M. Lobigs

发表

Membrane fusion process of Semliki Forest virus. II: Cleavage-dependent reorganization of the spike protein complex controls virus entry

J. Wahlberg, P. Liljeström, H. Garoff, 1992, The Journal of cell biology.

MHC class I up‐regulation by flaviviruses: Immune interaction with unknown advantage to host or pathogen

A. Müllbacher, M. Regner, M. Lobigs, 2003, Immunology and cell biology.

Spike protein oligomerization control of Semliki Forest virus fusion

J. Wahlberg, H. Garoff, M. Lobigs, 1990, Journal of virology.

Function of Semliki Forest virus E3 peptide in virus assembly: replacement of E3 with an artificial signal peptide abolishes spike heterodimerization and surface expression of E1

H. Garoff, H. Zhao, M. Lobigs, 1990, Journal of virology.

Fusion function of the Semliki Forest virus spike is activated by proteolytic cleavage of the envelope glycoprotein precursor p62

H. Garoff, M. Lobigs, 1990, Journal of virology.

Polymorphic peptide transporters in MHC class I monomorphic Syrian hamster

A. Müllbacher, R. Blanden, M. Lobigs, 2004, Immunogenetics.

High Peptide Affinity for MHC Class I does not Correlate with Immunodominance

J. Yewdell, J. Bennink, A. Müllbacher, 1999, Scandinavian journal of immunology.

Up-regulation of MHC class I by flavivirus-induced peptide translocation into the endoplasmic reticulum.

A. Müllbacher, M. Lobigs, 1995, Immunity.

Spontaneous mutation at position 114 in H‐2Kd affects cytotoxic T cell responses to influenza virus infection

J. Yewdell, J. Bennink, A. Müllbacher, 1999, European journal of immunology.

Surface on the Cellg 7 Peptide Associated with I-A ChainLevels of Class II-Associated Invariant Nonobese Diabetic Mice Display Elevated

P. Milburn, R. Blanden, A. Gautam, 2001 .

Nonobese Diabetic Mice Display Elevated Levels of Class II-Associated Invariant Chain Peptide Associated with I-Ag7 on the Cell Surface

P. Milburn, R. Blanden, A. Gautam, 2001, The Journal of Immunology.

Determinants of attenuation in the envelope protein of the flavivirus Alfuy.

J. Mackenzie, N. Prow, R. Hall, 2011, The Journal of general virology.

Exhaustion of Type I Interferon Response following an Acute Viral Infection

M. Alsharifi, A. Müllbacher, A. Koskinen, 2006, The Journal of Immunology.

Protective immune responses to the E and NS1 proteins of Murray Valley encephalitis virus in hybrids of flavivirus-resistant mice.

M. J. Howard, J. Mackenzie, M. Sangster, 1996, The Journal of general virology.

Proteolytic processing of peptides in the lumen of the endoplasmic reticulum for antigen presentation by major histocompatibility class I

G. Chelvanayagam, A. Müllbacher, M. Lobigs, 2000, European journal of immunology.

In situ reactions of monoclonal antibodies with a viable mutant of Murray Valley encephalitis virus reveal an absence of dimeric NS1 protein.

M. J. Howard, R. Hall, B. Blitvich, 2007, The Journal of general virology.

Properties Interferon Coexpression on Vaccine Vaccine and Evaluation of the Effect of Beta Attenuation of a Flavivirus Candidate Internal Ribosome Entry Site-Based

K. Matthaei, A. Khromykh, P. S. Lim, 2014 .

Internal Ribosome Entry Site-Based Attenuation of a Flavivirus Candidate Vaccine and Evaluation of the Effect of Beta Interferon Coexpression on Vaccine Properties

K. Matthaei, A. Khromykh, P. S. Lim, 2013, Journal of Virology.

Antigen-Dependent Release of IFN-γ by Cytotoxic T Cells Up-Regulates Fas on Target Cells and Facilitates Exocytosis-Independent Specific Target Cell Lysis

M. Simon, A. Müllbacher, T. Stehlé, 2002, The Journal of Immunology.

Murray Valley encephalitis virus field strains from Australia and Papua New Guinea: studies on the sequence of the major envelope protein gene and virulence for mice.

