Z. Zhao

发表

Brief pretreatment of radial artery conduits with phenoxybenzamine prevents vasoconstriction long term.

R. Guyton, J. Vinten-johansen, S. Muraki, 2001, The Annals of thoracic surgery.

Ischemic preconditioning reduces neutrophil accumulation and myocardial apoptosis.

R. Guyton, J. Wilcox, M. Nakamura, 1999, The Annals of thoracic surgery.

Ischemic preconditioning attenuates postischemic coronary artery endothelial dysfunction in a model of minimally invasive direct coronary artery bypass grafting.

R. Guyton, V. Thourani, M. Nakamura, 1999, The Journal of thoracic and cardiovascular surgery.

Preconditioning during simulated MIDCABG attenuates blood flow defects and neutrophil accumulation.

R. Guyton, V. Thourani, M. Nakamura, 1998, The Annals of thoracic surgery.

Reperfusion induces myocardial apoptotic cell death.

R. Guyton, J. Wilcox, M. Nakamura, 2000, Cardiovascular research.

Gradual reperfusion reduces infarct size and endothelial injury but augments neutrophil accumulation.

H. Sato, J. Vinten-johansen, Zhi-Qing Zhao, 1997, The Annals of thoracic surgery.

Broad-spectrum cardioprotection with adenosine.

R. Guyton, V. Thourani, M. Nakamura, 1999, The Annals of thoracic surgery.

Intracoronary L-arginine during reperfusion improves endothelial function and reduces infarct size.

D. Lefer, J. Vinten-johansen, Zhi-Qing Zhao, 1992, The American journal of physiology.

Coronary artery endothelial dysfunction after global ischemia, blood cardioplegia, and reperfusion.

J. Hammon, J. Vinten-johansen, Zhi-Qing Zhao, 1994, The Annals of thoracic surgery.

Supplemental L-arginine during cardioplegic arrest and reperfusion avoids regional postischemic injury.

J. Hammon, H. Sato, J. Vinten-johansen, 1995, The Journal of thoracic and cardiovascular surgery.

The role of nitric oxide and NO-donor agents in myocardial protection from surgical ischemic-reperfusion injury.

H. Sato, J. Vinten-johansen, Z. Zhao, 1995, International journal of cardiology.

A1 receptor mediated myocardial infarct size reduction by endogenous adenosine is exerted primarily during ischaemia.

J. Vinten-johansen, Z. Zhao, K. Nakanishi, 1994, Cardiovascular research.

Nitric oxide and the vascular endothelium in myocardial ischemia-reperfusion injury.

R. Guyton, V. Thourani, M. Nakamura, 1998, Annals of the New York Academy of Sciences.

Reduction in surgical ischemic-reperfusion injury with adenosine and nitric oxide therapy.

H. Sato, J. Vinten-johansen, Z. Zhao, 1995, The Annals of thoracic surgery.

Blood cardioplegia enhanced with nitric oxide donor SPM-5185 counteracts postischemic endothelial and ventricular dysfunction.

J. Hammon, J. Vinten-johansen, Zhi-Qing Zhao, 1995, The Journal of thoracic and cardiovascular surgery.

Pentostatin-augmented interstitial adenosine prevents postcardioplegia injury in damaged hearts.

J. Hammon, H. Sato, J. Vinten-johansen, 1995, The Annals of thoracic surgery.

Adenosine in blood cardioplegia prevents postischemic dysfunction in ischemically injured hearts.

J. Hammon, J. Vinten-johansen, Z. Zhao, 1994, The Annals of thoracic surgery.

Experimental off-pump coronary artery revascularization with adenosine-enhanced reperfusion.

R. Guyton, J. Vinten-johansen, S. Muraki, 2001, The Journal of thoracic and cardiovascular surgery.

Adenosine-supplemented blood cardioplegia attenuates postischemic dysfunction after severe regional ischemia.

R. Guyton, V. Thourani, J. Vinten-johansen, 1999, Circulation.

Adenosine inhibition of neutrophil damage during reperfusion does not involve K(ATP)-channel activation.

