A. Kovac

IDO (indolamine 2,3-dioxygenase) expression and function in the CNS.

I. Bechmann, F. Zipp, R. Nitsch, 2003, Advances in experimental medicine and biology.

Macrophage/microglia activation factor expression is restricted to lesion‐associated microglial cells after brain trauma

R. Nitsch, O. Ullrich, A. Diestel, 2006, Glia.

Self-tolerance in the immune privileged CNS: lessons from the entorhinal cortex lesion model.

I. Bechmann, F. Zipp, R. Nitsch, 2003, Journal of neural transmission. Supplementum.

Circulating monocytic cells infiltrate layers of anterograde axonal degeneration where they transform into microglia

U. Dirnagl, I. Bechmann, R. Nitsch, 2005, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

The correlation between severity of paraparesis and reduced density of resident antigen-presenting cells implicates an unknown role for the spinal perivascular macrophages in experimental autoimmune encephalomyelitis in rats

D. Angelov, W. Neiss, A. Kovac, 2003, Journal of Neuroimmunology.

CXCR3-Dependent Microglial Recruitment Is Essential for Dendrite Loss after Brain Lesion

I. Bechmann, H. Kettenmann, H. Boddeke, 2004, The Journal of Neuroscience.

Turnover of Rat Brain Perivascular Cells

U. Dirnagl, T. Horvath, I. Bechmann, 2001, Experimental Neurology.

Immune surveillance of mouse brain perivascular spaces by blood‐borne macrophages

U. Dirnagl, I. Bechmann, R. Nitsch, 2001, The European journal of neuroscience.

Entorhinal Cortex Lesion in the Mouse Induces Transsynaptic Death of Perforant Path Target Neurons

I. Bechmann, R. Nitsch, B. Heimrich, 2004, Brain pathology.