Wenling Chen

发表

Activation of nuclear receptor CAR ameliorates diabetes and fatty liver disease

S. Wakil, L. Abu-Elheiga, P. Saha, 2009, Proceedings of the National Academy of Sciences.

Latent Sensitization: A Model for Stress‐Sensitive Chronic Pain

B. Taylor, W. Walwyn, A. Minasyan, 2015, Current protocols in neuroscience.

Regulation of human sterol 27-hydroxylase gene (CYP27A1) by bile acids and hepatocyte nuclear factor 4α (HNF4α)

J. Chiang, Wenling Chen, 2003 .

Regulation of human sterol 27-hydroxylase gene (CYP27A1) by bile acids and hepatocyte nuclear factor 4alpha (HNF4alpha).

J. Chiang, Wenling Chen, 2003, Gene.

ABCG 2 and PKD 2 polymorphisms correlate with gout-related metabolic indices in Chinese Han and Tibetan populations

Y. Zhang, L. Kang, Xingguang Luo, 2016 .

A novel AAO-SBSPR process based on phosphorus mass balance for nutrient removal and phosphorus recovery from municipal wastewater.

Yongmei Li, T. Tao, Zhengyu Zhu, 2018, Water research.

Ability of veratridine and high potassium to produce μ -opioid receptor internalization by evoking the release of opioid peptides in the rat spinal cord

B. Song, J. Marvizón, Wenling Chen, 2009 .

Effects of veratridine and high potassium on μ-opioid receptor internalization in the rat spinal cord: Stimulation of opioid release versus inhibition of internalization

B. Song, J. Marvizón, Wenling Chen, 2008, Journal of Neuroscience Methods.

Enkephalins, dynorphins, and β‐endorphin in the rat dorsal horn: An immunofluorescence colocalization study

N. Murphy, J. Marvizón, Wenling Chen, 2009, The Journal of comparative neurology.

Neurokinin 1 receptor activation in the rat spinal cord maintains latent sensitization, a model of inflammatory and neuropathic chronic pain

J. Marvizón, Wenling Chen, 2020, Neuropharmacology.

Sustained Suppression of Hyperalgesia during Latent Sensitization by μ-, δ-, and κ-opioid receptors and α2A Adrenergic Receptors: Role of Constitutive Activity

H. Ennes, W. Walwyn, A. Minasyan, 2016, The Journal of Neuroscience.

A Src family kinase maintains latent sensitization in rats, a model of inflammatory and neuropathic pain

J. Marvizón, Wenling Chen, 2020, Brain Research.

Inhibition of opioid release in the rat spinal cord by α2C adrenergic receptors

B. Song, J. Marvizón, Wenling Chen, 2008, Neuropharmacology.

Noxious mechanical stimulation evokes the segmental release of opioid peptides that induce μ-opioid receptor internalization in the presence of peptidase inhibitors

B. Song, J. Marvizón, Wenling Chen, 2008, Brain Research.

Comparing analgesia and μ-opioid receptor internalization produced by intrathecal enkephalin: Requirement for peptidase inhibition

Woojae Kim, B. Song, J. Marvizón, 2007, Neuropharmacology.

Src family kinases mediate the inhibition of substance P release in the rat spinal cord by μ‐opioid receptors and GABAB receptors, but not α2 adrenergic receptors

J. Marvizón, Wenling Chen, Guohua Zhang, 2010, The European journal of neuroscience.

Cannabinoid CB1 receptor facilitation of substance P release in the rat spinal cord, measured as neurokinin 1 receptor internalization

J. Marvizón, Wenling Chen, Guohua Zhang, 2010, The European journal of neuroscience.

Phosphate release involving PAOs activity during anaerobic fermentation of EBPR sludge and the extension of ADM1

Ruyi Wang, Yongmei Li, Yinguang Chen, 2016 .

cAMP signaling through protein kinase A and Epac2 induces substance P release in the rat spinal cord

H. Ennes, J. McRoberts, J. Marvizón, 2021, Neuropharmacology.

Inhibition of opioid release in the rat spinal cord by serotonin 5-HT1A receptors

B. Song, J. Marvizón, Wenling Chen, 2007, Brain Research.

Corticotropin-Releasing Factor in the Brain and Blocking Spinal Descending Signals Induce Hyperalgesia in the Latent Sensitization Model of Chronic Pain

Y. Taché, J. Marvizón, Wenling Chen, 2018, Neuroscience.

INFLUENCING FACTORS OF EMBRYO RESCUE IN SEEDLESS GRAPE

Hong Lin, Zhendong Liu, Xiuwu Guo, 2015 .

Enzymatic reduction of oxysterols impairs LXR signaling in cultured cells and the livers of mice.

D. Russell, D. Mangelsdorf, Wenling Chen, 2007, Cell metabolism.

PXR induces CYP27A1 and regulates cholesterol metabolism in the intestine Published, JLR Papers in Press, November 6, 2006.

J. Chiang, Tiangang Li, Wenling Chen, 2007, Journal of Lipid Research.

Application of serum pepsinogen and carbohydrate antigen 72-4 (CA72-4) combined with gastrin-17 (G-17) detection in the screening, diagnosis, and evaluation of early gastric cancer.

Wenling Chen, Qiong Li, Huiqin Bian, 2021, Journal of gastrointestinal oncology.

BDNF released during neuropathic pain potentiates NMDA receptors in primary afferent terminals

H. Ennes, W. Walwyn, J. McRoberts, 2014, The European journal of neuroscience.

Mechanisms of μ-opioid receptor inhibition of NMDA receptor-induced substance P release in the rat spinal cord

H. Ennes, J. McRoberts, J. Marvizón, 2018, Neuropharmacology.

Neuropeptide Y release in the rat spinal cord measured with Y1 receptor internalization is increased after nerve injury

B. Taylor, J. Marvizón, Wenling Chen, 2019, Neuropharmacology.

Mu-opioid receptors in nociceptive afferents produce a sustained suppression of hyperalgesia in chronic pain

C. Gaveriaux-Ruff, B. Kieffer, Joshua K. Hakimian, 2018, Pain.

Inflammation enhances Y1 receptor signaling, neuropeptide Y-mediated inhibition of hyperalgesia, and substance P release from primary afferent neurons

B. Taylor, K. McCarson, G. Corder, 2014, Neuroscience.

Enkephalins, dynorphins, and β‐endorphin in the rat dorsal horn: An immunofluorescence colocalization study

N. Murphy, J. Marvizón, Wenling Chen, 2009, The Journal of comparative neurology.

Acute inflammation induces segmental, bilateral, supraspinally mediated opioid release in the rat spinal cord, as measured by μ-opioid receptor internalization

W. Chen, J. Marvizón, Wenling Chen, 2009, Neuroscience.

μ-Opioid receptor inhibition of substance P release from primary afferents disappears in neuropathic pain but not inflammatory pain

W. Chen, J. McRoberts, J. Marvizón, 2014, Neuroscience.