X. Ma, H. Sato, Z. Zhao, 1997, The American journal of physiology.

Acadesine Improves Surgical Myocardial Protection With Blood Cardioplegia in Ischemically Injured Canine Hearts

J. Vinten-johansen, Zhi-Qing Zhao, P. Tan, 1993, Circulation.

Basal nitric oxide expresses endogenous cardioprotection during reperfusion by inhibition of neutrophil-mediated damage after surgical revascularization.

J. Hammon, X. Ma, H. Sato, 1997, The Journal of thoracic and cardiovascular surgery.

The role of neutrophils in myocardial ischemia-reperfusion injury.

J. Vinten-johansen, Zhi-Qing Zhao, J E Jordan, 1999, Cardiovascular research.

L-Arginine inhibits neutrophil adherence and coronary artery dysfunction.

H. Sato, J. Vinten-johansen, Z. Zhao, 1996, Cardiovascular research.

Endogenous nitric oxide (NO) protects against ischaemia-reperfusion injury in the rabbit.

J. Vinten-johansen, Zhi-Qing Zhao, Z. Zhao, 1995, Cardiovascular research.

Dynamic progression of contractile and endothelial dysfunction and infarct extension in the late phase of reperfusion.

R. Guyton, M. Nakamura, K. Hewan-Lowe, 2000, The Journal of surgical research.

Adenosine attenuates reperfusion-induced apoptotic cell death by modulating expression of Bcl-2 and Bax proteins.

R. Guyton, Ningping Wang, J. Vinten-johansen, 2001, Journal of molecular and cellular cardiology.

Preconditioning decreases Bax expression, PMN accumulation and apoptosis in reperfused rat heart.

R. Guyton, J. Wilcox, V. Thourani, 2000, Cardiovascular research.

Adenosine A2 receptor activation attenuates reperfusion injury by inhibiting neutrophil accumulation, superoxide generation and coronary endothelial adherence.

H. Sato, J. Vinten-johansen, Zhi-Qing Zhao, 1997, The Journal of pharmacology and experimental therapeutics.

Nonanticoagulant heparin inhibits NF-kappaB activation and attenuates myocardial reperfusion injury.

V. Thourani, J. Vinten-johansen, J. Hoidal, 2000, American journal of physiology. Heart and circulatory physiology.

Comparative study of AMP579 and adenosine in inhibition of neutrophil-mediated vascular and myocardial injury during 24 h of reperfusion.

R. Guyton, K. Clark, J. Vinten-johansen, 2000, Cardiovascular research.

Intravascular adenosine at reperfusion reduces infarct size and neutrophil adherence.

H. Sato, J. Vinten-johansen, Zhi-Qing Zhao, 1996, The Annals of thoracic surgery.

A novel adenosine analog, AMP579, inhibits neutrophil activation, adherence and neutrophil-mediated injury to coronary vascular endothelium.

R. Guyton, M. Nakamura, K. Clark, 2000, European journal of pharmacology.

Acadesine reduces myocardial infarct size by an adenosine mediated mechanism.

H. Sato, J. Vinten-johansen, D. Van Wylen, 1995, Cardiovascular research.

Adenosine A2-receptor activation inhibits neutrophil-mediated injury to coronary endothelium.

H. Sato, J. Vinten-johansen, Zhi-Qing Zhao, 1996, The American journal of physiology.

Peroxynitrite, the breakdown product of nitric oxide, is beneficial in blood cardioplegia but injurious in crystalloid cardioplegia.

R. Guyton, V. Thourani, M. Nakamura, 1999, Circulation.

Recombinant human complement C5a receptor antagonist reduces infarct size after surgical revascularization.

V. Thourani, H. Sato, J. Vinten-johansen, 2000, The Journal of thoracic and cardiovascular surgery.

Adenosine A(3)-receptor stimulation attenuates postischemic dysfunction through K(ATP) channels.

R. Guyton, V. Thourani, M. Nakamura, 1999, The American journal of physiology.

Electroplegia: an alternative to blood cardioplegia for arresting the heart during conventional (on-pump) cardiac operation.

R. Guyton, J. Puskas, J. Vinten-johansen, 2001, The Annals of thoracic surgery.

Administration of adenosine during reperfusion reduces injury of vascular endothelium and death of myocytes.

R. Guyton, J. Wilcox, M. Nakamura, 1999, Coronary artery disease.