T. Haliotis

发表

Cellular ras gene activity is required for full neoplastic transformation by the large tumor antigen of SV40.

K. Wood, L. Raptis, H. Brownell, 1997, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

Regulation of natural antibody binding and susceptibility to natural killer cells through Zn++‐inducible ras oncogene expression

D. Tough, T. Haliotis, D. A. Chow, 1992, International journal of cancer.

Cellular ras gene activity is required for full neoplastic transformation by polyomavirus

L. Raptis, A. Krook, M. Corbley, 1991, Journal of virology.

v-Ras and v-Raf block differentiation of transformable C3H10T1/2-derived preadipocytes at lower levels than required for neoplastic transformation.

E. Schaefer, L. Raptis, H. Brownell, 1997, Experimental cell research.

Ras(leu61) blocks differentiation of transformable 3T3 L1 and C3H10T1/2-derived preadipocytes in a dose- and time-dependent manner.

L. Raptis, H. Brownell, R. Narsimhan, 1997, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

Chediak-Higashi gene in humans. I. Impairment of natural - killer function

J. Roder, A. Fauci, J. Ortaldo, 1980, The Journal of experimental medicine.

Mouse models of hereditary hemochromatosis do not develop early liver fibrosis in response to a high fat diet

H. Vali, C. Fillebeen, K. Pantopoulos, 2018, bioRxiv.

Do NK cells play a role in anti-tumor surveillance?

J. Roder, T. Haliotis, 1980, Immunology today.

Ras modulates commitment and maturation of 10T1/2 fibroblasts to adipocytes.

L. Raptis, M. Corbley, S. Anderson, 1992, Biochemistry and cell biology = Biochimie et biologie cellulaire.

Organization of a repetitive human 1.8 kb KpnI sequence localized in the heterochromatin of chromosome 15

J. Roder, B. White, Hungshu Wang, 2005, Chromosoma.

Gastrointestinal stromal tumor with autonomic nerve differentiation and coexistent mantle cell lymphoma involving the appendix.

R. Chetty, K. Rahimi, A. Gologan, 2008, International journal of clinical and experimental pathology.

p21ras and protein kinase C function in distinct and interdependent signaling pathways in C3H 10T1/2 fibroblasts

S. Anderson, A. Krook, D. Denhardt, 1993, Molecular and cellular biology.

Further studies of natural killer cell function in Chediak-Higashi patients.

J. White, J. Roder, D. Kozbor, 1982, Immunology.

Chediak-Higashi gene in humans. II. The selectivity of the defect in natural- killer and antibody-dependent cell-mediated cytotoxicity function

J. Roder, P. Katz, A. Fauci, 1980, The Journal of experimental medicine.

A new immunodeficiency disorder in humans involving NK cells

J. Roder, J. Jett, A. Fauci, 1980, Nature.

The Chediak-Higashi gene in humans. III. Studies on the mechanisms of NK impairment.

J. Roder, J. Werkmeister, S. Helfand, 1983, Clinical and experimental immunology.

A COMPARATIVE ANALYSIS OF THE NK CYTOLYTIC MECHANISM AND REGULATORY GENES1

J. Roder, T. Haliotis, 1980 .

Nerve growth factor receptors of human tumors of neural crest origin: characterization of binding site heterogeneity and alteration by theophylline.

J. Roder, R. Riopelle, T. Haliotis, 1983, Cancer research.

Tumor cell differentiation modulates susceptibility to natural killer cells.

J. Roder, J. Werkmeister, S. Helfand, 1982, Cellular immunology.

Expression of ras oncogene leads to down‐regulation of protein kinase C

W. Trimble, S. Chow, N. Hozumi, 1990, International journal of cancer.

THE CHEDIAK-HIGASHI GENE IN HUMANS CONTROLS NK FUNCTION

J. Roder, T. Haliotis, 1980 .

Disruption of direct 3D telomere-TRF2 interaction through two molecularly disparate mechanisms is a hallmark of primary Hodgkin and Reed-Sternberg cells.

N. Johnson, S. Mai, H. Knecht, 2017, Laboratory investigation; a journal of technical methods and pathology.

Identification and characterization of a transcript for a novel Rac GTPase-activating protein in terminally differentiating 3T3-L1 adipocytes.

C. Mueller, T. Haliotis, E. J. Wooltorton, 1999, DNA and cell biology.

EVIDENCE FOR IN VIVO NK REACTIVITY AGAINST PRIMARY TUMORS

J. Roder, T. Haliotis, D. Dexter, 1982 .

Spontaneous and induced primary oncogenesis in natural killer (nk)‐cell‐deficient beige mutant mice

J. Roder, J. Ball, T. Haliotis, 1985, International journal of cancer.

The effect of target cell differentiation on human natural killer cell activity: a specific defect in target cell binding and early activation events.

J. Roder, J. Werkmeister, S. Helfand, 1982, Journal of immunology.

Selective natural killer resistance in a clone of YAC lymphoma cells.

J. Roder, R. Kerbel, T. Beaumont, 1981, Proceedings of the National Academy of Sciences of the United States of America.

SPECIFICITY OF NATURAL KILLER (NK) CELLS: NATURE OF TARGET CELL STRUCTURES

J. Roder, J. Werkmeister, H. Pross, 1982 .