R. Ribeiro

发表

Systemic inhibition of nitric oxide synthesis in non-diabetic individuals produces a significant deterioration in glucose tolerance by increasing insulin clearance and inhibiting insulin secretion

M. Rossi, E. Ferrannini, A. Natali, 2013, Diabetologia.

Differential Age-Dependent Mitochondrial Dysfunction, Oxidative Stress, and Apoptosis Induced by Neonatal Hypoxia-Ischemia in the Immature Rat Brain

M. Wajner, C. Dalmaz, C. Netto, 2021, Molecular Neurobiology.

Experimental Evidence that In Vivo Intracerebral Administration of L-2-Hydroxyglutaric Acid to Neonatal Rats Provokes Disruption of Redox Status and Histopathological Abnormalities in the Brain

G. Leipnitz, M. Wajner, B. Seminotti, 2018, Neurotoxicity Research.

α-Ketoadipic Acid and α-Aminoadipic Acid Cause Disturbance of Glutamatergic Neurotransmission and Induction of Oxidative Stress In Vitro in Brain of Adolescent Rats

G. Leipnitz, C. Vargas, M. Wajner, 2017, Neurotoxicity Research.

Disturbance of Mitochondrial Dynamics, Endoplasmic Reticulum–Mitochondria Crosstalk, Redox Homeostasis, and Inflammatory Response in the Brain of Glutaryl-CoA Dehydrogenase-Deficient Mice: Neuroprotective Effects of Bezafibrate

G. Leipnitz, M. Wajner, B. Seminotti, 2022, Molecular Neurobiology.

l-Carnitine prevents oxidative stress in striatum of glutaryl-CoA dehydrogenase deficient mice submitted to lysine overload.

A. Sitta, C. Vargas, M. Wajner, 2019, Biochimica et biophysica acta. Molecular basis of disease.

3-Hydroxy-3-Methylglutaric Acid Impairs Redox and Energy Homeostasis, Mitochondrial Dynamics, and Endoplasmic Reticulum–Mitochondria Crosstalk in Rat Brain

G. Leipnitz, M. Wajner, M. Grings, 2019, Neurotoxicity Research.

S-Adenosylmethionine Promotes Oxidative Stress and Decreases Na+, K+-ATPase Activity in Cerebral Cortex Supernatants of Adolescent Rats: Implications for the Pathogenesis of S-Adenosylhomocysteine Hydrolase Deficiency

G. Leipnitz, M. Wajner, A. Wyse, 2018, Molecular Neurobiology.

Oxidative Stress, Disrupted Energy Metabolism, and Altered Signaling Pathways in Glutaryl-CoA Dehydrogenase Knockout Mice: Potential Implications of Quinolinic Acid Toxicity in the Neuropathology of Glutaric Acidemia Type I

G. Leipnitz, M. Wajner, B. Seminotti, 2016, Molecular Neurobiology.

S-adenosylmethionine induces mitochondrial dysfunction, permeability transition pore opening and redox imbalance in subcellular preparations of rat liver

G. Leipnitz, M. Wajner, B. Seminotti, 2021, Journal of Bioenergetics and Biomembranes.

Mevalonolactone disrupts mitochondrial functions and induces permeability transition pore opening in rat brain mitochondria: Implications for the pathogenesis of mevalonic aciduria

C. Vargas, M. Wajner, A. U. Amaral, 2017, Neurochemistry International.

Acute lysine overload provokes marked striatum injury involving oxidative stress signaling pathways in glutaryl-CoA dehydrogenase deficient mice

D. Souza, G. Leipnitz, M. Wajner, 2019, Neurochemistry International.

Disruption of mitochondrial functions involving mitochondrial permeability transition pore opening caused by maleic acid in rat kidney

M. Wajner, A. U. Amaral, S. Wajner, 2022, Journal of Bioenergetics and Biomembranes.

Plinia trunciflora Extract Administration Prevents HI-Induced Oxidative Stress, Inflammatory Response, Behavioral Impairments, and Tissue Damage in Rats

M. Wajner, C. Dalmaz, C. Netto, 2022, Nutrients.

Atividade Física Intensa: Papel do Óxido Nítrico na Apoptose. Intense Physical Activity: Role of Nitric Oxide in Apoptosis

