Anju Melkani

发表

A Drosophila model of dominant inclusion body myopathy type 3 shows diminished myosin kinetics that reduce muscle power and yield myofibrillar defects

D. Swank, S. Bernstein, B. Glasheen, 2017, Disease Models & Mechanisms.

A Failure to Communicate

S. Bernstein, G. Melkani, Anju Melkani, 2015, The Journal of Biological Chemistry.

ASSESSMENT OF THE POTENTIAL HEALTH HAZARDOUS CHEMICALS IN HOUSE HOLD DUST

Anju Melkani, 2013 .

Expression of the inclusion body myopathy 3 mutation in Drosophila depresses myosin function and stability and recapitulates muscle inclusions and weakness

S. Bernstein, A. Cammarato, G. Melkani, 2012, Molecular biology of the cell.

Prolonged cross-bridge binding triggers muscle dysfunction in a Drosophila model of myosin-based hypertrophic cardiomyopathy

D. Swank, Adriana S Trujillo, S. Bernstein, 2018, eLife.

Mapping Interactions between Myosin Relay and Converter Domains That Power Muscle Function*

S. Bernstein, G. Melkani, Anju Melkani, 2014, The Journal of Biological Chemistry.

Alternative relay and converter domains tune native muscle myosin isoform function in Drosophila.

S. Bernstein, G. Melkani, Anju Melkani, 2012, Journal of molecular biology.

Mutating the converter-relay interface of Drosophila myosin perturbs ATPase activity, actin motility, myofibril stability and flight ability.

S. Bernstein, G. Melkani, Anju Melkani, 2010, Journal of molecular biology.

A Restrictive Cardiomyopathy Mutation in an Invariant Proline at the Myosin Head/Rod Junction Enhances Head Flexibility and Function, Yielding Muscle Defects in Drosophila.

K. Ocorr, R. Bodmer, D. Swank, 2016, Journal of molecular biology.

A Drosophila model of dominant inclusion body myopathy type 3 shows diminished myosin kinetics that reduce muscle power and yield myofibrillar defects

D. Swank, S. Bernstein, B. Glasheen, 2017, Disease Models & Mechanisms.