D. J. Bonnet

发表

The conformation of apolipoprotein A-I in high-density lipoproteins is influenced by core lipid composition and particle size: a surface plasmon resonance study.

K. Rye, L. Curtiss, L K Curtiss, 2000, Biochemistry.

The impact of glycation on apolipoprotein A-I structure and its ability to activate lecithin:cholesterol acyltransferase

P. Barter, A. Jenkins, K. Rye, 2007, Diabetologia.

The LPS2 mutation in TRIF is atheroprotective in hyperlipidemic low density lipoprotein receptor knockout mice

G. Barish, P. Tobias, L. Curtiss, 2013, Innate immunity.

In vivo efficacy of HDL-like nanolipid particles containing multivalent peptide mimetics of apolipoprotein A-I[S]

M. Ghadiri, L. Curtiss, L. Leman, 2014, Journal of Lipid Research.

Nonenzymatic Glycation Impairs the Antiinflammatory Properties of Apolipoprotein A-I

P. Barter, A. Jenkins, G. Dusting, 2010, Arteriosclerosis, thrombosis, and vascular biology.

Participation of macrophages in atherosclerotic lesion morphology in LDLr−/− mices⃞s⃞ The online version of this article (available at http://www.jlr.org) contains an additional two figures. Published, JLR Papers in Press, June 1, 2004. DOI 10.1194/jlr.M400036-JLR200

L. Curtiss, N. K. Schiller, D. J. Bonnet, 2004, Journal of Lipid Research.

Atherosclerosis induced by endogenous and exogenous toll-like receptor (TLR)1 or TLR6 agonists[S]

P. Tobias, L. Curtiss, D. J. Bonnet, 2012, Journal of Lipid Research.

Localization of an apolipoprotein A-I epitope critical for activation of lecithin-cholesterol acyltransferase.

L. Curtiss, R. Smith, C. Banka, 1991, The Journal of biological chemistry.

Definition of a nonlinear conformational epitope for the apolipoprotein B-100-specific monoclonal antibody, MB47.

R. Koduri, S. Young, L. Curtiss, 1994, Journal of lipid research.

Bone marrow-derived HL mitigates bone marrow-derived CETP-mediated decreases in HDL in mice globally deficient in HL and the LDLr

L. Curtiss, N. Hime, D. J. Bonnet, 2014, Journal of Lipid Research.

Evidence that endothelial lipase remodels high density lipoproteins without mediating the dissociation of apolipoprotein A-I Published, JLR Papers in Press, February 1, 2005. DOI 10.1194/jlr.M400212-JLR200

D. Rader, K. Rye, L. Curtiss, 2005, Journal of Lipid Research.

Atheroprotective Potential of Macrophage-Derived Phospholipid Transfer Protein in Low-Density Lipoprotein Receptor–Deficient Mice Is Overcome by Apolipoprotein AI Overexpression

L. Lagrost, L. Curtiss, David T. Valenta, 2006, Arteriosclerosis, thrombosis, and vascular biology.

Reduced cholesterol and triglycerides in mice with a mutation in Mia2, a liver protein that localizes to ER exit sites[S]

N. Gekakis, L. Curtiss, Jeffrey L. Pitman, 2011, Journal of Lipid Research.

Macrophage PLTP is atheroprotective in LDLr-deficient mice with systemic PLTP deficiency Published, JLR Papers in Press, October 10, 2007.

L. Curtiss, David T. Valenta, Joshua J Bulgrien, 2008, Journal of Lipid Research.

Apolipoprotein A-II Inhibits High Density Lipoprotein Remodeling and Lipid-poor Apolipoprotein A-I Formation*

P. Barter, K. Rye, L. Curtiss, 2003, Journal of Biological Chemistry.

Evidence that phospholipids play a key role in pre-beta apoA-I formation and high-density lipoprotein remodeling.

P. Barter, R. Stocker, K. Rye, 2002, Biochemistry.