Mark P. Seldon

发表

Heme Oxygenase-1 Modulates the Expression of Adhesion Molecules Associated with Endothelial Cell Activation1

F. Bach, I. Grégoire, P. Berberat, 2004, The Journal of Immunology.

The Antiapoptotic Effect of Heme Oxygenase-1 in Endothelial Cells Involves the Degradation of p38α MAPK Isoform1

I. Grégoire, M. Soares, Gabriela Silva, 2006, The Journal of Immunology.

Heme Oxygenase-1 Inhibits the Expression of Adhesion Molecules Associated with Endothelial Cell Activation via Inhibition of NF-κB RelA Phosphorylation at Serine 2761

J. Anrather, I. Grégoire, M. Soares, 2007, The Journal of Immunology.

Termination of NF-κB activity through a gammaherpesvirus protein that assembles an EC5S ubiquitin-ligase

J. Anrather, M. Soares, Mark P. Seldon, 2009, The EMBO journal.

B RelA Phosphorylation at Serine 276 κ NF-Cell Activation via Inhibition of Adhesion Molecules Associated with Heme Oxygenase-1 Inhibits the Expression of

M. Soares, Gabriela Silva, Mark P. Seldon, 2013 .

Heme Oxygenase-1 Inhibits the Expression of Adhesion Molecules Associated with Endothelial Cell Activation via Inhibition of NF- B RelA Phosphorylation at Serine 276

M. Soares, Gabriela Silva, Mark P. Seldon, 2007 .

Heme oxygenase-1-derived carbon monoxide requires the activation of transcription factor NF-kappa B to protect endothelial cells from tumor necrosis factor-alpha-mediated apoptosis.

F. Bach, E. Tobiasch, P. Berberat, 2002, The Journal of biological chemistry.

Endothelial Cell Activation of Adhesion Molecules Associated with Heme Oxygenase-1 Modulates the Expression

P. Berberat, M. Soares, T. Tsui, 2004 .

Heme Oxygenase-1-derived Carbon Monoxide Requires the Activation of Transcription Factor NF-κB to Protect Endothelial Cells from Tumor Necrosis Factor-α-mediated Apoptosis*

F. Bach, E. Tobiasch, P. Berberat, 2002, The Journal of Biological Chemistry.