F. Debeer

Human Secretory Phospholipase A2 Mediates Decreased Plasma Levels of HDL Cholesterol and ApoA-I in Response to Inflammation in Human ApoA-I Transgenic Mice

D. Rader, U. Tietge, C. Maugeais, 2002, Arteriosclerosis, thrombosis, and vascular biology.

Genetic control of inflammatory gene induction and NF-kappa B-like transcription factor activation in response to an atherogenic diet in mice.

A. Lusis, A. Fogelman, A. Andalibi, 1993, The Journal of clinical investigation.

Major acute-phase reactant synthesis during chronic inflammation in amyloid-susceptible and -resistant mouse strains

J. Sipe, A. Whitehead, D. Steel, 1991, Inflammation.

Serum amyloid A is not incorporated into HDL during HDL biogenesis[S]

N. Webb, D. Jennings, A. Ji, 2020, Journal of Lipid Research.

The Evolution of a Department of Internal Medicine Under an Integrated Clinical Enterprise Model: The University of Kentucky Experience

D. Moliterno, R. Lofgren, M. Karpf, 2010, Academic medicine : journal of the Association of American Medical Colleges.

Biosynthesis of human acute-phase serum amyloid A protein (A-SAA) in vitro: the roles of mRNA accumulation, poly(A) tail shortening and translational efficiency.

A. Whitehead, J. Rogers, D. Steel, 1993, The Biochemical journal.

Association between serum amyloid A proteins and coronary artery disease: evidence from two distinct arteriosclerotic processes.

J. Rotter, A. Lusis, L. Rothenberg, 1997, Circulation.

A constitutively expressed serum amyloid A protein gene (SAA4) is closely linked to, and shares structural similarities with, an acute-phase serum amyloid A protein gene (SAA2).

A. Whitehead, G. Sellar, D. Steel, 1993, Genomics.

Genetic Control of Inflammatory Gene Induction and NF-KB-like Transcription

A. Lusis, A. Fogelman, A. Andalibi, 2003 .

Serum Amyloid A and Lipoprotein Retention in Murine Models of Atherosclerosis

A. Chait, T. McDonald, R. Leboeuf, 2005, Arteriosclerosis, thrombosis, and vascular biology.