E. Werner

Differential fates of invertase mutants in the yeast endoplasmic reticulum

J. Brodsky, A. McCracken, E. Werner, 2000, Yeast.

A soluble activin Type IIA receptor induces bone formation and improves skeletal integrity

M. Bouxsein, A. Grinberg, L. Stadmeyer, 2008, Proceedings of the National Academy of Sciences.

The Requirement for Molecular Chaperones during Endoplasmic Reticulum-associated Protein Degradation Demonstrates That Protein Export and Import Are Mechanistically Distinct*

J. Brodsky, A. McCracken, M. Dubas, 1999, The Journal of Biological Chemistry.

Inhibiting activin‐A signaling stimulates bone formation and prevents cancer‐induced bone destruction in vivo

M. Bouxsein, P. Croucher, T. Arnett, 2010, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

Grb10 Inhibits Insulin-stimulated Insulin Receptor Substrate (IRS)-Phosphatidylinositol 3-Kinase/Akt Signaling Pathway by Disrupting the Association of IRS-1/IRS-2 with the Insulin Receptor*

F. Liu, S. Shoelson, Lily Q. Dong, 2003, The Journal of Biological Chemistry.

Soluble Endoglin Specifically Binds Bone Morphogenetic Proteins 9 and 10 via Its Orphan Domain, Inhibits Blood Vessel Formation, and Suppresses Tumor Growth*

N. Solban, A. Grinberg, A. Mulivor, 2011, The Journal of Biological Chemistry.

Yeast mutants deficient in ER-associated degradation of the Z variant of alpha-1-protease inhibitor.

A. Dillin, W. Courchesne, A. McCracken, 1996, Genetics.

Insulin Resistance Due to Phosphorylation of Insulin Receptor Substrate-1 at Serine 302*

S. Shoelson, L. Hansen, Jongsoon Lee, 2004, Journal of Biological Chemistry.