A. Sloan

发表

Discoidin domain receptor 1 activation links extracellular matrix to podocyte lipotoxicity in Alport syndrome

M. Prunotto, H. Al-Ali, R. Fridman, 2020, EBioMedicine.

Angiotensin II contributes to podocyte injury by increasing TRPC6 expression via an NFAT-mediated positive feedback signaling pathway.

R. D. de Boer, G. Navis, J. Wetzels, 2011, The American journal of pathology.

FGF21-FGFR4 signaling in cardiac myocytes promotes concentric cardiac hypertrophy in mouse models of diabetes

A. Fornoni, C. Faul, A. Wende, 2022, Scientific reports.

FGF21-FGFR4 signaling in cardiac myocytes promotes concentric cardiac hypertrophy in mouse models of diabetes

A. Fornoni, C. Faul, A. Wende, 2021, bioRxiv.

APOL1 Risk Variants Affect Podocyte Lipid Homeostasis and Energy Production in Focal Segmental Glomerulosclerosis.

F. Fontanesi, A. Mendez, A. Fornoni, 2021, Human molecular genetics.

The calcineurin–NFAT pathway allows for urokinase receptor-mediated beta3 integrin signaling to cause podocyte injury

Juan Ma, C. Faul, W. Shi, 2012, Journal of molecular medicine.

The calcineurin–NFAT pathway allows for urokinase receptor-mediated beta3 integrin signaling to cause podocyte injury

Juan Ma, C. Faul, W. Shi, 2012, Journal of molecular medicine.

SMPDL3b modulates insulin receptor signaling in diabetic kidney disease

R. Inagi, A. Mendez, T. Yoo, 2019, Nature Communications.

ATP-binding cassette A1 deficiency causes cardiolipin-driven mitochondrial dysfunction in podocytes.

V. Nair, M. Kretzler, R. Nelson, 2019, The Journal of clinical investigation.

FGF23 induces left ventricular hypertrophy.

A. Go, M. Wolf, M. Keane, 2011, The Journal of clinical investigation.

The role of L3T4 in T cell activation: L3T4 may be both an Ia-binding protein and a receptor that transduces a negative signal.

C. Janeway, J. Tite, A. Sloan, 1986, The Journal of molecular and cellular immunology : JMCI.

Activation of Stimulator of IFN Genes (STING) Causes Proteinuria and Contributes to Glomerular Diseases

M. Kretzler, A. Fornoni, S. Eddy, 2022, Journal of the American Society of Nephrology : JASN.

Activation of Cardiac Fibroblast Growth Factor Receptor 4 Causes Left Ventricular Hypertrophy.

A. Ullrich, M. Wolf, J. Stypmann, 2015, Cell metabolism.

COQ6 mutations in human patients produce nephrotic syndrome with sensorineural deafness.

V. Pertegato, Y. Raphael, H. Prokisch, 2011, The Journal of clinical investigation.

Modular Microphysiological System for Modeling of Biologic Barrier Function

A. Fornoni, A. Agarwal, Laura Wubker, 2020, Frontiers in Bioengineering and Biotechnology.

SMPDL3b modulates insulin receptor signaling in diabetic kidney disease

R. Inagi, A. Mendez, T. Yoo, 2019, Nature Communications.

Hydroxypropyl-β-cyclodextrin protects from kidney disease in experimental Alport syndrome and focal segmental glomerulosclerosis.

M. Kretzler, A. Mendez, A. Fornoni, 2018, Kidney international.

Local TNF causes NFATc1-dependent cholesterol-mediated podocyte injury.

C. Forsblom, P. Groop, O. Kretz, 2016, The Journal of clinical investigation.

Erratum to: The calcineurin-NFAT pathway allows for urokinase receptor-mediated beta3 integrin signaling to cause podocyte injury

Juan Ma, C. Faul, W. Shi, 2012, Journal of Molecular Medicine.

FGF21-FGFR4 signaling in cardiac myocytes promotes concentric cardiac hypertrophy in mouse models of diabetes

A. Fornoni, C. Faul, A. Wende, 2022, Scientific Reports.

Compounds targeting OSBPL7 increase ABCA1-dependent cholesterol efflux preserving kidney function in two models of kidney disease

A. Cavalli, M. Prunotto, A. Mendez, 2021, Nature Communications.

The Role of Nuclear Factor of Activated T-Cells in Chronic Kidney Disease

A. Sloan, 2015 .

ATP-binding cassette A1 deficiency causes cardiolipin-driven mitochondrial dysfunction in podocytes.

V. Nair, M. Kretzler, R. Inagi, 2019, The Journal of clinical investigation.