H. Temple

High Mobility Group Box 1 Release by Cholangiocytes Governs Biliary Atresia Pathogenesis and Correlates With Increases in Afflicted Infants

G. Tiao, T. Jenkins, H. Poling, 2021, Hepatology.

Rhesus rotavirus receptor‐binding site affects high mobility group box 1 release, altering the pathogenesis of experimental biliary atresia

J. Meller, G. Tiao, H. Poling, 2022, Hepatology communications.

HMGB1 release by cholangiocytes governs biliary atresia pathogenesis and correlates with increases in afflicted infants

G. Tiao, H. Poling, Ruchi Bansal, 2021 .

Rotavirus Reassortant–Induced Murine Model of Liver Fibrosis Parallels Human Biliary Atresia

G. Tiao, H. Poling, Ruchi Bansal, 2020, Hepatology.

Innate Immunity and Pathogenesis of Biliary Atresia

G. Tiao, Ruchi Bansal, S. Mohanty, 2020, Frontiers in Immunology.

T-Bet Deficiency Attenuates Bile Duct Injury in Experimental Biliary Atresia

G. Tiao, S. Mohanty, M. McNeal, 2021, Cells.

A Rotavirus-Induced Mouse Model to Study Biliary Atresia and Neonatal Cholestasis.

G. Tiao, S. Mohanty, B. Donnelly, 2019, Methods in molecular biology.