L. Dalgarno, I. Marshall, R. Weir, 1988, Virology.

A Single Amino Acid Substitution in the West Nile Virus Nonstructural Protein NS2A Disables Its Ability To Inhibit Alpha/Beta Interferon Induction and Attenuates Virus Virulence in Mice

W. Liu, R. Hall, A. Khromykh, 2006, Journal of Virology.

Effector Cytolotic Funtion but not IFN‐γ Production in Cytotoxic T Cells Triggered by Virus‐Infected Target Cells In vitro

A. Müllbacher, M. Regner, R. Blanden, 2001, Scandinavian journal of immunology.

Proteolytic processing of a Murray Valley encephalitis virus non-structural polyprotein segment containing the viral proteinase: accumulation of a NS3-4A precursor which requires mature NS3 for efficient processing.

M. Lobigs, 1992, The Journal of general virology.

Recognition of vaccinia virus-encoded major histocompatibility complex class I antigens by virus immune cytotoxic T cells is independent of the polymorphism of the peptide transporters.

A. Müllbacher, M. Lobigs, 1993, Proceedings of the National Academy of Sciences of the United States of America.

following an Acute Viral Infection Exhaustion of Type I Interferon Response

M. Alsharifi, A. Müllbacher, A. Koskinen, 2006 .

Restricted Semliki Forest virus replication in perforin and Fas-ligand double-deficient mice.

M. Alsharifi, J. Bettadapura, A. Müllbacher, 2008, The Journal of general virology.

CD8+ T Cells Mediate Recovery andImmunopathology in West Nile VirusEncephalitis

A. Müllbacher, M. Lobigs, Eva Lee, 2003, Journal of Virology.

Proteolytic cleavage analysis at the Murray Valley encephalitis virus NS1-2A junction

J. Bettadapura, M. Lobigs, Eva Lee, 2015, Virology Journal.

CD8(+) T cell-mediated immune responses in West Nile virus (Sarafend strain) encephalitis are independent of gamma interferon.

A. Müllbacher, A. Koskinen, M. Lobigs, 2006, The Journal of general virology.

Evidence that a mechanism for efficient flavivirus budding upregulates MHC class I

A. Müllbacher, M. Lobigs, Eva Lee, 2004, Immunology and cell biology.

NK cell‐mediated immunopathology during an acute viral infection of the CNS

M. Alsharifi, M. Simon, A. Müllbacher, 2006, European journal of immunology.

Exocytosis and Fas mediated cytolytic mechanisms exert protection from West Nile virus induced encephalitis in mice

A. Müllbacher, M. Lobigs, Eva Lee, 2004, Immunology and cell biology.

Lack of both Fas Ligand and Perforin Protects from Flavivirus-Mediated Encephalitis in Mice

R. Langman, A. Müllbacher, R. Blanden, 2002, Journal of Virology.

Cytotoxic T Cells Specifically Induce Fas on Target Cells, Thereby Facilitating Exocytosis-Independent Induction of Apoptosis1

M. Simon, P. Waring, A. Müllbacher, 2000, The Journal of Immunology.

Apoptosis Exocytosis-Independent Induction of Target Cells , Thereby Facilitating Cytotoxic T Cells Specifically Induce Fas on

M. Simon, P. Waring, M. Lobigs, 2000 .

Cytolytic effector pathways and IFN‐γ help protect against Japanese encephalitis

M. Regner, M. Lobigs, Maximilian Larena, 2013, European journal of immunology.

Identification of residues in West Nile virus pre-membrane protein that influence viral particle secretion and virulence.

J. Hobson-Peters, P. Young, N. Prow, 2012, The Journal of general virology.

Cytotoxic T Cells Are the Predominant Players Providing Cross-Protective Immunity Induced by γ-Irradiated Influenza A Viruses

J. Chan, M. Alsharifi, A. Müllbacher, 2010, Journal of Virology.

Effect of inactivation method on the cross-protective immunity induced by whole 'killed' influenza A viruses and commercial vaccine preparations.

