A. Kohm

发表

Norepinephrine and beta 2-adrenergic receptor stimulation regulate CD4+ T and B lymphocyte function in vitro and in vivo.

V. Sanders, A. Kohm, 2001, Pharmacological reviews.

Natural CD4 + CD25 + Regulatory T Cells in Regulation of Autoimmune Disease

S. Miller, A. Kohm, 2008 .

CD4+CD25+ Regulatory T Cells in Autoimmune Disease

S. Miller, A. Kohm, 2007 .

Activation of Antigen-Specific CD4+ Th2 Cells and B Cells In Vivo Increases Norepinephrine Release in the Spleen and Bone Marrow1

V. Sanders, Stephen B. Jones, Yueming Tang, 2000, The Journal of Immunology.

Pathogenesis of NOD diabetes is initiated by reactivity to the insulin B chain 9-23 epitope and involves functional epitope spreading.

S. Miller, Xunrong Luo, A. Kohm, 2012, Journal of autoimmunity.

Mimicking the way to autoimmunity: an evolving theory of sequence and structural homology.

S. Miller, A. Kohm, K. Fuller, 2003, Trends in microbiology.

Modulates the Level of IgG 1-Adrenergic Receptor 2 β ( B 7-2 ) , and the Stimulation of the B Cell Receptor , CD 86

A. Sharpe, D. Kasprowicz, V. Sanders, 2000 .

Stimulation of the B Cell Receptor, CD86 (B7-2), and the β2-Adrenergic Receptor Intrinsically Modulates the Level of IgG1 and IgE Produced per B Cell1

D. Kasprowicz, V. Sanders, M. T. Berton, 2000, The Journal of Immunology.

Suppression of antigen-specific Th2 cell-dependent IgM and IgG1 production following norepinephrine depletion in vivo.

V. Sanders, A. Kohm, 1999, Journal of immunology.

Role of ICAM-1 and P-selectin expression in the development and effector function of CD4+CD25+regulatory T cells.

S. Miller, A. Kohm, 2003, Journal of autoimmunity.

CD4 T Cell Expressed CD80 Regulates Central Nervous System Effector Function and Survival during Experimental Autoimmune Encephalomyelitis

S. Miller, J. Podojil, A. Kohm, 2006 .

CD4+ T Cell Expressed CD80 Regulates Central Nervous System Effector Function and Survival during Experimental Autoimmune Encephalomyelitis1

S. Miller, J. Podojil, A. Kohm, 2006, The Journal of Immunology.

EncephalomyelitisExperimental Autoimmune System Inflammation During Active Immune Responses and Central Nervous Cells Suppress Antigen-Specific Autoreactive Regulatory T + CD25+ Cutting Edge: CD4

S. Miller, H. Anger, A. Kohm, 2022 .

Encephalomyelitis Established Experimental Autoimmune Unresponsiveness and Functional Reversal of T Cell + Monoclonal Antibody Induces CD 4 Treatment with Nonmitogenic Anti-CD 3

J. Bluestone, S. Miller, L. Chatenoud, 2005 .

Treatment with Nonmitogenic Anti-CD3 Monoclonal Antibody Induces CD4+ T Cell Unresponsiveness and Functional Reversal of Established Experimental Autoimmune Encephalomyelitis1

J. Bluestone, S. Miller, L. Chatenoud, 2005, The Journal of Immunology.

EncephalomyelitisExperimental Autoimmune System Inflammation During Active Immune Responses and Central Nervous Cells Suppress Antigen-Specific Autoreactive Regulatory T + CD25+ Cutting Edge: CD4

S. Miller, H. Anger, A. Kohm, 2002 .

CD28 regulates glucocorticoid-induced TNF receptor family-related gene expression on CD4+ T cells via IL-2-dependent mechanisms.

S. Miller, J. Podojil, A. Kohm, 2005, Cellular immunology.

Cutting Edge: Ligation of the Glucocorticoid-Induced TNF Receptor Enhances Autoreactive CD4+ T Cell Activation and Experimental Autoimmune Encephalomyelitis1

S. Miller, A. Kohm, Julie S. Williams, 2004, The Journal of Immunology.

Cutting Edge: Anti-CD25 Monoclonal Antibody Injection Results in the Functional Inactivation, Not Depletion, of CD4+CD25+ T Regulatory Cells1

S. Ziegler, S. Miller, D. Kasprowicz, 2006, The Journal of Immunology.

Sympathetic nervous system interaction with the immune system.

