J. Beech

发表

The membrane potential theory of carcinogenesis.

J. Beech, 1989, Medical hypotheses.

Mitosis stimulation near wounds by temporary membrane lesions of increased permeability.

J. Beech, 1991, Medical hypotheses.

Carcinogenesis and initiation of cell cycling by charge-induced membrane clusters may be due to mitogen receptors and Na+/H+ antiports.

J. Beech, 1994, Medical hypotheses.

A theory of carcinogenesis based on an analysis of the effects of carcinogens.

J. Beech, 1987, Medical hypotheses.

The membrane cluster hypothesis of mitogenesis and carcinogenesis.

J. Beech, 1993, Medical hypotheses.

Cell proliferation and carcinogenesis may share a common basis of permeable plasma membrane clusters.

J. Beech, 1992, Medical hypotheses.

Cancer promotion in cells initiated by polycyclic aromatic hydrocarbons and other non-viral carcinogens.

J. Beech, 1991, Medical hypotheses.

Plasma amino acids in normal humans and patients with shock.

R. Cowley, J. Mclaughlin, E. Labrosse, 1967, Surgery, gynecology & obstetrics.

Two stage carcinogenesis by membrane potential changes.

J. Beech, 1989, Medical hypotheses.

Polycyclic aromatic hydrocarbons initiate carcinogenesis by forming permanent membrane lesions of increased permeability. Other non-viral carcinogens may also form similar lesions.

J. Beech, 1991, Medical Hypotheses.