O. Akimova

发表

Modest intracellular acidification suppresses death signaling in ouabain-treated cells

S. Orlov, P. Hamet, O. Akimova, 2005, Pflügers Archiv.

Hyperosmotic and isosmotic shrinkage differentially affect protein phosphorylation and ion transport.

S. V. Kotelevtsev, S. Orlov, S. Koltsova, 2012, Canadian journal of physiology and pharmacology.

Death of ouabain-treated renal epithelial cells: evidence for p38 MAPK-mediated Nai+/Ki+-independent signaling

S. Orlov, P. Hamet, J. Tremblay, 2009, Apoptosis.

Search for intermediates of Na+,K+-ATPase-mediated [Na+]i/[K+]i-independent death signaling triggered by cardiotonic steroids.

S. Orlov, P. Hamet, O. Lopina, 2005, Pathophysiology : the official journal of the International Society for Pathophysiology.

Molecular origin of Na(+)/Li(+) exchanger: Evidence against the involvement of major cloned erythrocyte transporters.

S. Orlov, P. Hamet, S. Koltsova, 2011, Pathophysiology : the official journal of the International Society for Pathophysiology.

Revealing of Proteins Interacting with Na,K-ATPase

O. Lopina, A. Rubtsov, O. Akimova, 2003, Biochemistry (Moscow).

Decreased NKCC1 activity in erythrocytes from African Americans with hypertension and dyslipidemia.

D. Gaudet, Z. Pausova, S. Orlov, 2010, American journal of hypertension.

The rapid decline of MTT reduction is not a marker of death signaling in ouabain-treated cells.

S. Orlov, P. Hamet, A. A. Mongin, 2006, Cellular and molecular biology.

Cardiotonic Steroids Differentially Affect Intracellular Na+ and [Na+]i/[K+]i-independent Signaling in C7-MDCK Cells*

S. Orlov, P. Hamet, J. Tremblay, 2005, Journal of Biological Chemistry.

Cardiotonic steroid-resistant α1-Na+,K+-ATPase rescues renal epithelial cells from the cytotoxic action of ouabain: evidence for a Nai+,Ki+-independent mechanism

S. Orlov, P. Hamet, J. Tremblay, 2009, Apoptosis.

The death of ouabain-treated renal epithelial cells: evidence against anoikis occurrence

S. V. Kotelevtsev, S. Orlov, P. Hamet, 2008, Apoptosis.

Effects of ouabain on proliferation of human endothelial cells correlate with Na+,K+-ATPase activity and intracellular ratio of Na+ and K+

S. Orlov, L. Smolyaninova, E. Klimanova, 2016, Biochemistry (Moscow).

[Na+]i/[K+]i-independent death of ouabain-treated renal epithelial cells is not mediated by Na+,K+-ATPase internalization and de novo gene expression

S. Orlov, P. Hamet, O. Akimova, 2007, Pflügers Archiv - European Journal of Physiology.

Identification of proteins whose interaction with Na+,K+-ATPase is triggered by ouabain

S. Orlov, O. Lopina, L. Kapilevich, 2016, Biochemistry (Moscow).

Critical role of the α1-Na+, K+-ATPase subunit in insensitivity of rodent cells to cytotoxic action of ouabain

S. Orlov, A. A. Mongin, N. Dulin, 2015, Apoptosis.

Low efficiency of functional translation of ouabain-resistant α2-Na(+),K(+)-ATPase mRNA in C7-MDCK epithelial cells.

S. Orlov, J. Tremblay, J. V. Van Huysse, 2012, Canadian journal of physiology and pharmacology.

Investigation of mechanism of p38 MAPK activation in renal epithelial cell from distal tubules triggered by cardiotonic steroids

S. Orlov, P. Hamet, O. Lopina, 2010, Biochemistry (Moscow).

Altered Phosphorylation of RRXS*/T* Motif in Ouabain-Treated Renal Epithelial Cells is not Mediated by Inversion of the [Na+]i/[K+]i Ratio

S. Orlov, P. Hamet, J. Tremblay, 2008, Cellular Physiology and Biochemistry.

Regulation of myofibroblast differentiation by cardiac glycosides.

S. Orlov, R. Hamanaka, G. Mutlu, 2016, American journal of physiology. Lung cellular and molecular physiology.

Purinergic inhibition of Na+,K+,Cl− cotransport in C11-MDCK cells: Role of stress-activated protein kinases

S. Orlov, N. Dulin, S. Taurin, 2007, Purinergic Signalling.

A protein whose binding to Na,K-ATPase is regulated by ouabain

S. Orlov, O. Lopina, A. Rubtsov, 2007, Biochemistry (Moscow).

Transient Activation and Delayed Inhibition of Na+,K+,Cl– Cotransport in ATP-treated C11-MDCK Cells Involve Distinct P2Y Receptor Subtypes and Signaling Mechanisms*

P. Insel, S. Orlov, M. Gekle, 2006, Journal of Biological Chemistry.

Transcriptomic Changes Triggered by Hypoxia: Evidence for HIF-1α -Independent, [Na+]i/[K+]i-Mediated, Excitation-Transcription Coupling

S. Orlov, P. Hamet, J. Tremblay, 2014, PloS one.

[Na+]i‐induced c‐Fos expression is not mediated by activation of the 5′‐promoter containing known transcriptional elements

S. Orlov, P. Hamet, J. Tremblay, 2007, The FEBS journal.

Ubiquitous [Na+]i/[K+]i-Sensitive Transcriptome in Mammalian Cells: Evidence for Ca2+ i-Independent Excitation-Transcription Coupling

S. Orlov, P. Hamet, J. Tremblay, 2012, PloS one.

The Na(+)/K(+)-ATPase as [K(+)](o) sensor: Role in cardiovascular disease pathogenesis and augmented production of endogenous cardiotonic steroids.

S. Orlov, P. Hamet, J. Tremblay, 2006, Pathophysiology : the official journal of the International Society for Pathophysiology.