M. Dumont

发表

Mitochondrial dihydrolipoyl succinyltransferase deficiency accelerates amyloid pathology and memory deficit in a transgenic mouse model of amyloid deposition.

M. Beal, G. Gibson, N. Calingasan, 2009, Free radical biology & medicine.

PGC‐1α: overexpression exacerbates β‐amyloid and tau deposition in a transgenic mouse model of Alzheimer's disease

M. Beal, N. Starkova, N. Calingasan, 2014, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Resveratrol protects against peripheral deficits in a mouse model of Huntington's disease

M. Beal, N. Calingasan, E. Wille, 2010, Experimental Neurology.

In Vivo Pathogenic Role of Mutant SOD1 Localized in the Mitochondrial Intermembrane Space

J. Glass, A. Starkov, G. Manfredi, 2011, The Journal of Neuroscience.

N-iminoethyl-l-lysine improves memory and reduces amyloid pathology in a transgenic mouse model of amyloid deposition

M. Beal, Michael T. Lin, C. Nathan, 2010, Neurochemistry International.

Behavioral deficit, oxidative stress, and mitochondrial dysfunction precede tau pathology in P301S transgenic mice

N. Starkova, Lichuan Yang, A. Starkov, 2011, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Influence of mitochondrial enzyme deficiency on adult neurogenesis in mouse models of neurodegenerative diseases

M. Beal, G. Gibson, N. Calingasan, 2008, Neuroscience.

Behavioral improvement after chronic administration of coenzyme Q10 in P301S transgenic mice.

M. Beal, Lichuan Yang, A. Starkov, 2012, Journal of Alzheimer's disease : JAD.

Coenzyme Q10 decreases amyloid pathology and improves behavior in a transgenic mouse model of Alzheimer's disease.

M. Beal, Michael T. Lin, G. Gouras, 2011, Journal of Alzheimer's disease : JAD.

Loss of spatacsin function alters lysosomal lipid clearance leading to upper and lower motor neuron degeneration

A. Brice, G. Stevanin, F. Santorelli, 2017, Neurobiology of Disease.

Behavioral deficits and progressive neuropathology in progranulin‐deficient mice: a mouse model of frontotemporal dementia

M. Beal, Michael T. Lin, C. Nathan, 2010 .

PGC-1 (cid:1) overexpression exacerbates (cid:2) -amyloid and tau deposition in a transgenic mouse model of Alzheimer’s disease

M. Beal, N. Starkova, N. Calingasan, 2014 .

Characterization of hemizygous SOD1/wild-type transgenic mice with the SHIRPA primary screen and tests of sensorimotor function and anxiety

Robert Lalonde, C. Strazielle, M. Dumont, 2004, Brain Research Bulletin.

Apocynin administration does not improve behavioral and neuropathological deficits in a transgenic mouse model of Alzheimer's disease

M. Beal, N. Calingasan, E. Wille, 2011, Neuroscience Letters.

Neuroprotective strategies involving ROS in Alzheimer disease.

M. Beal, M Flint Beal, Magali Dumont, 2011, Free radical biology & medicine.

Neurobehavioral characterization of APP23 transgenic mice with the SHIRPA primary screen

M. Staufenbiel, Robert Lalonde, C. Strazielle, 2005, Behavioural Brain Research.

Pioglitazone halts axonal degeneration in a mouse model of X-linked adrenoleukodystrophy.

M. Beal, E. Galea, M. Portero-Otín, 2013, Brain : a journal of neurology.

Behavioral deficits and progressive neuropathology in progranulin‐deficient mice: a mouse model of frontotemporal dementia

M. Beal, C. Nathan, Rebecca Banerjee, 2010, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Methylene blue upregulates Nrf2/ARE genes and prevents tau-related neurotoxicity.

