C. Fernández

发表

P38 MAPK protects against TNF-alpha-provoked apoptosis in LNCaP prostatic cancer cells.

M. Ricote, I. García-Tuñón, B. Fraile, 2006, Apoptosis : an international journal on programmed cell death.

P38 MAPK protects against TNF-α-provoked apoptosis in LNCaP prostatic cancer cells

M. Ricote, I. García-Tuñón, B. Fraile, 2006, Apoptosis.

Cholesterol is essential for mitosis progression and its deficiency induces polyploid cell formation.

D. Gómez-Coronado, M. A. Lasunción, C. Fernández, 2004, Experimental cell research.

Inhibition of cholesterol biosynthesis by Delta22-unsaturated phytosterols via competitive inhibition of sterol Delta24-reductase in mammalian cells.

D. Gómez-Coronado, M. A. Lasunción, Y. Suárez, 2002, The Biochemical journal.

Differential effects of catalase on apoptosis induction in human promonocytic cells. Relationships with heat-shock protein expression.

P. Sancho, E. de Blas, C. Fernández, 2003, Molecular pharmacology.

Pharmacological inhibitors of PI3K/Akt potentiate the apoptotic action of the antileukemic drug arsenic trioxide via glutathione depletion and increased peroxide accumulation in myeloid leukemia cells

A. Ramos, P. Sancho, C. Fernández, 2005 .

P38 MAPK protects against TNF-α-provoked apoptosis in LNCaP prostatic cancer cells

M. Ricote, I. García-Tuñón, B. Fraile, 2006, Apoptosis.

p38 MAPK protects against TNF-α-provoked apoptosis in LNCaP prostatic cancer cells

M. Ricote, I. García-Tuñón, B. Fraile, 2006, Apoptosis.

Genistein selectively potentiates arsenic trioxide‐induced apoptosis in human leukemia cells via reactive oxygen species generation and activation of reactive oxygen species‐inducible protein kinases (p38‐MAPK, AMPK)

E. de Blas, C. Fernández, P. Aller, 2008, International journal of cancer.

The role of intracellular oxidation in death induction (apoptosis and necrosis) in human promonocytic cells treated with stress inducers (cadmium, heat, X-rays).

N. Vilaboa, L. García-Bermejo, E. de Blas, 2001, European journal of cell biology.

Pharmacological inhibitors of extracellular signal-regulated protein kinases attenuate the apoptotic action of cisplatin in human myeloid leukemia cells via glutathione-independent reduction in intracellular drug accumulation.

A. Ramos, P. Sancho, M. Palacios, 2005, Biochimica et biophysica acta.

Effect of Glutathione Depletion on Antitumor Drug Toxicity (Apoptosis and Necrosis) in U-937 Human Promonocytic Cells

P. Sancho, E. de Blas, C. Fernández, 2001, The Journal of Biological Chemistry.

Arsenic trioxide sensitizes promonocytic leukemia cells to TNFalpha-induced apoptosis via p38-MAPK-regulated activation of both receptor-mediated and mitochondrial pathways.

A. Ramos, J. Bréard, E. de Blas, 2007, Biochimica et biophysica acta.

Regulation of apoptosis/necrosis execution in cadmium-treated human promonocytic cells under different forms of oxidative stress

A. Ramos, V. Yuste, P. Sancho, 2006, Apoptosis.

Sterol stringency of proliferation and cell cycle progression in human cells.

D. Gómez-Coronado, M. A. Lasunción, Y. Suárez, 2005, Biochimica et biophysica acta.

Dose-dependent effects of lovastatin on cell cycle progression. Distinct requirement of cholesterol and non-sterol mevalonate derivatives.

D. Gómez-Coronado, M. A. Lasunción, Y. Suárez, 2001, Biochimica et biophysica acta.

12-O-Tetradecanoylphorbol-13-acetate May Both Potentiate and Decrease the Generation of Apoptosis by the Antileukemic Agent Arsenic Trioxide in Human Promonocytic Cells

A. Ramos, P. Sancho, E. de Blas, 2004, Journal of Biological Chemistry.

Differential effects of ergosterol and cholesterol on Cdk1 activation and SRE-driven transcription.

D. Gómez-Coronado, M. A. Lasunción, Y. Suárez, 2002, European journal of biochemistry.

The selection between apoptosis and necrosis is differentially regulated in hydrogen peroxide-treated and glutathione-depleted human promonocytic cells

P. Bernardi, P. Sancho, E. D. Blas, 2003, Cell Death and Differentiation.

Effects of distal cholesterol biosynthesis inhibitors on cell proliferation and cell cycle progression Published, JLR Papers in Press, February 1, 2005. DOI 10.1194/jlr.M400407-JLR200

D. Gómez-Coronado, M. A. Lasunción, C. Fernández, 2005, Journal of Lipid Research.

Modulation of the stress response during apoptosis and necrosis induction in cadmium-treated U-937 human promonocytic cells.

N. Vilaboa, E. de Blas, C. Fernández, 2001, Biochimica et biophysica acta.

Characterization of an anandamide degradation system in prostate epithelial PC‐3 cells: synthesis of new transporter inhibitors as tools for this study

M. A. Lasunción, A. M. Sánchez, C. Hillard, 2004, British journal of pharmacology.

Synthesis, cytotoxic activities and proposed mode of binding of a series of bis([(9-oxo-9,10-dihydroacridine-4-carbonyl)amino]alkyl) alkylamines.

M. Braña, G. Domínguez, B. de Pascual-Teresa, 2002, European journal of medicinal chemistry.

Crosstalk of the EphA2 receptor with a serine/threonine phosphatase suppresses the Akt-mTORC1 pathway in cancer cells.

E. Pasquale, Zhan Xiao, Melanie Richter, 2011, Cellular signalling.

Synergistic upregulation of low-density lipoprotein receptor activity by tamoxifen and lovastatin.

Alberto Dávalos, D. Gómez-Coronado, M. A. Lasunción, 2004, Cardiovascular research.

Cancer somatic mutations disrupt functions of the EphA3 receptor tyrosine kinase through multiple mechanisms.

Elena B Pasquale, E. Pasquale, Erika M Lisabeth, 2012, Biochemistry.

Pharmacologic inhibitors of extracellular signal‐regulated kinase (ERKs) and c‐Jun NH2‐terminal kinase (JNK) decrease glutathione content and sensitize human promonocytic leukemia Cells to arsenic trioxide‐induced apoptosis

A. Ramos, M. A. Palacios, E. de Blas, 2006, Journal of cellular physiology.

Pharmacologic inhibitors of PI3K/Akt potentiate the apoptotic action of the antileukemic drug arsenic trioxide via glutathione depletion and increased peroxide accumulation in myeloid leukemia cells.

A. Ramos, P. Sancho, E. de Blas, 2005, Blood.

Pharmacologic inhibitors of PI 3 K / Akt potentiate the apoptotic action of the antileukemic drug arsenic trioxide via glutathione depletion and increased peroxide accumulation in myeloid leukemia cells

A. Ramos, P. Sancho, C. Fernández, 2005 .