M. Booty

发表

Multiple Inflammatory Cytokines Converge To Regulate CD8+ T Cell Expansion and Function during Tuberculosis

S. Behar, Stephen M. Carpenter, Pushpa Jayaraman, 2016, The Journal of Immunology.

An autoinflammatory disease with deficiency of the interleukin-1-receptor antagonist.

P. Gregersen, P. Babyn, E. Remmers, 2009, The New England journal of medicine.

Inflammatory signaling in human Tuberculosis granulomas is spatially organized

M. Mann, E. Rubin, K. Sharma, 2016, Nature Medicine.

Apoptosis is an innate defense function of macrophages against Mycobacterium tuberculosis

Xiaomin Zhao, S. Behar, H. Gan, 2011, Mucosal Immunology.

Nitric oxide prevents a pathogen permissive granulocytic inflammation during tuberculosis

Xinchun Chen, R. Baker, Andrew J. Olive, 2017, Nature Microbiology.

Microfluidic Squeezing Enables MHC Class I Antigen Presentation by Diverse Immune Cells to Elicit CD8+ T Cell Responses with Antitumor Activity

K. Jensen, U. V. von Andrian, R. Langer, 2022, The Journal of Immunology.

STAT4 and the risk of rheumatoid arthritis and systemic lupus erythematosus.

Wentian Li, Geoffrey Hom, Lars Alfredsson, 2007, The New England journal of medicine.

IL-1 blockade in Schnitzler syndrome: ex vivo findings correlate with clinical remission.

J. G. Ryan, D. Kastner, L. Beck, 2008, The Journal of allergy and clinical immunology.

Antitumor Activity T Cell Responses with + Cells to Elicit CD8 Antigen Presentation by Diverse Immune Microfluidic Squeezing Enables MHC Class I

K. Jensen, R. Langer, D. Yarar, 2022 .

Efferocytosis is an innate antibacterial mechanism.

S. Fortune, S. Behar, Iris Keren, 2012, Cell host & microbe.

Familial Mediterranean fever with a single MEFV mutation: where is the second hit?

S. Holland, E. Remmers, D. Kastner, 2009, Arthritis and rheumatism.

In search of a new paradigm for protective immunity to TB

A. Rothchild, S. Behar, Cláudio Nunes-Alves, 2014, Nature Reviews Microbiology.

IL-21 signaling is essential for optimal host resistance against Mycobacterium tuberculosis infection

G. Beamer, S. Behar, Stephen M. Carpenter, 2016, Scientific Reports.

Orchestration of pulmonary T cell immunity during Mycobacterium tuberculosis infection: immunity interruptus.

D. Barber, S. Behar, Stephen M. Carpenter, 2014, Seminars in immunology.

Human and Murine Clonal CD8+ T Cell Expansions Arise during Tuberculosis Because of TCR Selection

A. Leslie, A. Rothchild, S. Behar, 2015, PLoS pathogens.

A Higher Activation Threshold of Memory CD8+ T Cells Has a Fitness Cost That Is Modified by TCR Affinity during Tuberculosis

S. Way, S. Behar, Stephen M. Carpenter, 2016, PLoS pathogens.

Clinical features and functional significance of the P369S/R408Q variant in pyrin, the familial Mediterranean fever protein

J. G. Ryan, E. Remmers, D. Kastner, 2009, Annals of the rheumatic diseases.

Microarray-based gene expression profiling in patients with cryopyrin-associated periodic syndromes defines a disease-related signature and IL-1-responsive transcripts

J. G. Ryan, D. Kastner, Hong-Wei Sun, 2012, Annals of the rheumatic diseases.

Inflammatory signaling in human Tuberculosis granulomas is spatially organized

M. Mann, E. Rubin, K. Sharma, 2016, Nature Network Boston.

Regulation of Effector CD8+ T Cells During Mycobacterium Tuberculosis Infection

M. Booty, 2015 .

A Higher Activation Threshold of Memory CD 8 + T Cells Has a Fitness Cost That Is Modified by TCR Affinity during Tuberculosis

S. Way, S. Behar, Stephen M. Carpenter, 2016 .

Human and Murine Clonal CD8+ T Cell Expansions Arise during Tuberculosis Because of TCR Selection

A. Leslie, A. Rothchild, S. Behar, 2015, PLoS pathogens.