M. Beal, N. Starkova, M. Portero-Otín, 2014, Human molecular genetics.

Reduction of oxidative stress, amyloid deposition, and memory deficit by manganese superoxide dismutase overexpression in a transgenic mouse model of Alzheimer's disease

M. Beal, N. Calingasan, E. Wille, 2009, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Triterpenoid CDDO‐methylamide improves memory and decreases amyloid plaques in a transgenic mouse model of Alzheimer’s disease

M. Sporn, M. Flint Beal, G. Gouras, 2009, Journal of neurochemistry.

Aberrant regulation of the GSK‐3β/NRF2 axis unveils a novel therapy for adrenoleukodystrophy

M. Beal, M. Portero-Otín, R. Pamplona, 2018, EMBO molecular medicine.

Transgenic mice expressing the human C99 terminal fragment of βAPP: effects on cytochrome oxidase activity in skeletal muscle and brain

Robert Lalonde, C Strazielle, R Lalonde, 2004, Journal of Chemical Neuroanatomy.

Effects of Synaptic Modulation on β-Amyloid, Synaptophysin, and Memory Performance in Alzheimer's Disease Transgenic Mice

E. Capetillo-Zarate, G. Gouras, D. Tampellini, 2010, The Journal of Neuroscience.

Synaptic Activity Reduces Intraneuronal Aβ, Promotes APP Transport to Synapses, and Protects against Aβ-Related Synaptic Alterations

T. Ma, D. Nanus, E. Capetillo-Zarate, 2009, The Journal of Neuroscience.

Regional acetylcholinesterase activity and its correlation with behavioral performances in 15-month old transgenic mice expressing the human C99 fragment of APP

Robert Lalonde, K. Fukuchi, C. Strazielle, 2006, Journal of Neural Transmission.

Spatial learning, exploration, anxiety, and motor coordination in female APP23 transgenic mice with the Swedish mutation

M. Staufenbiel, Robert Lalonde, R. Lalonde, 2002, Brain Research.

Bezafibrate administration improves behavioral deficits and tau pathology in P301S mice.

M. Beal, N. Starkova, Lichuan Yang, 2012, Human molecular genetics.

Benfotiamine treatment activates the Nrf2/ARE pathway and is neuroprotective in a transgenic mouse model of tauopathy

M. Beal, L. Bettendorff, N. Starkova, 2018, Human molecular genetics.

Mitochondria and antioxidant targeted therapeutic strategies for Alzheimer's disease.

M. Beal, M. Dumont, M. Lin, 2010, Journal of Alzheimer's disease : JAD.

Mono‐ and dual‐frequency fast cerebellar oscillation in mice lacking parvalbumin and/or calbindin D‐28k

G. Cheron, S. Schiffmann, C. De Saedeleer, 2005, The European journal of neuroscience.

Transgenic mice expressing the human C99 terminal fragment of βAPP: effects on spatial learning, exploration, anxiety, and motor coordination

Robert Lalonde, K. Fukuchi, C. Strazielle, 2002, Experimental Gerontology.

C9ORF72 knockdown triggers FTD-like symptoms and cell pathology in mice

A. Brice, D. Saracino, S. Boluda, 2023, Frontiers in Cellular Neuroscience.

Triterpenoids CDDO-ethyl amide and CDDO-trifluoroethyl amide improve the behavioral phenotype and brain pathology in a transgenic mouse model of Huntington's disease.

M. Sporn, M. Beal, N. Calingasan, 2010, Free radical biology & medicine.

Developmental and behavioral effects of toe clipping on neonatal and preweanling mice with and without vapocoolant anesthesia.

E. Riedel, N. Lipman, S. Monette, 2014, Journal of the American Association for Laboratory Animal Science : JAALAS.

Spatial learning and exploration of environmental stimuli in 24-month-old female APP23 transgenic mice with the Swedish mutation

M. Staufenbiel, Robert Lalonde, C. Strazielle, 2004, Brain Research.

Behavioral phenotyping of mouse models of neurodegeneration.

M. Dumont, 2011, Methods in molecular biology.