V. Sanders, A. Kohm, 2002, International review of neurobiology.

B Cell Receptor- and β2-Adrenergic Receptor-Induced Regulation of B7-2 (CD86) Expression in B Cells1

V. Sanders, Afsaneh Mozaffarian, A. Kohm, 2002, The Journal of Immunology.

Expression in B Cells Receptor-Induced Regulation of B 7-2 ( CD 86 )-Adrenergic 2 β B Cell Receptor-and Sanders

V. Sanders, Afsaneh Mozaffarian, A. Kohm, 2002 .

Norepinephrine: a messenger from the brain to the immune system.

V. Sanders, A. Kohm, Virginia M. Sanders, 2000, Immunology today.

Norepinephrine Release in the Spleen and Cells and B Cells In Vivo Increases Th2 + Activation of Antigen-Specific CD4

V. Sanders, Stephen B. Jones, Yueming Tang, 2000 .

Regulatory Cells T + CD 25 + Inactivation , Not Depletion , of CD 4 Antibody Injection Results in the Functional Cutting Edge : Anti-CD 25 Monoclonal

S. Ziegler, S. Miller, D. Kasprowicz, 2006 .

EncephalomyelitisActivation and Experimental Autoimmune T Cell + Enhances Autoreactive CD4 Glucocorticoid-Induced TNF Receptor Cutting Edge: Ligation of the

S. Miller, A. Kohm, Julie S. Williams, 2004 .

CCL22 regulates experimental autoimmune encephalomyelitis by controlling inflammatory macrophage accumulation and effector function

S. Miller, W. Karpus, Rukiye-Nazan E. Dogan, 2011, Journal of leukocyte biology.

Targeting the TCR: T-Cell Receptor and Peptide-Specific Tolerance–Based Strategies for Restoring Self-Tolerance in CNS Autoimmune Disease

S. Miller, D. Turley, A. Kohm, 2005, International reviews of immunology.

Anti-thymocyte globulin (ATG) prevents autoimmune encephalomyelitis by expanding myelin antigen-specific Foxp3+ regulatory T cells.

T. Korn, M. Oukka, S. Miller, 2007, International immunology.

Exacerbation of Facial Motoneuron Loss after Facial Nerve Transection in Severe Combined Immunodeficient (scid) Mice

V. Sanders, C. Huppenbauer, A. Kohm, 1999, The Journal of Neuroscience.

Effect of hematopoietic growth factors on severity of experimental autoimmune encephalomyelitis

S. Miller, L. Verda, Y. Oyama, 2006, Bone Marrow Transplantation.

Endoplasmic reticulum stress response as a potential therapeutic target in multiple sclerosis.

S. Miller, D. Getts, M. T. Getts, 2008, Therapy.

Nitric oxide synthase inhibition blocks octopus touch learning without producing sensory or motor dysfunction

J. Bonaventura, J. D. Robertson, A. Kohm, 1995, Proceedings of the Royal Society of London. Series B: Biological Sciences.

T Regulatory + CD25+ Depletion, of CD4 Results in the Functional Inactivation, Not Anti-CD25 Monoclonal Antibody Injection Response to Comment on ''Cutting Edge:

S. Miller, A. Kohm, Stephen D. Miller, 2006 .

Regulation of experimental autoimmune encephalomyelitis (EAE) by CD4+CD25+ regulatory T cells.

S. Miller, A. Kohm, P. Carpentier, 2003, Novartis Foundation symposium.

Adaptive immunity in mice lacking the β2-adrenergic receptor

D. Kasprowicz, V. Sanders, J. Podojil, 2003, Brain, Behavior, and Immunity.

Adaptive immunity in mice lacking the beta(2)-adrenergic receptor.

D. Kasprowicz, V. Sanders, J. Podojil, 2003, Brain, behavior, and immunity.

Nitric oxide is required for tactile learning in Octopus vulgaris

J. Bonaventura, J. D. Robertson, A. Kohm, 1994, Proceedings of the Royal Society of London. Series B: Biological Sciences.

Nitric oxide is necessary for visual learning in Octopus vulgaris

J. Bonaventura, J. D. Robertson, A. Kohm, 1996, Proceedings of the Royal Society of London. Series B: Biological Sciences.

T Regulatory + CD25+ Depletion, of CD4 Results in the Functional Inactivation, Not Anti-CD25 Monoclonal Antibody Injection Response to Comment on ''Cutting Edge:

S. Miller, A. Kohm, 2006 .

EncephalomyelitisExperimental Autoimmune System Inflammation During Active Immune Responses and Central Nervous Cells Suppress Antigen-Specific Autoreactive Regulatory T + CD25+ Cutting Edge: CD4

S. Miller, H. Anger, A. Kohm, 2002 .