M. Alsharifi, A. Müllbacher, A. Koskinen, 2010, The Journal of general virology.

InfectionLymphocyte Activation in Response to Viral Type I Interferons Trigger Systemic, Partial

M. Alsharifi, A. Müllbacher, A. Koskinen, 2005 .

Mechanism of Virulence Attenuation of Glycosaminoglycan-Binding Variants of Japanese Encephalitis Virus and Murray Valley Encephalitis Virus

M. Lobigs, Eva Lee, 2002, Journal of Virology.

An Inactivated Cell Culture Japanese Encephalitis Vaccine (JE-ADVAX) Formulated with Delta Inulin Adjuvant Provides Robust Heterologous Protection against West Nile Encephalitis via Cross-Protective Memory B Cells and Neutralizing Antibody

N. Petrovsky, N. Prow, R. Hall, 2013, Journal of Virology.

An inactivated Vero cell-grown Japanese encephalitis vaccine formulated with Advax, a novel inulin-based adjuvant, induces protective neutralizing antibody against homologous and heterologous flaviviruses

N. Petrovsky, R. Hall, T. Komiya, 2010, The Journal of general virology.

Antibodyand Pre-Exposure Neutralizing T + Memory B Cells in the Absence of CD8 Japanese Encephalitis Virus Mediated by JE-ADVAX Vaccine Protection against

N. Prow, M. Lobigs, Maximilian Larena, 2014 .

JE-ADVAX Vaccine Protection against Japanese Encephalitis Virus Mediated by Memory B Cells in the Absence of CD8+ T Cells and Pre-Exposure Neutralizing Antibody

N. Petrovsky, N. Prow, R. Hall, 2013, Journal of Virology.

JE-ADVAX™ Vaccine Protection against Japanese Encephalitis

N. Petrovsky, N. Prow, R. Hall, 2013 .

Pre-exposure Neutralizing Antibody

N. Petrovsky, N. Prow, R. Hall, 2013 .

Glycosaminoglycans in Entry Tropism and Reveal a Role for Flavivirus Alter Virulence and Host Cell Receptor-Binding Site of an Encephalitic Substitutions at the Putative

M. Lobigs, Eva Lee, 2000 .

The mitochondrial protein Bak is pivotal for gliotoxin-induced apoptosis and a critical host factor of Aspergillus fumigatus virulence in mice

C. Borner, P. Ekert, M. Simon, 2006, The Journal of cell biology.

Immunobiology of Japanese Encephalitis Virus

M. Lobigs, Maximilian Larena, 2011 .

Genetic differentiation of Murray Valley encephalitis virus in Australia and Papua New Guinea.

L. Dalgarno, I. Marshall, R. Weir, 1986, The Australian journal of experimental biology and medical science.

Biological, antigenic and phylogenetic characterization of the flavivirus Alfuy.

J. Mackenzie, A. Broom, R. Hall, 2006, The Journal of general virology.

DNA-based and alphavirus-vectored immunisation with prM and E proteins elicits long-lived and protective immunity against the flavivirus, Murray Valley encephalitis virus.

R. Hall, R. Hall, M. Pavy, 1998, Virology.

Substitutions at the Putative Receptor-Binding Site of an Encephalitic Flavivirus Alter Virulence and Host Cell Tropism and Reveal a Role for Glycosaminoglycans in Entry

M. Lobigs, Eva Lee, 2000, Journal of Virology.

Antiviral effect of the heparan sulfate mimetic, PI-88, against dengue and encephalitic flaviviruses.

M. Pavy, C. Freeman, M. Lobigs, 2006, Antiviral research.

Flavivirus premembrane protein cleavage and spike heterodimer secretion require the function of the viral proteinase NS3.

M. Lobigs, 1993, Proceedings of the National Academy of Sciences of the United States of America.

Role of type I and type II interferon responses in recovery from infection with an encephalitic flavivirus.

A. Müllbacher, M. Pavy, M. Lobigs, 2003, The Journal of general virology.

Immunobiology of mosquito-borne encephalitic flaviviruses.

