H. Dhillon

发表

Adipocyte-specific glucocorticoid inactivation protects against diet-induced obesity.

J. Flier, J. Seckl, N. Morton, 2005, Diabetes.

Selective inactivation of Socs3 in SF1 neurons improves glucose homeostasis without affecting body weight.

B. Lowell, J. Flier, A. Yoshimura, 2008, Endocrinology.

βAR Signaling Required for Diet-Induced Thermogenesis and Obesity Resistance

B. Lowell, B. Kobilka, A. Bianco, 2002, Science.

The Metabolic and Cardiovascular Effects of Hyperthyroidism Are Largely Independent of β-Adrenergic Stimulation

I. Amende, Ju-feng Wang, A. Hollenberg, 2004 .

Neuropeptide Y Counteracts the Anorectic and Weight Reducing Effects of Ciliary Neurotropic Factor

L. Moldawer, S. Kalra, P. Kalra, 2000, Journal of neuroendocrinology.

PACAP Neurons in the Hypothalamic Ventromedial Nucleus Are Targets of Central Leptin Signaling

D. Bechtold, B. Lowell, S. Luckman, 2009, The Journal of Neuroscience.

Dose-dependent effects of central leptin gene therapy on genes that regulate body weight and appetite in the hypothalamus.

S. Kalra, P. Kalra, H. Dhillon, 2001, Molecular therapy : the journal of the American Society of Gene Therapy.

Mice lacking beta-adrenergic receptors have increased bone mass but are not protected from deleterious skeletal effects of ovariectomy.

M. Bouxsein, V. Glatt, D. Pierroz, 2009, Endocrinology.

PI3K signaling in the ventromedial hypothalamic nucleus is required for normal energy homeostasis.

B. Lowell, J. Elmquist, Jean J. Zhao, 2010, Cell metabolism.

Synaptic glutamate release by ventromedial hypothalamic neurons is part of the neurocircuitry that prevents hypoglycemia.

B. Lowell, Zongfang Yang, G. Shulman, 2007, Cell metabolism.

Leptin Directly Activates SF1 Neurons in the VMH, and This Action by Leptin Is Required for Normal Body-Weight Homeostasis

Robert A. McGovern, Joel K. Elmquist, B. Lowell, 2006, Neuron.

EFFECTS OF RECOMBINANT ADENO-ASSOCIATED VIRUS ENCODING LEPTIN ON BODY WEIGHT REGULATION AND ENERGY HOMEOSTASIS

H. Dhillon, 2000 .

Hypoxia as a therapy for mitochondrial disease

Neville E. Sanjana, V. Mootha, W. Goessling, 2016, Science.

Central LIF gene therapy suppresses food intake, body weight, serum leptin and insulin for extended periods

S. Kalra, P. Kalra, H. Dhillon, 2002, Peptides.

High-fat Feeding Promotes Obesity via Insulin Receptor/PI3k-Dependent Inhibition of SF-1 VMH Neurons

B. Lowell, T. Horvath, A. Husch, 2011, Nature Neuroscience.

Central leptin gene therapy blocks high-fat diet-induced weight gain, hyperleptinemia, and hyperinsulinemia: increase in serum ghrelin levels.

M. G. Dube, S. Kalra, P. Kalra, 2002, Diabetes.

Central leptin gene therapy suppresses body weight gain, adiposity and serum insulin without affecting food consumption in normal rats: a long-term study

L. Moldawer, N. Muzyczka, S. Kalra, 2001, Regulatory Peptides.

Long-term differential modulation of genes encoding orexigenic and anorexigenic peptides by leptin delivered by rAAV vector in ob/ob mice Relationship with body weight change

L. Moldawer, N. Muzyczka, S. Kalra, 2000, Regulatory Peptides.

Selective inactivation of Socs3 in SF1 neurons improves glucose homeostasis without affecting body weight.

B. Lowell, J. Flier, A. Yoshimura, 2008, Endocrinology.