A. Dandapat

发表

Overexpression of TGFβ1 by adeno-associated virus type-2 vector protects myocardium from ischemia–reperfusion injury

J. Mehta, P. Hermonat, H. Chen, 2008, Gene Therapy.

LOX-1 deletion decreases collagen accumulation in atherosclerotic plaque in low-density lipoprotein receptor knockout mice fed a high-cholesterol diet.

J. Mehta, T. Sawamura, F. Romeo, 2008, Cardiovascular research.

Variant cry1Ab entomocidal Bacillus thuringiensis toxin gene facilitates the recovery of an increased number of lepidopteran insect resistant independent rice transformants against yellow stem borer (Scirpophaga incertulus) inflicted damage

Anirban Chakraborty, C. Mandal, S. Gayen, 2013, Journal of Plant Biochemistry and Biotechnology.

Small Concentrations of oxLDL Induce Capillary Tube Formation From Endothelial Cells via LOX-1–Dependent Redox-Sensitive Pathway

J. Mehta, Changping Hu, A. Dandapat, 2007, Arteriosclerosis, thrombosis, and vascular biology.

Dependent Redox-Sensitive Pathway − via LOX-1 Small Concentrations of oxLDL Induce Capillary Tube Formation From Endothelial Cells

J. Mehta, Changping Hu, A. Dandapat, 2007 .

LOX-1 deletion alters signals of myocardial remodeling immediately after ischemia-reperfusion.

J. Mehta, K. Jishage, Hiroshi Suzuki, 2007, Cardiovascular research.

Regulation of TGFβ1-mediated Collagen Formation by LOX-1

J. Mehta, P. Hermonat, Changping Hu, 2008, Journal of Biological Chemistry.

LOX-1 abrogation reduces myocardial ischemia-reperfusion injury in mice.

J. Mehta, K. Jishage, Hiroshi Suzuki, 2008, Journal of molecular and cellular cardiology.

Blockade of hypoxia-reoxygenation-mediated collagen type I expression and MMP activity by overexpression of TGF-beta1 delivered by AAV in mouse cardiomyocytes.

J. Mehta, P. Hermonat, Chang-Ping Hu, 2007, American journal of physiology. Heart and circulatory physiology.

Modulation of ADP-Induced Platelet Activation by Aspirin and Pravastatin: Role of Lectin-Like Oxidized Low-Density Lipoprotein Receptor-1, Nitric Oxide, Oxidative Stress, and Inside-Out Integrin Signaling

J. Mehta, T. Sawamura, Jiawei Chen, 2007, Journal of Pharmacology and Experimental Therapeutics.

Angiotensin II Induces Capillary Formation From Endothelial Cells Via the LOX-1–Dependent Redox-Sensitive Pathway

J. Mehta, Changping Hu, A. Dandapat, 2007, Hypertension.

A functional signal profiling test for identifying a subset of HER2-negative breast cancers with abnormally amplified HER2 signaling activity

Lance G. Laing, C. Lange, I. Macneil, 2016, Oncotarget.

Modulation of Angiotensin II–Mediated Hypertension and Cardiac Remodeling by Lectin-Like Oxidized Low-Density Lipoprotein Receptor-1 Deletion

J. Mehta, K. Jishage, Hiroshi Suzuki, 2008, Hypertension.

Over-expression of angiotensin II type 2 receptor (agtr2) decreases collagen accumulation in atherosclerotic plaque.

J. Mehta, Jiawei Chen, P. Hermonat, 2008, Biochemical and biophysical research communications.

Lectin-Like Oxidized Low-Density Lipoprotein Receptor-1 Deletion Mediated Hypertension and Cardiac Remodeling by − Modulation of Angiotensin II

J. Mehta, K. Jishage, Hiroshi Suzuki, 2008 .

Prospective isolation of human fibroadipogenic progenitors with CD73

M. Kyba, P. Iaizzo, A. Dandapat, 2020, Heliyon.

Expression of the human FSHD-linked DUX4 gene induces neurogenesis during differentiation of murine embryonic stem cells.

M. Kyba, L. Hartweck, D. Bosnakovski, 2013, Stem cells and development.

Deletion of LOX-1 Reduces Atherogenesis in LDLR Knockout Mice Fed High Cholesterol Diet

J. Mehta, K. Jishage, Hiroshi Suzuki, 2007, Circulation research.

Development of a test that measures real-time HER2 signaling function in live breast cancer cell lines and primary cells

Lance G. Laing, C. Lange, I. Macneil, 2017, BMC Cancer.

GSK3β inhibition activates the CDX/HOX pathway and promotes hemogenic endothelial progenitor differentiation from human pluripotent stem cells.

M. Kyba, J. Tolar, M. Toyoda, 2016, Experimental hematology.

The DUX4 homeodomains mediate inhibition of myogenesis and are functionally exchangeable with the Pax7 homeodomain

M. Kyba, R. Perlingeiro, A. Magli, 2017, Journal of Cell Science.

High Frequency Hearing Loss and Hyperactivity in DUX4 Transgenic Mice

M. Kyba, J. Ervasti, A. Bartolomucci, 2016, PloS one.

Dominant lethal pathologies in male mice engineered to contain an X-linked DUX4 transgene.

M. Kyba, R. Perlingeiro, F. Hamra, 2014, Cell reports.

A focal domain of extreme demethylation within D4Z4 in FSHD2

M. Kyba, R. Lemmers, R. Tawil, 2013, Neurology.

Life span and stress resistance of Caenorhabditis elegans are differentially affected by glutathione transferases metabolizing 4-hydroxynon-2-enal

E. Siegel, Sharda P. Singh, P. Zimniak, 2007, Mechanisms of Ageing and Development.

Anirban Chakraborty, C. Mandal, S. Gayen, 2013, Journal of Plant Biochemistry and Biotechnology.

Lifespan extension in hypomorphic daf‐2 mutants of Caenorhabditis elegans is partially mediated by glutathione transferase CeGSTP2‐2

Sharda P. Singh, P. Zimniak, S. Ayyadevara, 2005, Aging cell.

Lifespan and stress resistance of Caenorhabditis elegans are increased by expression of glutathione transferases capable of metabolizing the lipid peroxidation product 4‐hydroxynonenal

Cheryl F. Lichti, E. Liebau, Sharda P. Singh, 2005, Aging cell.

Aspirin inhibits oxidant stress, reduces age-associated functional declines, and extends lifespan of Caenorhabditis elegans.

J. Mehta, M. Khaidakov, Sona Mitra, 2013, Antioxidants & redox signaling.

Mesp1 patterns mesoderm into cardiac, hematopoietic, or skeletal myogenic progenitors in a context-dependent manner.

Daniel J Garry, Michael Kyba, M. Kyba, 2013, Cell Stem Cell.

Overexpression of TGFbeta1 by adeno-associated virus type-2 vector protects myocardium from ischemia-reperfusion injury.

J. Mehta, P. Hermonat, H. Chen, 2008, Gene therapy.

Over-expression of angiotensin II type 2 receptor (agtr2) reduces atherogenesis and modulates LOX-1, endothelial nitric oxide synthase and heme-oxygenase-1 expression.

J. Mehta, Jiawei Chen, P. Hermonat, 2008, Atherosclerosis.