A. Müllbacher, M. Lobigs, Eva Lee, 2003, Advances in virus research.

The Ectromelia Virus SPI-2 Protein Causes Lethal Mousepox by Preventing NK Cell Responses

D. Tscharke, R. Buller, Y. Wong, 2011, Journal of Virology.

Creation of Killer Poxvirus Could Have Been Predicted

A. Müllbacher, Arno Müllbacher, Mario Lobigs, 2001, Journal of Virology.

A flavivirus signal peptide balances the catalytic activity of two proteases and thereby facilitates virus morphogenesis.

M. Ng, M. Pavy, M. Lobigs, 2010, Virology.

Inefficient Signalase Cleavage Promotes Efficient Nucleocapsid Incorporation into Budding Flavivirus Membranes

M. Lobigs, Eva Lee, 2004, Journal of Virology.

Modulation of Transporter Associated with Antigen Processing (TAP)-Mediated Peptide Import into the Endoplasmic Reticulum by Flavivirus Infection

F. Momburg, A. Müllbacher, M. Lobigs, 2001, Journal of Virology.

Recombinant expression of vaccinia virus‐encoded TAP1 and TAP2 promotes MHC class I‐restricted antigen presentation in a Syrian hamster cell line

A. Mullbacher, M. Lobigs, 1995, Immunology and cell biology.

Both E Protein Glycans Adversely Affect Dengue Virus Infectivity but Are Beneficial for Virion Release

S. Leang, A. Davidson, M. Lobigs, 2010, Journal of Virology.

Pivotal Role of Antibody and Subsidiary Contribution of CD8+ T Cells to Recovery from Infection in a Murine Model of Japanese Encephalitis

M. Regner, M. Lobigs, Maximilian Larena, 2011, Journal of Virology.

Mutagenesis of the Signal Sequence of Yellow Fever Virus prM Protein: Enhancement of Signalase Cleavage In Vitro Is Lethal for Virus Production

C. Rice, S. M. Amberg, Sean M. Amberg, 2000, Journal of Virology.

Type I Interferons Trigger Systemic, Partial Lymphocyte Activation in Response to Viral Infection

M. Alsharifi, A. Müllbacher, A. Koskinen, 2005, The Journal of Immunology.

Caspase-Dependent Inhibition of Mousepox Replication by gzmB

M. Simon, A. Müllbacher, A. Koskinen, 2009, PloS one.

Feasibility of cross-protective vaccination against flaviviruses of the Japanese encephalitis serocomplex

M. Diamond, M. Lobigs, 2012, Expert review of vaccines.

Signal Peptidase Cleavage at the Flavivirus C-prM Junction: Dependence on the Viral NS2B-3 Protease for Efficient Processing Requires Determinants in C, the Signal Peptide, and prM

M. Lobigs, C. Stocks, 1998, Journal of Virology.

Physical and Functional Interactions of the Cytomegalovirus US6 Glycoprotein with the Transporter Associated with Antigen Processing*

F. Momburg, H. Hengel, H. Reinhard, 2006, Journal of Biological Chemistry.

Intranasal Flu Vaccine Protective against Seasonal and H5N1 Avian Influenza Infections

M. Alsharifi, L. Trinidad, T. Bowden, 2009, PloS one.

16 Immunobiology of Japanese Encephalitis Virus

M. Lobigs, Maximilian Larena, 2017 .

Can we really learn from model pathogens?

M. Simon, A. Müllbacher, M. Regner, 2004, Trends in immunology.

Live Chimeric and Inactivated Japanese Encephalitis Virus Vaccines Differ in Their Cross-Protective Values against Murray Valley Encephalitis Virus

M. Alsharifi, M. Pavy, M. Lobigs, 2008, Journal of Virology.

Antiviral Cytotoxic T Cells Cross-Reactively Recognize Disparate Peptide Determinants from Related Viruses but Ignore More Similar Self- and Foreign Determinants

P. Milburn, A. Müllbacher, M. Regner, 2001, The Journal of Immunology.

Immunogenicity of two peptide determinants in the cytolytic T-cell response to flavivirus infection: inverse correlation between peptide affinity for MHC class I and T-cell precursor frequency.