R. Ribeiro, J. A. Junior, W. N. Sousa, 2013 .

Influence of Altered Thyroid Hormone Mechanisms in the Progression of Metabolic Dysfunction Associated with Fatty Liver Disease (MAFLD): A Systematic Review

S. Wajner, R. Ribeiro, R. Marschner, 2022, Metabolites.

mitochondrial permeability transition pore opening in rat brain and liver : possible implications for the pathogenesis of MCAD deficiency

M. Wajner, A. U. Amaral, R. Ribeiro, 2016 .

Induction of Neuroinflammatory Response and Histopathological Alterations Caused by Quinolinic Acid Administration in the Striatum of Glutaryl-CoA Dehydrogenase Deficient Mice

D. Souza, G. Leipnitz, C. Vargas, 2017, Neurotoxicity Research.

Free Radical Scavengers Prevent Argininosuccinic Acid-Induced Oxidative Stress in the Brain of Developing Rats: a New Adjuvant Therapy for Argininosuccinate Lyase Deficiency?

G. Leipnitz, M. Wajner, B. Seminotti, 2019, Molecular Neurobiology.

Disruption of Brain Redox Homeostasis, Microglia Activation and Neuronal Damage Induced by Intracerebroventricular Administration of S-Adenosylmethionine to Developing Rats

G. Leipnitz, M. Wajner, A. Wyse, 2018, Molecular Neurobiology.

Disruption of mitochondrial bioenergetics and calcium homeostasis by phytanic acid in the heart: Potential relevance for the cardiomyopathy in Refsum disease.

M. Wajner, A. U. Amaral, S. Wajner, 2023, Biochimica et biophysica acta. Bioenergetics.

Sulfite Impairs Bioenergetics and Redox Status in Neonatal Rat Brain: Insights into the Early Neuropathophysiology of Isolated Sulfite Oxidase and Molybdenum Cofactor Deficiencies

G. Leipnitz, M. Wajner, A. Wyse, 2023, Cellular and Molecular Neurobiology.

Neuronal Death, Glial Reactivity, Microglia Activation, Oxidative Stress and Bioenergetics Impairment Caused by Intracerebroventricular Administration of D-2-hydroxyglutaric Acid to Neonatal Rats

G. Leipnitz, M. Wajner, B. Seminotti, 2021, Neuroscience.

Acute lysine overload provokes protein oxidative damage and reduction of antioxidant defenses in the brain of infant glutaryl-CoA dehydrogenase deficient mice: A role for oxidative stress in GA I neuropathology

G. Leipnitz, M. Wajner, B. Seminotti, 2014, Journal of the Neurological Sciences.

Uncovering Actions of Type 3 Deiodinase in the Metabolic Dysfunction-Associated Fatty Liver Disease (MAFLD)

S. Wajner, Larisse Longo, M. Álvares-da-Silva, 2023, Cells.

Mitochondrial dysfunction, oxidative stress, ER stress and mitochondria-ER crosstalk alterations in a chemical rat model of Huntington's disease: potential benefits of bezafibrate.

G. Leipnitz, M. Wajner, B. Seminotti, 2023, Toxicology letters.

l-2-Hydroxyglutaric Acid Administration to Neonatal Rats Elicits Marked Neurochemical Alterations and Long-Term Neurobehavioral Disabilities Mediated by Oxidative Stress

M. Wajner, A. U. Amaral, C. Netto, 2022, Neurotoxicity Research.

G. Leipnitz, M. Wajner, A. Wyse, 2018, Molecular Neurobiology.

Glycine disrupts myelin, glutamatergic neurotransmission, and redox homeostasis in a neonatal model for non ketotic hyperglycinemia.

G. Leipnitz, M. Wajner, B. Seminotti, 2023, Biochimie.

Disruption of Brain Redox Homeostasis, Microglia Activation and Neuronal Damage Induced by Intracerebroventricular Administration of S-Adenosylmethionine to Developing Rats

G. Leipnitz, M. Wajner, A. Wyse, 2018, Molecular Neurobiology.

cis-4-Decenoic and decanoic acids impair mitochondrial energy, redox and Ca(2+) homeostasis and induce mitochondrial permeability transition pore opening in rat brain and liver: Possible implications for the pathogenesis of MCAD deficiency.

M. Wajner, A. U. Amaral, R. Ribeiro, 2016, Biochimica et biophysica acta.