Y. Kuppumbatti

发表

Cellular retinol-binding protein expression and breast cancer.

I. Bleiweiss, S. Waxman, J. Mandeli, 2000, Journal of the National Cancer Institute.

Epigenetic CRBP downregulation appears to be an evolutionarily conserved (human and mouse) and oncogene-specific phenomenon in breast cancer

S. Nakajo, Y. Kuppumbatti, R. Mira-y-Lopez, 2004, Molecular Cancer.

CRBP suppresses breast cancer cell survival and anchorage-independent growth

S. Nakajo, K. Nakaya, B. Rexer, 2001, Oncogene.

Retinol conversion to retinoic acid is impaired in breast cancer cell lines relative to normal cells

D. Ong, B. Rexer, Y. Jing, 2000 .

Methylation of conserved CpG sites neighboring the beta retinoic acid response element may mediate retinoic acid receptor beta gene silencing in MCF-7 breast cancer cells

Y. Kuppumbatti, Alice Arapshian, R. Mira‐Y‐Lopez, 2000, Oncogene.

Tumor necrosis-factor-alpha (TNF-alpha) induces rapid insertion of Ca2+-permeable alpha-amino-3-hydroxyl-5-methyl-4-isoxazole-propionate (AMPA)/kainate (Ca-A/K) channels in a subset of hippocampal pyramidal neurons.

J. Weiss, H. Yin, Y. Kuppumbatti, 2005, Experimental neurology.

Tumor necrosis-factor-alpha (TNF-α) induces rapid insertion of Ca2+-permeable α-amino-3-hydroxyl-5-methyl-4-isoxazole-propionate (AMPA)/kainate (Ca-A/K) channels in a subset of hippocampal pyramidal neurons

J. Weiss, H. Yin, Y. Kuppumbatti, 2005, Experimental Neurology.

Cellular retinol-binding protein I, a regulator of breast epithelial retinoic acid receptor activity, cell differentiation, and tumorigenicity.

E. Farias, N. Ghyselinck, D. Ong, 2005, Journal of the National Cancer Institute.

Retinol conversion to retinoic acid is impaired in breast cancer cell lines relative to normal cells.

D. Ong, B. Rexer, W. Zheng, 2000, Journal of cellular physiology.