospholemman with Unique C-Terminal Immunoreactivity

S. Cala, M. L. Ram, C. Kelly, 2004, The Journal of Membrane Biology.