A. Müllbacher, M. Regner, R. Blanden, 2001, Viral immunology.

Interferon‐inducible chemokines and immunity to poxvirus infections

G. Karupiah, P. Foster, J. Farber, 2000, Immunological reviews.

Host cell selection of Murray Valley encephalitis virus variants altered at an RGD sequence in the envelope protein and in mouse virulence.

R C Weir, L. Dalgarno, L Dalgarno, 1990, Virology.

Antigen presentation in Syrian hamster cells: substrate selectivity of TAP controlled by polymorphic residues in TAP1 and differential requirements for loading of H2 class I molecules

F. Momburg, G. Hämmerling, A. Müllbacher, 1999, Immunogenetics.

Flavivirus‐Induced Up‐regulation of MHC Class I Antigens; Implications for the Induction of CD8+ T‐Cell‐Mediated Autoimmunity

A. Müllbacher, R. Blanden, M. Lobigs, 1996, Immunological reviews.

The Chemokine Receptor CCR5, a Therapeutic Target for HIV/AIDS Antagonists, Is Critical for Recovery in a Mouse Model of Japanese Encephalitis

M. Regner, M. Lobigs, Maximilian Larena, 2012, PloS one.

E Protein Domain III Determinants of Yellow Fever Virus 17D Vaccine Strain Enhance Binding to Glycosaminoglycans, Impede Virus Spread, and Attenuate Virulence

M. Lobigs, Eva Lee, 2008, Journal of Virology.

Partial dysfunction of STAT1 profoundly reduces host resistance to flaviviral infection.

M. Lobigs, Maximilian Larena, 2017, Virology.

Genetic analysis of Kunjin virus isolates using HaeIII and TaqI restriction digests of single-stranded cDNA to virion RNA.

L. Dalgarno, R. Weir, M. Lobigs, 1986, Australian Journal of Experimental Biology and Medical Science.

Virulence attenuation of Dengue virus due to augmented glycosaminoglycan-binding affinity and restriction in extraneural dissemination.

A. Davidson, P. J. Wright, M. Lobigs, 2006, The Journal of general virology.

Common E Protein Determinants for Attenuation of Glycosaminoglycan-Binding Variants of Japanese Encephalitis and West Nile Viruses

R. Hall, M. Lobigs, Eva Lee, 2004, Journal of Virology.

and prM Peptide, Requires Determinants in C, the Signal NS2B-3 Protease for Efficient Processing C-prM Junction: Dependence on the Viral Signal Peptidase Cleavage at the Flavivirus

M. Lobigs, C. Stocks, 2014 .

Location of a neutralization determinant in the E protein of yellow fever virus (17D vaccine strain).

J. Schlesinger, L. Dalgarno, R. Weir, 1987, Virology.

Cytotoxic T-cell immunity as a target for influenza vaccines.

M. Alsharifi, A. Müllbacher, M. Regner, 2006, The Lancet. Infectious diseases.

Cross-protective and infection-enhancing immunity in mice vaccinated against flaviviruses belonging to the Japanese encephalitis virus serocomplex.

R. Hall, M. Pavy, M. Lobigs, 2003, Vaccine.

Ectromelia virus N1L is essential for virulence but not dissemination in a classical model of mousepox.

D. Tscharke, A. Müllbacher, A. Koskinen, 2017, Virus research.

CD4 + and CD8 + T-Cell Immune Responses in West Nile Virus Infection

A. Müllbacher, M. Regner, M. Lobigs, 2009 .

The flavivirus nonstructural protein NS3 is a dominant source of cytotoxic T cell peptide determinants.

A. Müllbacher, R. Blanden, M. Lobigs, 1994, Virology.

Proteolytic cleavage analysis at the Murray Valley encephalitis virus NS1-2A junction

J. Bettadapura, M. Lobigs, Eva Lee, 2015, Virology Journal.

Ectromelia virus N1L is essential for virulence but not dissemination in a classical model of mousepox.

D. Tscharke, A. Müllbacher, M. Regner, 2017, Virus research.