K. Stockero

发表

Familial 22q11.2 deletions in DiGeorge/velocardiofacial syndrome are predominantly smaller than the commonly observed 3Mb

R. Ketterling, H. Flynn, A. Adeyinka, 2004, Genetics in Medicine.

Interphase fluorescence in situ hybridization with an IGH probe is important in the evaluation of patients with a clinical diagnosis of chronic lymphocytic leukaemia

T. Shanafelt, G. Dewald, J. Hoyer, 2005, British journal of haematology.

Prospective evaluation of clonal evolution during long-term follow-up of patients with untreated early-stage chronic lymphocytic leukemia.

T. Shanafelt, G. Dewald, S. Geyer, 2006, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.

JAK2(V617F) mutation in myelodysplastic syndrome (MDS) with del(5q) arises in genetically discordant clones.

L. Sokol, A. List, G. Caceres, 2010, Leukemia research.

Metaphase cells with normal G-bands have cryptic interstitial deletions in 13q14 detectable by fluorescence in situ hybridization in B-cell chronic lymphocytic leukemia.

T. Shanafelt, G. Dewald, N. Kay, 2006, Cancer genetics and cytogenetics.

Short communication Loss of TP53 is due to rearrangements involving chromosome region 17p10~p12 in chronic lymphocytic leukemia

T. Shanafelt, E. Thorland, G. Dewald, 2006 .

Preclinical validation of fluorescence in situ hybridization assays for clinical practice

E. Thorland, G. Dewald, R. Ketterling, 2006, Genetics in Medicine.

Loss of TP53 is due to rearrangements involving chromosome region 17p10∼p12 in chronic lymphocytic leukemia

Tait D. Shanafelt, Erik C. Thorland, T. Shanafelt, 2006 .

Frequency, hematopathology, and detection of a new isodicentric variant of deletion 20q.

G. Dewald, P. Nguyen, C. Hanson, 2006, Cancer genetics and cytogenetics.

Efficacy of conventional cytogenetics and FISH for EGR1 to detect deletion 5q in hematological disorders and to assess response to treatment with Lenalidomide.

A. Tefferi, H. Tun, C. Reeder, 2006, Leukemia research.

Frequency of Clonal Evolution by FISH in Untreated, Early Stage Patients with CLL: A Prospective, Longitudinal Study with Long Clinical Follow-Up.

T. Shanafelt, G. Dewald, S. Geyer, 2005 .

Loss of TP53 is due to rearrangements involving chromosome region 17p10 approximately p12 in chronic lymphocytic leukemia.

Daniel L Van Dyke, T. Shanafelt, E. Thorland, 2006, Cancer genetics and cytogenetics.