S. Hehner

发表

Sesquiterpene lactones specifically inhibit activation of NF-kappa B by preventing the degradation of I kappa B-alpha and I kappa B-beta.

K. Schulze-Osthoff, W. Dröge, M. Schmitz, 1998, The Journal of biological chemistry.

Hydrogen peroxide-induced apoptosis is CD95-independent, requires the release of mitochondria-derived reactive oxygen species and the activation of NF-kappaB.

M. Ueffing, T. Hofmann, W. Dröge, 1999, Oncogene.

Mixed-Lineage Kinase 3 Delivers CD3/CD28-Derived Signals into the IκB Kinase Complex

T. Hofmann, C. Scheidereit, W. Dröge, 2000, Molecular and Cellular Biology.

CD95‐induced JNK activation signals are transmitted by the death‐inducing signaling complex (DISC), but not by Daxx

T. Hofmann, A. Möller, W. Dröge, 2001, International journal of cancer.

Inhibition of tyrosine phosphatases antagonizes CD95-mediated apoptosis.

T. Hofmann, W. Dröge, M. Schmitz, 1999, European Journal of Biochemistry.

Enhancement of T Cell Receptor Signaling by a Mild Oxidative Shift in the Intracellular Thiol Pool1

K. Schulze-Osthoff, W. Dröge, M. Schmitz, 2000, The Journal of Immunology.

The pro- or anti-apoptotic function of NF-kappaB is determined by the nature of the apoptotic stimulus.

T. Hofmann, W. Dröge, M. Schmitz, 2000, European journal of biochemistry.

Sesquiterpene Lactones Specifically Inhibit Activation of NF-κB by Preventing the Degradation of IκB-α and IκB-β*

K. Schulze-Osthoff, W. Dröge, M. Schmitz, 1998, The Journal of Biological Chemistry.

Cross-talk between steroids and NF-κB: what language?

W. Fiers, G. Haegeman, P. Herrlich, 1998 .

Cross-talk between steroids and NF-kappa B: what language?

W. Fiers, G. Haegeman, P. Herrlich, 1998, TIBS -Trends in Biochemical Sciences. Regular ed.

Protein Kinase C θ Cooperates with Vav1 to Induce JNK Activity in T-cells*

A. Möller, W. Dröge, M. Schmitz, 2001, The Journal of Biological Chemistry.

Vav Synergizes with Protein Kinase CΘ to Mediate IL-4 Gene Expression in Response to CD28 Costimulation in T Cells1

P. Krammer, M. Li‐Weber, W. Dröge, 2000, The Journal of Immunology.

Co‐stimulatory effect of nitric oxide on endothelial NF‐κB implies a physiological self‐amplifying mechanism

T. Hofmann, W. Dröge, M. Schmitz, 1998 .

Tumor Necrosis Factor-α-induced Cell Killing and Activation of Transcription Factor NF-κB Are Uncoupled in L929 Cells*

T. Hofmann, W. Dröge, M. Schmitz, 1998, Journal of Biological Chemistry.

Glutathione is a factor of resistance of Jurkat leukemia cells to nitric oxide‐mediated apoptosis

W. Dröge, R. Breitkreutz, Mariana Bucur, 2000, Journal of cellular biochemistry.

Synergistic Activation of NF-κB by Functional Cooperation between Vav and PKCθ in T Lymphocytes*

W. Dröge, M. Schmitz, S. Hehner, 2000, The Journal of Biological Chemistry.

Caspase-dependent cleavage and inactivation of the Vav1 proto-oncogene product during apoptosis prevents IL-2 transcription

T. Hofmann, W. Dröge, M. Schmitz, 2000, Oncogene.

The Human Papillomavirus Oncoprotein E7 Attenuates NF-κB Activation by Targeting the IκB Kinase Complex*

T. Hofmann, D. Spitkovsky, A. Möller, 2002, The Journal of Biological Chemistry.

B Kinase Complex κ the I B by Targeting κ Parthenolide Inhibits NF-The Antiinflammatory Sesquiterpene Lactone

T. Hofmann, W. Dröge, S. Hehner, 1999 .

The Antiinflammatory Sesquiterpene Lactone Parthenolide Inhibits NF-κB by Targeting the IκB Kinase Complex

T. Hofmann, W. Dröge, M. Schmitz, 1999, The Journal of Immunology.

Tyrosine-phosphorylated Vav1 as a Point of Integration for T-cell Receptor- and CD28-mediated Activation of JNK, p38, and Interleukin-2 Transcription* 210

T. Hofmann, W. Dröge, M. Schmitz, 2000, The Journal of Biological Chemistry.

The human papillomavirus oncoprotein E7 attenuates NF-kappa B activation by targeting the Ikappa B kinase complex.

T. Hofmann, D. Spitkovsky, A. Möller, 2002, The Journal of biological chemistry.

Synergistic activation of NF-kappa B by functional cooperation between vav and PKCtheta in T lymphocytes.

M. Schmitz, S. Hehner, O. Dienz, 2000, Journal of Biological Chemistry.

Tumor necrosis factor-alpha-induced cell killing and activation of transcription factor NF-kappaB are uncoupled in L929 cells.

T. Hofmann, W. Dröge, M. Schmitz, 1998, The Journal of biological chemistry.

The antiinflammatory sesquiterpene lactone parthenolide inhibits NF-kappa B by targeting the I kappa B kinase complex.

T. Hofmann, W. Dröge, M. Schmitz, 1999, Journal of immunology.

Repression of NF-κB impairs HeLa cell proliferation by functional interference with cell cycle checkpoint regulators

Christian Kaltschmidt, Barbara Kaltschmidt, W. Dröge, 1999, Oncogene.

Various glucocorticoids differ in their ability to induce gene expression, apoptosis and to repress NF‐κB‐dependent transcription

T. Hofmann, W. Dröge, S. Bacher, 1998, FEBS letters.

Hydrogen peroxide-induced apoptosis is CD95-independent, requires the release of mitochondria-derived reactive oxygen species and the activation of NF-κB

M. Ueffing, T. Hofmann, W. Dröge, 1999, Oncogene.

Inhibition of tyrosine phosphatases induces apoptosis independent from the CD95 system

T. Hofmann, W. Dröge, M. Schmitz, 1999, Cell Death and Differentiation.